PubMed Health⌕ Search

Biomedical subjects

H Illig

Publications and source records attributed to H Illig.

At least 19 recordsLinked to original sources

[Changes of medical in-patients from 1960 to 1977 (author's transl)].

In comparison to 1960 double the number of patients were treated as in-patients in 1977 whereas the duration of stay was reduced by a third. Despite the more advanced age of patients the number of deaths diminished. Many of the present-day in-patients are older than previously. Cardiac arrhythmias, cardiac infarction, arterial vascular disease, diabetes, cancer and alcoholism have increased 2- to 6-fold.

Age Factors↗

[Death from or in asthma ? (author's transl)].

23 deaths of patients with severe asthmatic dyspnea were analyzed. There was no correlation between clinical presentation and cause of death certified by post-mortem examination. The cause of death was found outside the airways in 14 patients. Of diagnostic-therapeutic implications are the relative frequency of spontaneous pneumothorax (5) and pulmonary embolism (3). Six times another cause of death was discovered (like tumor invasion, arterial thrombosis, hemorrhagic pancreatitis, myocardial infarction). Acute asthmatic death was prone to happen in the middle-aged asthmatic with less than two years of bronchial asthma. Here like in 5 patients with chronic obstructive airways disease lack of awareness of the seriousness of the patients' state, sometimes cessation of cortisone long-term therapy, seldom abuse of bronchodilator-aerosols seems to be important for the lethal outcome.

Adult↗

[Synthetic depot-ACTH, plasmacortisol levels, and asthmatic syndrome (author's transl)].

Plasmacortisol levels were measured in 33 patients with asthmatic syndrome before and after they received treatment with 6 mg of depot-ACTH (Tetracosactid) over seven days. 75 percent of the patients showed improved VC and FEF after therapy. This effect was not in any way correlated to the basal level of cortisol, nor to the increment during treatment.

Adrenocorticotropic Hormone↗