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F Métivier

Publications and source records attributed to F Métivier.

3 recordsLinked to original sources

Diffusivelike buffering and saturation of large rivers.

We use mass balance data from Asian rivers together with a first-order diffusive simplification of the St. Venant-Exner equations to characterize river flood plain processes and discuss the reaction of a large model river to a hill slope supply of eroded masses. The simple analytical solution derived for the long-term profile of the river bed shows that (i) the system converges towards a state in which it reacts to perturbations in erosion of the landscape by small-amplitude oscillations around an average "stationary" state, (ii) to have an effective influence on the river plain profile, the perturbations need to have frequencies smaller than the characteristic frequencies of the river system, and (iii) this river buffering might be linked with a possible long-term saturation of the system carrying capacity.

Journal Article↗

[Cardiovascular repercussions of chronic hemodialysis].

Coronary disease and left ventricular failure are the two main causes of death in haemodialysis patients. In these particular patients it is the accumulation of several risk factors rather than accelerated atherogenesis which seems to be the responsible for the frequent manifestations of a coronary disease which is sometimes difficult to diagnose. Independently of any cardiovascular history, the so-called "uraemic cardiopathy" is characterized by left ventricular dilatation associated with an increased myocardial mass but with a low mass/volume ratio. The systolic function is preserved whereas the ventricular diastolic compliance is altered due to the left ventricular hypertrophy.

Coronary Disease↗

Contributive factors to cardiovascular hypertrophy in renal failure.

Cardiovascular diseases are the leading cause of morbidity and mortality among end-stage renal disease (ESRD) patients. This is related to the accumulation of nonspecific cardiovascular risk factors in this population, aggravated by some specific features, proper to chronic uremia. The principal abnormalities observed in ESRD patients, with no history of cardiovascular disease, consist in an increased left ventricular mass and a chronic left ventricular dilation. The enlargement of the ventricular cavities is due to a chronic flow overload in which the anemia and dialysis arteriovenous fistula play a dominant role. The increase in ventricular mass is usually less pronounced than the ventricular dilation and for a given arterial pressure the ventricular mass-to-volume ratio is inadequately low. The "inadequate" cardiac hypertrophy is more frequently observed in patients with severe secondary hyperparathyroidism and results in a chronic increase in ventricular stress and oxygen consumption. On the other hand, ESRD patients with aluminum overload exhibit an increased ventricular mass which compensates for ventricular dilation. The echocardiographic indices of ventricular systolic function are normal, contrasting with a pronounced impairment of diastolic properties. The impairment of ventricular filling is in part related to the increased left ventricular mass.

Cardiomegaly↗