Syringocystadenoma papilliferum of the eyelid.
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Biomedical subjects
Publications and source records attributed to G F Buerger.
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Lower lid retraction is commonly seen in dysthyroid orbitopathy. We have treated 55 lower lids in 38 patients with lower lid retraction by a tarsal transplant from the upper lid to the lower lid. An overall effect of 2-mm improvement +/- 0.7 mm occurred. Eighty-nine percent of the lids achieved the position of the lower lid within 1 mm of the limbus. We think that the upper to lower tarsal transplant offers a technically easy and reasonable solution to moderate lower lid retraction associated with dysthyroid orbitopathy.
The success of a procedure to reanimate paralyzed eyelids is determined by the functional and cosmetic results. When the cornea is covered during blinking and sleeping, function has been restored, while a pleasing cosmetic result has been achieved if the eyes appear symmetrical when the lids are open. Several procedures have been developed to restore closure of the paralyzed upper eyelid (implantation of gold weights or open wire springs) or to correct lower lid lagophthalmos and ectropion (lower lid tightening with a Bick procedure or insertion of a closed eyelid spring). In some cases, even a combination of the Bick procedure and insertion of a spring may be insufficient to correct lower lid droop; therefore, we developed a technique to place cartilage into the lower eyelid to correct lid droop. The procedure, suggested by one of us (D.B.S.), has been performed on 51 patients to date. This article reviews our experience with these 51 consecutive patients.
In 516 consecutive dacryocystorhinostomies, a soft stent of 0.25 in. Penrose drain tubing was utilized. The Penrose drain remained in place for approximately 1 month to enhance ostium patency. Ostium patency and alleviation of epiphora were achieved in 99.4% of these cases. Patient comfort, simplicity of follow-up, success rate of ostium patency, and the low incidence of complications suggest that Penrose drain tubing is an excellent material to use when the surgeon feels that an ostium stent is indicated.
Prominent fatty infiltration of Müller's muscle and the anterior levator muscle was noted at the time of surgery in nine of 115 patients undergoing external levator resection for ptosis. This distribution of adipose tissue was confirmed by light microscopy. Fatty infiltration appeared to be a degenerative change found in adults with congenital and acquired ptosis. Preoperatively these patients exhibited moderate to severe ptosis, fair to good levator function, and no elevation of the lid crease. The desired postoperative results were achieved with standard external levator resection and advancement techniques. The fatty appearance may lead to difficulty in identifying customary anatomical landmarks during surgery. With the exception of Horner's syndrome, abnormalities of Müller's muscle have not previously been described in association with ptosis, to these authors' knowledge.
We used a modified Müller's muscle-conjunctival resection operation to correct cases of minimal blepharoptosis with good levator function. After we everted the eyelid, we placed a marking suture through the conjunctiva and Müller's muscle 4 mm above the superior tarsal border. The suture was used to tent up Müller's muscle and the conjunctiva from the underlying levator aponeurosis, facilitating placement of a resection clamp. These improvements ensure excision of the proper amount of tissue and reduce intraoperative trauma. Eighteen (95%) of 19 eyelids treated with this modification were cosmetically acceptable.
A 3-week-old male infant had an extreme form of entropion caused by a congenital horizontal tarsal kink, which led to corneal ulceration and visual impairment. The combination of absence of the upper eyelid fold, irritability, and blepharospasm in the absence of congenital blepharoptosis is a sign that may facilitate early recognition of this problem.
Based on stained smears of material obtained from the canaliculus, three cases of canaliculitis were presumed to be caused by Actinomyces. Cultures of these cases showed Fusobacterium to be the probable infectious agent producing the disease.
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