Biomedical subjects
Publications and source records attributed to H Hops.
Interest in lifespan research and cross-generational associations in parenting practices and child behaviors has grown rapidly in recent years. The four papers presented in this journal test three key intergenerational research questions regarding intergenerational continuities for externalizing behaviors, using different 3-generational samples.
A developing body of research points to the role of family relationships and interactions as factors relevant to understanding unipolar depressive symptomatology and disorder in adolescents. This paper is focused on examining mechanisms by which adverse family processes may operate to increase adolescents' vulnerability to depression. The review is organized into a description and evaluation of four potential mechanisms by which families may influence adolescents' risk: (a) Stress/Support, (b) Social Interactional, (c) Cognitive, and (d) Affect Regulation. The presentation of the individual mechanisms is followed by a brief discussion of how they could be integrated to develop a fuller understanding of familial processes relevant to the etiology and maintenance of depressive conditions. Finally, we conclude with a discussion of overall limitations in the literature and recommendations for future research.
OBJECTIVE: This study examined issues of heterogeneity in multiple stage development as it corresponds to qualitatively different developmental trajectories in alcohol use during adolescence. METHOD: Using a piecewise growth mixture modeling methodology, a two-piece growth model capturing growth trajectories in adolescent alcohol use from middle school (Grades 6 through 8) to high school (Grades 9 through 12) was examined (N = 179; 54% male). It was hypothesized that (1) two stages of alcohol use development with varying trajectories would exist in these data (the first corresponding to development during middle school, followed by a second stage of continuing growth during high school) and (2) there would be multiple growth trajectories (subgroups) of alcohol use in the stage-wise development, with varying effects in initial alcohol use and growth rates of alcohol use. RESULTS: Results indicated the tenability of the two-piece growth model of alcohol use with heterogeneity in the population comprising two distinct latent developmental trajectory classes. Class 1, with a high initial status of alcohol use at Grade 6, showed an upward increase in trajectory only during high school. Class 2, with a low initial status of alcohol use at Grade 6, showed a linear increase in middle school with a second growth spurt at high school entry and continuity in growth throughout the high school years. Analyses, incorporating time-invariate covariates, indicated varying influences of gender, early levels of deviant behavior, family structure (single vs two parent), peer encouragement and parent disapproval of alcohol use, and adolescent deviant behavior upon high school entry, on the two trajectory classes. Results also showed effects of the identified trajectories, with varying magnitudes, on later substance use in young adulthood, with Class 1 showing the strongest continuity in later substance use. CONCLUSIONS: Findings suggest heterogenous development of alcohol use in the adolescent population, associated with varying background and covariate influences. In addition, this heterogeneity is linked to alcohol and other substance use in young adulthood.
The purpose of this study was to explore the relations between child responses to interparental depressive behavior and subsequent child depressive symptomatology. Data were collected on 156 two-parent families. Families completed questionnaire data and participated in problem-solving interactions, which were coded using a microsocial observational system. Three potential child responses to interparental depressive behavior were examined: facilitative, aggressive, and depressive. Results indicated that for female adolescents the displays of facilitative and depressive behavior as well as the suppression of aggressive behavior in response to interparental depressive behavior were related to increases in depressive symptomatology. For males, on the other hand, the display of aggressive and depressive behavior in response to interparental depressive behavior was related to increases in depression. Results provide preliminary support both for the examination of child responses to conflictual marital behavior and for expanding the conceptualization of conflictual marital behaviors to include depressive as well as the more traditionally examined aggressive behaviors, in order to better understand the link between conflictual marital behavior and child depressive symptomatology.
This study tested a model wherein the family conflict, depression, and antisocial behavior of 254 adolescents (mean age = 17 years; 63% female) are prospectively related to functioning within a marital (51%) or dating relationship in young adulthood (mean age = 23 years). Family aversive communication in adolescence and adolescent antisocial behavior predicted couple physical aggression. Family aversive communication predicted dyadic satisfaction and aversive couple communication for married women and dating men. Among those with partners who reported little antisocial behavior, adolescent antisocial behavior inversely predicted couple satisfaction and facilitative behavior. Partner antisocial behavior did not mediate the relation between adolescent characteristics and couple functioning. Findings emphasize the importance of the early family environment and psychopathology of the adolescent in the development of adaptive couple relationships.
