Biomedical subjects
G Wagenaar
Publications and source records attributed to G Wagenaar.
[Incidence of Clostridium botulinum in the rumen contents and faeces of cattle fed brewers' grains naturally contaminated with Clostridium botulinum (author's transl)].
The number of Clostridium botulinum type B organisms excreted by cattle fed brewers' grains in which these organisms were found to be present and the period for which they were excreted, were studied. Large numbers (10(5) - 10(7) per gramme) of these organisms were detected in the rumen contents and faeces of the animals. When feeding brewers' grains was discontinued, Cl. botulinum type B was still detectable in the faeces for a considerable period (greater than eight weeks). There was evidence to suggest that the number of Cl. botulinum organisms multiplies in the gastrointestinal tract of cattle.
[The purchaser's veterinarian and latent defects (author's transl)].
When the purchaser of an animal is dissatisfied with the animal bought, the question may arise whether the animal has any latent defect. The purchaser's veterinarian should see to it that he retains the initiative in this matter so that he can calmly and carefully decide whether a certificate of latent defect can or cannot be issued. The various possibilities are reviewed.
[Food poisoning in cattle caused by ingestion of brewers' grains contaminated with Clostridium botulinum type B (author's transl)].
In 1977, outbreaks of disease associated with serious losses occurred on twenty cattle farms. This disease was due to the fact that the animals had been fed brewers' grains. The clinical picture was marked by anorexia, profuse salivation, regurgitation and dehydration. The course of the disease varied with the severity of the attack. Toxicological tests were negative. Cl. botulinum type B as well as toxin type B were found to be present in the brewers' grains fairly often. As a rule, Cl. botulinum was also detected in the rumen contents and faeces. The treatment of the animals is described; this resulted in recovery in a number of cases. When experimental animals were fed brewers' grains from farms on which outbreaks had occurred, this induced similar clinical pictures. The results of studies on the aetiology and pathogenesis are discussed. It is concluded that the clinical picture was an atypical form of botulism caused by Cl. botulinum type B. Possible causes of this abnormal clinical picture are discussed in greater detail.
[The seller's veterinary surgeon and the hidden faults in animals (author's transl)].
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[Iron Dextran Administered to Horses (author's transl)].
A description is given of three cases in which horses died very shortly after being given an intramuscular injection of iron dextran. The use of iron dextran in the case of horses is inadvisable.
[The number of veterinary surgeons today and in 1980 (author's transl)].
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