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G Cochard

Publications and source records attributed to G Cochard.

8 recordsLinked to original sources
PubMed2006

[Arterial gas embolism originating from the lung in anaesthesia and intensive care].

We report seven cases of arterial gas embolism originating from the lung that occurred in anaesthesia and intensive care unit in the very hospital where our regional hyperbaric oxygen facility is. They complicated lung surgery or trauma and/or followed a support by positive-pressure ventilation. Diagnosis was most often delayed, because of some scepticism of the physicians confronted with a variety of clinical features. The prognosis was bad with four deaths, despite treatment with hyperbaric oxygen in three cases.

Adult↗
PubMed2004

[Idiopathic medullary decompression sickness: myth or reality?].

Severe decompression sickness occurs unfrequently, with, generally an identifying cause (error in decompression protocols, promoting factors.). We report a case of severe spinal cord damage; onset after a common dive, neither deep nor long, without any promoting factor, absence of responsiveness to recompression, three hours post-dive, importance of MRI signal abnormalities, make us to point out the confounding variability of onset and evolution of such illness.

Adult↗
PubMedDate not supplied

Pulmonary edema in scuba divers: recurrence and fatal outcome.

Pulmonary edema occurring in divers using a self-contained underwater breathing apparatus (scuba) is an uncommon, probably under-reported, but potentially life-threatening and recurrent condition. We report six episodes of pulmonary edema in five scuba divers seen during a period of 15 months. The four men and one woman ranged in age from 37 to 56 years and two were treated for hypertension. Symptoms were mostly dyspnea onset at depth, cough, hemoptysis and hypoxemia, which in the recurrent case led to cardiac arrest and death. All cases occurred in rather cold water. Findings on thoracic computed tomography (CT) scanning ranged from pleural effusion to ground-glass opacities restricted to a few areas of the lung. The complex underlying mechanisms that would contribute to a raised transalveolar pressure or to a disruption of the blood-gas barrier are discussed. It is important for emergency care providers to be aware of this syndrome for prompt recognition and optimal treatment.

Adult↗