This paper presents evidence that the Patterson et al. (1992) model of development of antisocial behavior in children generalizes to the development of a wide array of problem behaviors during later adolescence and that youth antisocial behavior, high-risk sexual behavior, academic failure and substance use form a single problem behavior construct. Structural equation modeling methods were applied to 24-month longitudinal data from 204 adolescents and parents. The model fit the data well, accounting for 52% of the variance in adolescent problem behavior. Specifically, families experiencing high levels of conflict were more likely to have low levels of parent-child involvement. These family conditions were related to poor parental monitoring and association with deviant peers one year later. Poor parental monitoring and associations with deviant peers were strong proximal predictors of engagement in an array of problem behaviors at two-year follow-up.
This study examined the ability of several childhood, school-based, social variables to correctly classify antisocial adolescents. Children (N = 314; 163 boys, 151 girls) in the 3rd-5th grade were assessed on academic and social variables (i.e., peer rejection, aggression, withdrawal, and low prosocial behavior) and followed forward for 6-7 years until the 9th and 10th grade. Adolescent antisocial outcomes included a consensus measure formed from diagnostic interviews, contact with juvenile authorities, adolescent self-report, and mother's report. The gender-differential predictive accuracy and efficacy of the early predictor domains to a consensus measure of antisocial behavior were compared with the same estimates found for adolescent self-report of antisocial behavior. Both gender and criterion-method differences were found. For girls, regardless of the measure of antisocial behavior, early academic problems were the strongest predictors of future problems. For boys' self-reported antisocial outcomes, peer rejection was the strongest independent predictor. For consensus-reported antisocial outcomes, both early fighting-anger and withdrawn behavior displayed equally strong predictive relations. For boys, the combination of early fighting-anger and disruptive and withdrawn behavior was the strongest set of predictors for the consensus measure of antisocial functioning. Predictive accuracy and efficacy estimates are discussed in terms of predictive strength as well as the cost-benefit of misidentification.
OBJECTIVE: This trial examined the effects of both acute and maintenance cognitive-behavioral therapy (CBT) for depressed adolescents. METHOD: Adolescents with major depression or dysthymia (N = 123) were randomly assigned to 1 of 3 eight-week acute conditions: adolescent group CBT (16 two-hour sessions); adolescent group CBT with a separate parent group; or waitlist control. Subsequently, participants completing the acute CBT groups were randomly reassigned to 1 of 3 conditions for the 24-month follow-up period: assessments every 4 months with booster sessions; assessments only every 4 months; or assessments only every 12 months. RESULTS: Acute CBT groups yielded higher depression recovery rates (66.7%) than the waitlist (48.1%), and greater reduction in self-reported depression. Outcomes for the adolescent-only and adolescent + parent conditions were not significantly different. Rates of recurrence during the 2-year follow-up were lower than found with treated adult depression. The booster sessions did not reduce the rate of recurrence in the follow-up period but appeared to accelerate recovery among participants who were still depressed at the end of the acute phase. CONCLUSIONS: The findings, which replicate and expand upon a previous study, support the growing evidence that CBT is an effective intervention for adolescent depression.
