[The effect of Duraphat lacquer on the condition of the permanent teeth in children consuming fluoridated table salt].
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Child↗
Biomedical subjects
G Sávay
Publications and source records attributed to G Sávay.
[Structure and composition of salivary calculi].
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Calcium Phosphates↗
Histochemical evidence for the role of Ca2+ and neutral protease in the development of the subacute myopathy induced by organophosphorous compounds.
A temporal and causal relationship has been established between inhibition of acetylcholinesterase (AChE) by organophosphorous compounds, accumulation of Ca2+, an increase of neutral protease activity and the development of the ultrastructural signs of subacute myopathy.
Acetylcholinesterase↗
Appearance of histochemically detectable ionic calcium in degenerating primary sensory neurons.
The effect of neonatal capsaicin treatment on the fine structure and localization of ionic calcium in sensory ganglion cells has been investigated in rats. Neonatal capsaicin treatment resulted in the degeneration of certain type B sensory neurons. At the same time ionic calcium was demonstrated histochemically in the perikarya of numerous small-sized ganglion cells. The appearance of histochemically detectable ionic calcium in these cells in relation to the pharmacologically evoked degeneration of certain primary sensory neurons is discussed.
Animals↗
[Calcium utilization in the neuromuscular junction after acetylcholin treatment (author's transl)].
In the site of the myoneural junction a liberation of Ca++ ions, which is similar to that described previously after an electric stimulation or use of cholinesterase inhibitor, can be revealed by histochemical methods (heavy metal substitution, tetracycline fluorescence technique and glyoxal-bis-(2-hydroxyanil) complex formation) using repeated acetylcholine and SCh administration. The possible aspecific reaction due to the presence of Mg++ ions can be excluded by means of the glyoxal-bis(2-hydroxyanil) method. It can be suggested that there is a correlation between the liberation of Ca++ ions an the function dependent mitochondrial energetic processes.
Acetylcholine↗
Time course of proteolytic enzyme alterations in the motor end-plates after stimulation.
Authors studied the proteolytic enzyme activity of motor end plates in the diaphragms and M. flexor digitorum brevis of rats after administration of cholinesterase inhibitors and after supramaximal electric stimulation, using the colour film digestion technique. Enhanced enzyme activity characterizing stimulated motor end plates persisted up to 12 h after stimulation. No proteolytic activity could be shown any more 24 h after stimulation, indicating restoration of the resting state. The role of metabolites (oligopeptides and amino-acids) released by the function-dependent axoterminal proteolysis is discussed in relation to neurochemical transmission processes.
Animals↗
[Prove of the proteolytic enzyme activity using color diafilms as substrate (author's transl)].
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Animals↗
Function-dependent proteinase activity in the neuromuscular synapse.
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Aminopeptidases↗
The effect of vasoactive substances on the circulation and glucose absorption of an isolated jejunal loop in the dog.
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Acetylcholine↗
Pb-thiocholine techniques for the electron histochemical localization of acetylcholinesterase.
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Acetylcholine↗
A new modification of the Koelle-Friedenwald method for the histochemical demonstration of cholinesterase activity.
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Acetylcholinesterase↗
A light and electron microscopic histochemical study on the mechanism of DFP-induced acute and subacute myopathy.
The histochemical changes occurring in association with the development of acute and subacute myopathy have been studied in the rat diaphragm 30 min-48 h after a single i.p. injection of 1.82 mg/kg of the irreversible cholinesterase inhibitor organophosphate, diisopropylfluorophosphate (DFP). In addition to a considerable inhibition of the AChE activity of the motor end-plates, accumulation of ionic Ca2+ and an increase in neutral protease activity in the subjunctional sarcoplasm have been demonstrated. A temporal and causal relationship has been established between the histochemical changes and the development of the ultrastructural signs of myopathy.
Acute Disease↗