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G L Diggory

Publications and source records attributed to G L Diggory.

6 recordsLinked to original sources

Decreased 5-HT2 but not 5-HT1 receptor binding in cortex of rat after chronic administration of dothiepin. Application of the Woolf plot to analysis of binding parameters.

Prolonged administration of the antidepressant drug, dothiepin hydrochloride (30 mg/kg orally twice daily for 24 days), resulted in a significant decrease in the population of serotonin2 (5-HT2) binding sites in the frontal cortex of rats whereas serotonin1 (5-HT1) binding sites remained unaltered. No significant differences in affinity constants for either ligand-binding site interaction were observed. Analyses of the binding parameters was performed using linear transformation methods of the specific binding isotherms according to Scatchard (1949) [Ann. N.Y. Acad. Sci. 51: 660-672] or Woolf (see Haldane, 1957: Nature 179: 832). The resulting parameter estimates generated in each analysis were compared. Although both methods demonstrated the decreased Bmax for 5-HT2 binding sites with no change in 5-HT1 sites after prolonged administration of dothiepin, Woolf analyses gave reliably better estimates of the binding parameters as judged by examination of the respective correlation coefficients for best fit linear regression lines.

Animals↗

Chronic antidepressant administration fails to attenuate apomorphine-induced decreases in rat striatal dopamine metabolites.

The responsiveness of the rat striatal dopamine (DA) receptor system to apomorphine (APO) was assessed after 10 days of antidepressant administration. Desipramine (DMI), dothiepin (DOTH), iprindole (IPR) and nomifensine (NOM) were administered intra-peritoneally, twice daily, to rats for 10 days and 42 h after the last drug dose, animals were injected with APO (25 or 200 micrograms/kg s.c., 15 min) or vehicle. Striatal content of 3,4-dihydroxyphenylacetic acid (DOPAC) and homovanillic acid (HVA), assayed using a recently developed high-performance liquid chromatography-electrochemical detection (HPLC-ECD) method, showed that prolonged administration of all four antidepressant drugs failed to modify the effect of APO on DA metabolism. The results of these experiments therefore do not provide evidence to support the suggestion that subsensitivity in presynaptic DA 'autoreceptors' is a significant biochemical correlate of chronic antidepressant drug administration.

3,4-Dihydroxyphenylacetic Acid↗

An automated method to measure monoamines and metabolites using elevated temperature reversed phase HPLC with electrochemical detection. Application to striatal dopamine and hippocampal serotonin turnover.

High pressure liquid chromatography with electrochemical detection has been successfully used for the analysis of noradrenaline, dopamine(DA), serotonin(5-HT), and selected metabolites in brain. Automated sample injection allows up to 100 samples per day to be analyzed; precise thermostatic control of the chromatography at 45 degrees C increases both method reproducibility and separation efficiency while increasing column life. The method requires minimal sample pretreatment and is rapid and inexpensive. It has been applied to the analysis of rat and mouse whole brain and, in particular, to milligram samples of rat striatum and hippocampus, thus permitting the measurement of regional DA and 5-HT turnover. Effects of selected psychotropic drugs on these processes illustrate the value of the method to either DA or 5-HT turnover studies.

3,4-Dihydroxyphenylacetic Acid↗

Behavioural and neurochemical proteins of 1-[1-([indol-3-yl] methyl) piperid-4-yl]-3-benzoylurea (Wy 25093) in rodents.

Wy 25093 is a novel, selective and potent inhibitor of the neuronal 5-hydroxytryptamine (5-HT) uptake process in vitro and in vivo. The compound was more potent than clomipramine and fluoxetine as an inhibitor of 5-HT uptake in vitro and did not significantly inhibit catecholamine uptake. In addition, Wy 25093 potentiated the behavioural syndrome induced by 5-hydroxytryptophan (5-HTP) and antagonised the hyperactivity produced by p-chloroamphetamine (P-CA). WY 25093 antagonised the (P-CA)-induced depletion of 5-HT in rat brain and reduced the probenecid-induced increase in rat brain 5-hydroxyindole-3-acetic acid (5-HIAA). Acute administration of the agent to rats resulted in reduced 5-HIAA levels without affecting 5-HT; chronic treatment with the compound produced decreases in the levels of both 5-HIAA and 5-HT. It is concluded that Wy 25093 is a selective and potent inhibitor of the neuronal re-uptake process for 5-HT both in vitro and in vivo, and may possess potential antidepressant activity.

5-Hydroxytryptophan↗