OBJECTIVE: This study examined early school-based academic and social variables with concurrent family conflict in predicting adolescent alcohol and other drug use. METHOD: 365 children were assessed initially in grades 2-4 on academic-related and social behavior variables using teacher ratings and rankings, peer nominations and ratings and direct observation of playground and classroom behavior. They were reassessed in grades 9-10, using interviews and questionnaires to determine the initiation and sequence of their use of alcohol, tobacco, marijuana and other hard drugs. RESULTS: In three sets of analyses, independently for males and females, lifetime abstainers were compared with adolescents who had used (1) only alcohol; (2) alcohol and tobacco; (3) alcohol, tobacco and marijuana; and (4) all three plus other hard drugs. The drug classifications represent a normative-deviant continuum of adolescent drug use. Constructs were developed for early academic and social predictors as well as concurrent family conflict. The results showed more wide-ranging academic and social difficulties during elementary school for children falling at the more deviant end of the drug use continuum. For girls, the concurrent home environment appeared to moderate the effect of early academic and social variables. CONCLUSIONS: Substance use established by ages 14-15 can be predicted by academic and social behavior displayed at ages 7-9. This suggests that prevention efforts for alcohol and other drugs may be more effective if directed at earlier antecedent behaviors rather than those that are concurrent with substance use.
Problem-solving interactions were observed in 86 families of depressed adolescents and 408 families of non-depressed adolescents. Sequential analyses indicated that mothers of depressed adolescents were more likely than mothers of non-depressed adolescents to increase facilitative behavior in response to adolescent depressive behavior. Additionally, fathers of depressed adolescents were more likely than their counterparts in families of non-depressed adolescents to decrease aggressive behavior subsequent to adolescent depressive behavior. These analyses suggest that parents of depressed adolescents may be inadvertently reinforcing depressive behavior. Decreases in adolescent depressive symptomatology from year 1 to year 2 were not accompanied by changes in family interaction patterns.
This study examined the progressive relations among adolescent use of alcohol, cigarettes and marijuana using latent growth curve analyses. Specifically, the present study examined three models to determine (1) the effect of prior cigarette use on alcohol use and development and the relationship between change in cigarette use and the development of alcohol use (N = 115), (2) the effect of prior alcohol use on cigarette use and development and the relationship between change in alcohol use and the development of cigarette use (N = 199); and (3) the effect of prior alcohol and cigarette use on marijuana use and development, and the relationship between change in alcohol use and cigarette use and the development, of marijuana use (N = 287). Support was found for the relation between prior levels of substance use and involvement in other substances. Cigarette use, in particular, was particularly important in the subsequent involvement of alcohol and marijuana.
OBJECTIVE: This article demonstrates use of a latent variable model for longitudinal data which encompasses nested structures. Using Multilevel Latent Growth Modeling (LGM), levels of alcohol use and development of alcohol use over 4 years were examined among individuals (adolescents and parents) nested within families. METHOD: An LGM model was tested for alcohol use with a sample of 435 families (435 target adolescents, 203 sibling and 566 parents [168 fathers and 398 mothers]). Adolescents (targets and siblings) comprised 312 boys and 326 girls, with a mean (+/-SD) age at Time 1 (T1) of 13.69 +/- 1.95 years. It was hypothesized that there would be homogeneity in level and development of alcohol use among family members and heterogeneity in alcohol use and development across families. The effects of family status (single-parent, two-parent intact and stepparent families) and socioeconomic status (SES) on family levels of alcohol use were also examined. RESULTS: Results suggested that stepparent families, and less educated and more economically disadvantaged families, had higher family levels of alcohol use and developed in their use of alcohol at a faster rate. CONCLUSIONS: Findings suggest that the alcohol use of individuals in the same family is more alike than that of individuals from different families and that family alcohol use may be influenced by family-level variables such as family status or SES. Methods such as those presented, which incorporate family-level clustering, are likely to provide additional information regarding the etiology of alcohol use and risk factors for alcohol use within and across families.
Conventional covariance structure analysis, such as factor analysis, is often applied to data that are obtained in a hierarchical fashion, such as parents and siblings observed within families. However, multivariate modeling of such data are most frequently done as if the data were obtained as a simple random sample from a single population. An alternative specification is presented which explicitly models the within-level and between-level covariance matrices in familial substance use. Results demonstrate homogeneity in substance use within families but heterogeneity across families which could be accounted for by family-level variables of marital status, economic status, and biological relationships. It is shown that conventional covariance structure software can be easily adapted to handle hierarchical models, providing a large set of new analysis possibilities for multi-level data.
