Preoperative localization of a testosterone-secreting ovarian tumor by retrograde venous catheterization and selective sampling.
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17-Ketosteroids↗
Biomedical subjects
H L Judd
Publications and source records attributed to H L Judd.
Elucidation of mechanism(s) of the nocturnal rise of testosterone in men.
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Adult↗
Effect of luteinizing hormone-releasing factor on Leydig cell function.
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Adult↗
The nocturnal rise of plasma testosterone in pubertal boys.
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Adolescent↗
Endocrine function of the postmenopausal ovary: concentration of androgens and estrogens in ovarian and peripheral vein blood.
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Adult↗
The concentrations of androgens, oestrogens, progesterone and luteinizing hormone in the serum of foetal calves throughout the course of gestations.
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Androgens↗
Familial hyperthecosis: comparison of endocrinologic and histologic findings with polycystic ovarian disease.
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17-Hydroxycorticosteroids↗
Y to X translocation in a woman with reproductive failure. A new rearrangement.
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Abortion, Habitual↗
Maternal virilization developing during a twin pregnancy; demonstration of excess ovarian androgen production associated with theca lutein cysts.
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17-Ketosteroids↗
Pulsatile patterns of gonadotropins and testosterone in man: the effects of clomiphene, with and without testosterone added.
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Adult↗
Serum androstenedione and testosterone levels during the menstrual cycle.
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Adult↗
Androgen and gonadotropin dynamics in testicular feminization syndrome.
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17-Ketosteroids↗
Long-term effect of wedge resection on androgen production in a case of polycystic ovarian disease.
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17-Ketosteroids↗
Pure gonadal dysgenesis with progressive hirsutism.
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17-Hydroxycorticosteroids↗
Changes of ovarian hormonal function with aging.
Striking changes of ovarian function occur with aging. These changes begin subtly with reductions of fecundability being observed after age 25. The transition from reproductive to postreproductive life is characterized by menstrual irregularity including anovulation, or short luteal phases. The most prominent hormonal changes at the menopause are drastic reductions of estradiol and progesterone secretion by the ovary, reflecting the cessation of folliculogenesis and ovulation. Elevations of gonadotropins and reduction of inhibin levels also reflect the loss of folliculogenesis and ovulation. There are accompanying decreases of ovarian androgen secretion; however, the postmenopausal gonad directly secretes more testosterone after, than before, the menopause.
Aging↗
Estrogen replacement therapy and gallbladder disease in postmenopausal women.
OBJECTIVE: To examine the role of estrogen replacement therapy on the development of gallbladder disease in postmenopausal women. DESIGN: Systematic review of the English literature was conducted. All studies that addressed the association between hormone replacement therapy and gallbladder disease published from 1970 to the present were reviewed. RESULTS: Seven observational studies, two clinical trials, two case series, and one nonrandomized and three randomized investigations were reviewed. The results of each study were reported and analyzed. CONCLUSIONS: Estrogen replacement therapy in postmenopausal women increased the chances for gallstone formation.
Bile↗