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Biomedical subjects

G Hornung

Publications and source records attributed to G Hornung.

At least 19 recordsLinked to original sources
PubMed1993

[The hypoglossal channel and its contents in the posterolateral access to the petroclival area].

Measurements of the hypoglossal canal: The midpoint of the inner orifice of the hypoglossal canal is on average somewhat over 26 mm away from the opisthion. The distances of the canal centre come to approximately 30 mm, those of the outer canal orifice to 34 mm. The distance of the intracranial pores from each other amounts to 28 xmm. The medial portal for the extracranial pore is on an average 33 mm away from the portal of the opposite side, the lateral 40 mm from the lateral portal of the opposite side. The intracranial pore of the canal for the XII. nerve is situated on an average 13 mm in front of the posterior rim of the occipital condyle, the midpoint of the canal is 15.85 mm in front of this measuring zone, and the exit of the canal 19 mm. Some differences between the sexes and measurements of the postnatal increase of these distances have been included in this investigation. The diagnostic and surgical importance of the hypoglossal canal are discussed.

Adult↗
PubMed1987

[Progressive multifocal leukoencephalopathy. Late complication in chronic lymphatic leukemia].

In a 44 year old patient with chronic lymphatic leukemia and secondary antibody deficiency syndrome, a disorder of articulation and a left hemiparesis developed during a thrombocytopenic phase. Computer tomography of the cranium, CSF diagnostics and the electroencephalogram did not provide any indication for the cause of the rapidly progressive cerebral symptoms. In the NMR tomogram, a diffusedly increasing intensity in the T2-weighted tomograms were shown around the central region in the right brain. The patient died of a Pseudomonas septicemia. At autopsy, a typical finding of progressive multifocal leukoencephalopathy was found in the areas altered in the NMR tomography. Papova-like virions could be demonstrated in glial cells by electron microscopy.

Adult↗
PubMed1977

Corticosteroid-induced changes in glucose metabolism of chondrocytes.

Immature mice were treated for up to 12 weeks with daily doses of triamcinolone diacetate. Using quantitative histochemical methods, the proximal epiphyseal plate of the humerus was studied at regular intervals. By the tenth injection significant decrease was noted in the acid glycosaminoglycans content (25%) and in the neutral mucopolysaccharides (30%). Concomitantly, a marked increase was noted in the intracellular depots of glycogen (70%). It is suggested that the hormone's antiglycolytic effect in cartilage caused the glucose metabolic pathway to divert in the direction of glycogen synthesis, and thereby interfered with the normal synthetic pathway of the chondrocytes. The hormone's primary effect on the cells' hexose metabolism could be responsible, at least in part, for its inhibitory influence on bone growth.

Animals↗