This study used latent growth modeling to examine the effects of level of alcohol use and development of alcohol use during adolescence, on young adult outcomes for males and females. Adolescents (N = 480; mean = 13.03 years, S.D. = 1.44; 264 female) were assessed annually over a 4-year period and then 5-6 years later in young adulthood (mean = 22.49 years, S.D. = 1.50). Chronicity of alcohol use in adolescence was related to higher alcohol use, alcohol-related problems, aggressive behavior, theft, and suicide ideation in young adulthood among both males and females. Development of alcohol use during adolescence was related to alcohol-related problems in young adulthood for males and females, and to higher levels of alcohol use and aggressive behavior for males only. The results indicate that development of alcohol use as well as level of alcohol use in adolescence is important for future adjustment outcomes, and that these relationships vary as a function of gender.
The relations between family support, family conflict, and adolescent depressive symptomatology were examined longitudinally in a sample of 231 female and 189 male adolescents and their mothers. Structural equation models revealed that less supportive and more conflictual family environments were associated with greater depressive symptomatology both concurrently and prospectively over a 1-year period. Conversely, adolescent depressive symptomatology did not predict deterioration in family relationships. Depressive symptomatology and, to a greater extent, family characteristics showed high levels of stability over the 1-year period. Counter to our expectations, the relations between family variable and depressive symptomatology were similar for boys and girls. The results suggest that the quality of family interactions is relevant for understanding the development of depressive symptoms in adolescents.
Latent growth modeling (LGM) was used to analyse longitudinal data for adolescent substance use from five overlapping age cohorts (11, 12, 13, 14, and 15 years at first assessment) measured at four annual time points. An associative cohort-sequential model was tested for alcohol, cigarette and marijuana use with a sample of 345 adolescents (11-18 years old) from an urban area in the Pacific Northwestern region of the United States. Hypotheses concerning the shape of the growth curve, the extent of individual differences in the common trajectory over time, and the influence of family cohesion, peer encouragement and gender on initial substance use and shape of the growth curve were tested. Results indicated similarities between alcohol, cigarette and marijuana initial use and development, with peer encouragement and family cohesion predictive of initial levels of use, and changes in peer encouragement influencing the developmental trajectories of the three substances. Females were higher than males in initial status and developed less rapidly in their use of the substances than did males. Findings are discussed in terms of the similarities and differences in the developmental trajectories of the three substances and the importance of family and peer influences on these trends.
In a prospective study of adolescent depression, adolescents (N = 1,508) were assessed at Time 1 and after 1 year (Time 2) on psychosocial variables hypothesized to be associated with depression. Most psychosocial variables were associated with current (n = 45) depression. Formerly depressed adolescents (n = 217) continued to differ from never depressed controls on many of the psychosocial variables. Many of the depression-related measures also acted as risk factors for future depression (n = 112), especially past depression, current other mental disorders, past suicide attempt, internalizing behavior problems, and physical symptoms. Young women were more likely to be, to become, and to have been depressed. Controlling for the psychosocial variables eliminated the gender difference for current and future but not for past depression.
This article demonstrates a latent growth curve methodology for analyzing longitudinal data for adolescent alcohol use by combining information from different overlapping age cohorts to form a single developmental trajectory. Hypotheses concerning the form of growth in alcohol use, the extent of individual differences in the common trajectory over time, and covariates influencing both initial status and the form of growth were tested. Utilizing five separate age cohorts each measured over the same 4-year period, results suggested a common trajectory existed across the 8 years represented by the cohort-sequential analysis, with alcohol use increasing more rapidly during the adolescents' transition to high school. Family cohesion and peer encouragement for alcohol use were hypothesized to influence both initial status and the trajectory of alcohol consumption during adolescence. While family cohesion served to suppress initial levels of consumption delaying the upward trajectory of alcohol use, peer encouragement was related not only to initial, and elevated, levels of use, but was predictive of those changes that occurred during adolescence. Discussion involves the importance of family and peer influences in the development of adolescent alcohol use and the utility of the cohort-sequential approach in the analysis of longitudinal data.