Biomedical subjects

G Csabai

Publications and source records attributed to G Csabai.

4 recordsLinked to original sources

PubMed1992

A suicidal death by explosives.

A 23-year-old man committed suicide by detonating an explosive substance clutched to his abdomen. The face was singed, the hands were lacerated and the chest and the upper part of abdomen were badly mutilated. The death was due to the destruction of several vital organs. The injuries had a directional nature. Only the consideration of all the facts of the case in relation to the situation, nature, distribution and extent of wounds and the preceding medical and social history allowed a reconstruction of the circumstances.

Abdominal Injuries↗

PubMed1985

Increase of alcohol dehydrogenase and protein content of liver following chronic ethanol administration.

Increased alcohol tolerance following chronic alcohol administration has been explained by increased mitochondrial oxidation of NADH and or increased activation of MEOS. According to our experiments this increased tolerance after chronic alcohol consumption is connected with an increased activity of ADH.

Alcohol Dehydrogenase↗

PubMed1982

[Fatal intoxications in Debrecen. Analysis of the cases from 1958 to 1981 (author's transl)].

In this report the toxicologic material obtained since 1958 in our institute has been evaluated. Of the 15,144 postmortem examinations, 997 toxicologic examinations have been carried out. In addition, 739 clinical cases have been studied during this period. There has been a monotonous increase in the number of toxicologic cases up to now. The number of cases is doubled during a 10-year period. Barbiturates are the main type of intoxicating agents found, but there is a rapid increase in the use of other drugs in the material studied. The use of nicotine is steadily decreasing but there is an increasing number of toxicologic cases in connection with the use of other plant protectives. The evaluation of toxicologic cases involving alcohol will be reported elsewhere.

Humans↗

PubMed1982

Histochemical examination of alcoholic liver injury.

The histochemical changes in the rat liver induced by chronic alcohol treatment were investigated. A significant decrease in glycogen content was observed as the first sign of alcoholic liver injury. The change started at the periphery of the hepatic lobule. With the decrease of the glycogen content a fatty degeneration of the involved parts of the hepatic lobule occurred. Fatty degeneration of the liver is to be ascribed to an inhibition of glycogen metabolism. Alcohol in the organism transfers the glucose to the pyruvate-acetyl CoA-triacyl glycerol pathway.

Animals↗