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Biomedical subjects

H H Petersen

Publications and source records attributed to H H Petersen.

26 records · Page 2Linked to original sources
PubMed1995

[Acute pancreatitis--induced by 5-aminosalicylic acid or an extraintestinal manifestation of ulcerative colitis?].

5-aminosalicylic acid has been reported to cause acute pancreatitis. We report the first Scandinavian case. A 27 year-old woman with a three month history of ulcerative colitis treated with Mesalazine developed acute abdominal pain. Serum and urine amylase were raised, and ultrasonographic and computed tomography scan showed oedema of the pancreas. Immediately following withdrawal of the drug, serum and urine amylase returned to normal. We discuss the possibility of acute pancreatitis as an extraintestinal manifestation of ulcerative colitis.

Acute Disease↗
PubMed1994

[Not Available].

Explore the source record for details and available documents.

Disease↗
PubMed1992

[The pacemaker syndrome].

The pacemaker syndrome is a complex of symptoms consisting of heart failure, near fainting, sensations of pulsation in the neck or abdomen or cough which develop or are aggravated after cardiac pacing. Objectively, a fall in systolic blood pressure is observed in the majority of cases and also canon waves in the neck veins, signs of heart failure, retrograde arterial activation and possibly canon-a-awaves in the central venous pressure. The syndrome occurs in approximately 15% of the patients with ventricular pacing. The condition is most probably caused by lack of atrioventricular synchrony with resultant distension of the atria which results in a reflex mediated decrease or defective increase in the total peripheral resistance and, thus, a fall in systolic blood pressure. Treatment consists of establishing normal atrioventricular synchrony either by implantation of an atrial or AV-sequential pacemaker or by re-programming so that the patient has, primarily, his own rhythm. Ensuring normal atrioventricular synchrony has also other advantages as several investigations have shown that 60-80% of the patients prefer this form of pacing rather than ventricular pacing. The working capacity improves and the patients feel subjectively better and the risk for development of chronic atrial fibrillation and heart failure decreases.

Blood Pressure↗
PubMed1992

[The pacemaker syndrome: an overlooked complication?].

The pacemaker syndrome is a frequently occurring and frequently overlooked syndrome which can, as a rule, be treated. The diagnosis is often overlooked on account of the diffuse symptoms together with the frequency of symptoms in elderly patients. The temporal connection with VVI pacing is often, but not always, helpful in establishing the diagnosis. Three case histories with various manifestations of the syndrome are reviewed.

Aged↗
PubMed1990

[Models for calculating risks as a tool in screening for cardiovascular diseases].

In connection with screening for risk factors for ischaemic heart disease in Bispebjerg Hospital, we have assessed three different models for calculation of the risk, employed on our own material. A total of 462 persons participated in the screening and 275 of these were under the age of 65 years. Out of these 275, 92 had plasma cholesterol values over or equal to 7.0 mmol/l and or smoked over 20 gram tobacco daily. On comparison between three models for calculation of the risk: one American, one British and one Swedish, moderate agreement was observed: the correlation coefficients varied between 0.75 and 0.89. The reason for this may be that the models for calculation of the risk are constructed on the basis of statistics already described from epidemiological investigations in which coincidence is demonstrated between selected observable factors and ischaemic heart disease. It is thus possible that the factors which we measure and possibly attempt to influence are not pathogenetic. We consider, therefore, that risk scoring should be employed with caution. As causal connection between ischaemic heart disease and cholesterol and smoking, respectively, have been demonstrated with reasonable certainty, we consider that it is reasonable to screen and intervene for these factors alone.

Adult↗
PubMed1989

Clonidine-induced immune complex disease.

We report upon a 46-year-old woman, who developed immune complex disease after treatment with clonidine for one year. The diagnosis was verified with histological demonstration of IgG and IgM complexes as well as complement C1q, C3c and C4 between muscle fibres and at the dermo-epidermal junctions. The patient's symptoms abated and the abnormal results of blood tests reverted to normal following cessation of clonidine therapy.

Clonidine↗