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Biomedical subjects

F N Zusmanovich

Publications and source records attributed to F N Zusmanovich.

16 recordsLinked to original sources
PubMed1999

[Revascularising osteotrepanation in treatment of lower limbs critical ischemia].

The results of treatment of 202 patients with chronic critical ischemia of the extremities by the method of revascularising osteotrepanation are presented. There were 139 patients with atherosclerosis (AO) and 63 with thromboangiitis obliterans (TO). 69% of (AO) patients showed improvement of the circulation exactly after the operation 73%--in 1 year, 65%--in 3 years and 52%--in 5 years. In TO patients these percentases were--88%, 78%, 73%, 58%, respectively. It was established that the operation is most effective in TO and in distal forms of atherosclerosis. In occlusions of aorto-iliac segment it is indicated when rautine reconstruction procedure is impossible. In patients with severe and protracted painful syndrome unresponsive to narcotics, with "positional" oedemas and humid gangrene of the foot the operation is not advisable. Curative effect increases when revascularising osteotrepanation is combined with profundoplasty, sympathectomy, femoro-popliteal bypass grafting and devitalisation of the adrenal glands.

Arterial Occlusive Diseases↗
PubMed1999

[Method of puncture electrocoagulation in the treatment of patients with varicose veins].

The author proposes to treat varicose veins by electrocoagulation using the needles of different diameter connected with the device EC 500 M. The method was used in treatment of 91 patients mostly with the 2nd and 3rd stages of chronic venous insufficiency. Ligation of the opening of the great subcutaneous vein and coagulation of it was simultaneously fulfilled in 56 patients. The treatment was performed in the out-patient clinic. In most of the patients (97.9%) good clinical and cosmetic results were noted. The method is sufficiently radical and allows the complications characteristic of sclerotherapy to be avoided.

Adolescent↗
PubMed1996

[The treatment of obliterating arterial diseases of the extremities by a revascularizing osteotrepanation method].

A new method to treat obliterative atherosclerosis and thromboangiitis is proposed. The method consists in local osteal trepanation of the ischemic extremity. The access to the bones was preferably performed in bioactive points. The operation called osteotrepanation was performed in 307 patients. 219 of them had obliterative atherosclerosis, 75--thromboangiitis obliterans, 13--Raynaud's disease. In 63% of patients there was 3d and 4th stage of extremity ischemia. In 90.5% of patients improvement was observed. After 1 year the improvement of blood circulation in the extremity was observed in 92.8% of patients, after 5 years--in 92,8% also. Revascularizing osteal trepanation is a method of choice in thromboangiitis obliterans and in impossibility to perform reconstructive surgery in case of grafts thrombosis.

Arterial Occlusive Diseases↗
PubMed1992

[Osteotrepanation--an alternative to amputation of an ischemic extremity].

Local trepanation of bones of an ischemic extremity, mainly in biologically active points, is conducted successfully for activation of collateral circulation in thromboses of reconstructed arteries and shunts when the blood flow in the main arteries cannot be restored for various reasons. The collateral blood flow increases noticeably due to opening of reserve collaterals and the development of new ones.

Adult↗
PubMed1983

[Changes in the skeletal muscles during experimental ischemia of the extremity].

Histochemical and morphological changes in the tibial and calf muscles of the forelimbs were studied in dogs 3 months after experimentally induced ischemia of varying severity. In moderate ischemia, the activity of oxidation-reduction enzymes increases, that of adenozine triphosphatase decreases. The diameter of muscle fibers increases. These changes are considered to be compensatory. Severe ischemia is accompanied by suppressed activity of succinate dehydrogenase, myofibril damage, and formation of centres of ischemic dystrophy.

Animals↗