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Biomedical subjects

H Tanboga

Publications and source records attributed to H Tanboga.

5 recordsLinked to original sources
PubMed1998

Detection of source of haematuria after extracorporeal shock wave lithotripsy (ESWL) by automated measurement of urinary red cell volume.

Midstream urine samples taken from 35 patients with secondary haematuria due to nephrolithiasis and 31 patients with haematuria after ESWL were compared using a red cell analyser (RCA) to differentiate the source of haematuria. Urine samples obtained from both groups were examined by RCA for urinary red cell mean corpuscular volume (UMCV) and urinary red cell volume distribution curves (RCVDC). To rule out the influence of blood MCV (BMCV), BMCVs were determined separately and the ratio of UMCV/BMCV (R) was calculated. Although our findings showed no difference between haematurias after ESWL and nephrolithiasis, we cannot exclude a direct effect of shock waves on renal tissue.

Adult↗
PubMed1980

Alternate day steroids and blood pressure control after renal transplantation.

Conversion to alternate day prednisone therapy resulted in a reduction in blood pressure in hypertensive renal transplant recipients. Plasma renin activity was also reduced and may be connected with the fall in blood pressure. There was evidence of increased aldosterone activity, possibly mediated by ACTH, but there was no evidence that the fall in blood pressure was due to changes in sodium status.

Aldosterone↗
PubMed1980

Hypertension following renal transplantation: the role of the host's kidney.

Renin, aldosterone and exchangeable sodium were measured in 38 hypertensive and 56 normotensive renal transplant recipients with good renal function and without renal artery stenosis. Response to competitive blockade of angiotensin II using saralasin was studied in 20 of the hypertensive group. Hypertension was uncommon when bilateral nephrectomy had been performed. When diseased kidneys remained in situ, blood pressure after transplantation correlated well with blood pressure on dialysis. Plasma renin activity was higher in hypertensive patients despite higher exchangeable sodium levels. Urinary aldosterone was also higher and correlated well with blood pressure and plasma renin activity. Angiotensin II blockade produced a fall in blood pressure proportional to plasma renin activity. These observations suggest that hypersecretion of renin is an aetiological factor in the hypertension of renal transplant recipients. It appears to act both through the stimulation of aldosterone secretion, with resultant salt retention, and through the direct vasoconstrictor action of angiotensin II. Since renin levels were lower in patients subjected to bilateral nephrectomy the source of the excess renin is probably the host's kidneys.

Adult↗
PubMed1978

Haemodialysis-induced leucopenia and activation of complement: effects of different membranes.

The effects on neutrophil count and complement activity of five different haemodialysis membranes were studied. There was no correlation between the degree of neutropenia and intensity of complement activation. With cuprophan membrane both occurred simultaneously but to unrelated degrees; polyacronitrile induced mild, not significant neutropenia but marked activation of complement; polycarbonate membranes induced severe neutropenia without detectable complement activation. Where complement activation occurred it was via the alternative pathway. Haemodialysis induced neutropenia may have many causes and complement activation is probably not the major responsible factor.

Acrylic Resins↗