PubMed Health⌕ Search

Biomedical subjects

G Piekarski

Publications and source records attributed to G Piekarski.

At least 19 recordsLinked to original sources
PubMed1985

[Transmission of urogenital trichomoniasis in female prostitutes].

A group of female prostitutes (n = 41) was found to be infected with Trichomonas vaginalis to 36.6%, diagnosed by using culture methods. The examination of these women for T. vaginalis and their treatment is not obligatory, therefore the transmission of these parasites by infected prostitutes is not properly controlled; transmission of the trichomoniasis continues under nearly optimal conditions. It is urgently necessary to examine female prostitutes for T. vaginalis infections without exception. In these cases culture methods must be used and all infected women must be treated immediately with effective drugs. The last meeting of the German Society of Gynecologists in 1984 has stressed the necessity to control trichomoniasis systematically.

Adult↗
PubMed1981

Infection of murine peritoneal macrophages with Toxoplasma gondii exposed to ultraviolet light.

Exposure of Toxoplasma trophozoites to ultraviolet light (UV; 2,539 A) remarkably inhibited intracellular multiplication of the toxoplasmas within cultured mouse peritoneal macrophages. These toxoplasmas possessed the ability to induce normal parasitophorous vacuoles (PV) and underwent gradual degeneration in the PV without participation of host-cell lysosomes. Apparently, the basic conformation of the PV, i.e., the association of mitochondria, rough endoplasmic reticulum, and microtubules of the host cell and the presence of microvillous infoldings, was maintained as seen under the electron microscope even after the toxoplasmas had died within the PV. Even PV, in which debris of the toxoplasmas could be observed, did not show the sign of fusion with ferritin-labeled secondary lysosomes.

Animals↗
PubMed1981

[Medical parasitology and development aid (author's transl)].

The impact of severe parasitic infestation on the population of 3rd world countries has been shown; how this heavy physical and psychic burden affects the CNS of the individual and his capacity for work--considered in the light of the experience of veterinarians with parasitic infestation in domestic animals. Economic progress is coupled with the increase in the capacity for work of the individual, which is greatly impaired through severe parasitic infestation. Effective remedies can be found by systematic health education and individual chemotherapy with new drugs appropriate for mass treatment.

Animals↗
PubMed1981

Behavioral alterations caused by parasitic infection in case of latent toxoplasma infection.

In order to assess the effects of a latent Toxoplasma infection on behaviour and learning capacity, maze experiments with rats and mice were performed. The learning performance of rats and especially of mice was impaired, the memory of mice and their overall activity were reduced by infection. Theses results are supported by Hutchinson who also found that the activity of mice was reduced by Toxoplasma infection, and that they were less responsive to novel stimuli. These results indicate that Toxoplasma infection probably affects the animal's response to its environment, and the stimulation arising from it, and may even affect endogenous regulatory processes in the brain. Similar observations in humans suggest that an acquired Toxoplasma infection without giving rise to disease may have similar effects in man. These observations touch the general problem of the influence of parasitic infections on the CNS.

Animals↗
PubMed1980

[New aspects in the field of parasitology and parasitic diseases of man].

The increasing interest in the parasites of man in the Federal Reublic of Germany is connected with the unbounded keeness of German people to travel. They favor southern regions with warm climates which are usually infested with parasites. Thus the general practitioner is nowadays confronted in his daily routine with "imported" pathogenic organisms and diseases as yet unknown to him or with which he need not have reckoned in the past. Furthermore, new information now exists on the development of well-known parasites of our regions, which can be pathogenic to man. Fortunately, new reliable drugs have come on the market, rendering some parasitic diseases harmless.

Animals↗
PubMed1980

Concurrent infections of Trichinella spiralis and Toxoplasma gondii in mice.

Concurrent infections with two parasites: a nematode, Trichinella spiralis, and a protozoon. Toxoplasma gondii, were investigated. Antibody production (total immunoglobulin and IgM) was similar in double and single infections. However, the number of Toxoplasma cysts in the brains of mice infected with Trichinella and challenged 1-6 weeks later with Toxoplasma was higher than in mice infected with Toxoplasma alone, while mice infected with Toxoplasma and challenged 4-14 days later with Trichinella had lower worm burdens in the intestine than animals infected with Trichinella alone. Greater loss in body weight was observed in mice infected with both parasites than in those infected with either parasite alone.

Animals↗
PubMed1979

Pathology of Anopheles stephensi after infection with Plasmodium berghei berghei. I. Mortality rate.

The mortality of P. berghei-infected Anopheles stephensi females can be about 30% higher during the first three days than in normal blood-fed mosquitoes. As expected the mortality is higher after feeding on highly infected mice but also depends on the date of feeding and the temperature. Infected mosquitoes kept at 25 degrees C die more often than those kept at 21 degrees C. On the other hand sporozoite production needs the low temperature of 21 degrees C. So the sporozoite production rate falls with increasing temperature, and the mortality rate increases.

Animal Feed↗
PubMed1979

Pathology of Anopheles stephensi after infection with Plasmodium berghei berghei. II. Changes in amino acid contents.

Infection with Plasmodium berghei results in the disease of a relatively high percentage of mosquitoes depending on the experimental conditions. The damage caused by the parasites may be so severe that the host dies. It can also become manifest for instance in a change in the amino acid content of the mosquito homogenate. The amino acid content of mosquitoes fed on a glucose solution, normal mouse blood, or the blood of infected mice was analysed qualitatively and quantitatively over a period of 14 days. The amino acids lysine, phenylalanine, proline, threonine, and tyrosine are always found in higher concentrations in infected mosquitoes. The content of leucine (and/or isoleucine) increased from the 6th day and glutamic acid from the 9th day compared to the controls. Lower concentrations were found for alanine, aspartic acid, glycine, and serine as compared to uninfected mosquitoes. Further investigations on this subject might help to find the causes for the susceptibility or resistance of individual mosquitoes to plasmodia.

Aging↗