The effect of distending the junction between the superior vena cava and the right atrium in the dog.
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Animals↗
Biomedical subjects
H M Snow
Publications and source records attributed to H M Snow.
The effect of hypoxia on the pulmonary veins.
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Animals↗
Further evidence for right atrial receptors affecting heart rate.
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Action Potentials↗
Functional distribution of sympathetic and parasympathetic nerves to the heart.
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Animals↗
Chronotropic and inotropic effects of isoprenaline and L-noradrenaline in the dog heart.
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Animals↗
Response to stimulation of the cardiac sympathetic nerves.
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Animals↗
The response of the left ventricle to stimulation of the vagus nerves.
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Animals↗
The measurement of pH, PCO2 and PO2 in blood.
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Acid-Base Equilibrium↗
An assessment of the effect of the sympathetic nerves to the heart.
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Animals↗
Conway Review Lecture 2001. Atheroma and the mechanics of blood flow in arteries.
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Animals↗
The potentiation of sinus arrhythmia by vasoactive intestinal polypeptide (VIP) in the anaesthetized dog.
Vasoactive intestinal polypeptide (VIP) is a neuropeptide released from the vagus, which in contrast to acetylcholine has a long-acting positive chronotropic effect on the heart. The aim of this study, in the anaesthetized dog, was to examine the effects of VIP and a VIP antagonist when injected into the sinus node artery of a vagally intact heart in sinus arrhythmia. The response was compared to that produced by noradrenaline (NAD) infusion and stimulation of the sympathetic nerves to the heart. Mean +/- S.D. of 30 R-R intervals was used to describe mean heart rate interval and heart rate variability. VIP, a VIP antagonist, NAD and sympathetic nerve stimulation all caused increases in heart rate without significant increases in blood pressure. However, only VIP caused an increase in heart rate variability; VIP antagonism and NAD caused a decrease and sympathetic nerve stimulation had no effect. These results suggest that VIP and acetylcholine when released from the vagus act synergistically to increase sinus arrhythmia.
Adrenergic Fibers↗