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Biomedical subjects

H Trettin

Publications and source records attributed to H Trettin.

8 recordsLinked to original sources
PubMed1993

[Craniocerebral trauma caused by sports. Pathogenic mechanism, clinical aspects and physical therapy with special reference to manual lymph drainage].

On top of one comprehensive English accident statistics of head injuries in sports there is riding, followed by rugby and football. In Germany there is not very little of head and brain damage due to football as wide spread sports, followed by riding, athletics and gymnastics, whereas in boxing recurring head injuries of dosage underlaying concussion of the brain do play a part even in spite of wearing boxing caps. Anyway, damage of the brain will always be the most heavy complication. Besides uncomplicated wounds of the soft parts such as wounds of the scalp or contusion of the face the closed skull and brain damage will be the most generally form of head injury in sports, followed by fractures of the skull with contusion of the brain and intracranial hemorrhage. In the treatment of the post-concussion and post-contusion syndrome manual lymph drainage will achieve more importance by its anti-edematous and therefore depressant and also sedative effects.

Athletic Injuries↗
PubMed1992

[Neurologic principles of edema in inactivity].

The complete immobilisation of a limb alone can lead to the formation of oedema. Whereas the oedema secondary to inactivity induced by immobilisation is completely reversible, and will only lead to tissue damage in the longterm, neglect of oedema secondary to inactivity in the presence of central and peripheral paresis (apoplectic insult, paraplegia, damage to the plexus brachialis) may entail serious consequences due to the danger of tissue fibrosis. With paresis of an extremity, the lymphovenous return is impaired by two decisive factors: increased hydrostatic pressure in the distal limb segment, and absence of the muscle pump. In flaccid paresis, where there is low muscle tone and no muscle pump action, there is also a low venous tone and the resultant hydrostatic pressure is especially high. Venous stasis in the sub- and prefascial veins leads to increased protein loss from the venous limb of the capillaries and the venules. Compensation initially occurs in the prefascial lymph outflow region (latent oedema) which becomes decompensated if overloaded (visible oedema). Fibrosis of the subcutis and trophic skin changes are the result. In spastic paresis the regional subfascial lymphatic system responds with lymphangiospasm. Where the sympathetic innervation is interrupted (e.g. brachial plexus paralysis) there is passive hyperaemia of the terminal vessels with vascular dilatation and lymphangioparalysis. Insufficiency of the vascular walls results in an accumulation of protein in the tissues, which ultimately ends in fibrosis with ankylosis and shortening of the tendons and muscles. The early administration of complex physical decongestion therapy with manual lymphatic drainage can prevent this state.

Autonomic Nervous System↗
PubMed1989

[Manual lymph drainage in migraine treatment--a pathophysiologic explanatory model].

Recent advances in migraine research indicate that migraines are most probably a genetically caused malfunction of the neurotransmitting system in the human brain stem and diencephalon. As a consequence of a dysfunction of serotonergic and noradrenergic neurons, a disequilibrium of the vegetative nervous system occurs with sympathetic hyperexcitability. Exogenic and endogenic stress factors then precipitate migraine attacks. When biochemical substances are released, vascular spasms, vasodilatation and edema of the vascular walls are triggered. The actual migraine pain occurs via pain mediator sensitization of nociceptors in the vascular walls. A pain attack can be suppressed effectively, and in many cases even arrested, by performing manual lymph drainage at an early stage. The active mechanism can be explained by peripheral-analgesic and central sedative and analgesic effects. The interval mechanism is based on the sympathetic-suppressive effect of ML. These mechanisms will be discussed.

Drainage↗
PubMed1989

[Pathogenesis, diagnosis and therapy of nerve lesions following combined (surgical-radiotherapy) cancer therapy].

Classical cancer therapy (surgical and/or radiotherapeutic measures) is not uncommonly subject to late complications which frequently confront the patient and therapist with problems which are difficult to resolve. Typical examples of such late complications are damage to the nerve plexuses which are usually associated with pain, functional deficits and pareses which can hardly be influenced. The topography of these two nerve plexuses as well as the pathogenesis, clinical symptoms and therapy of plexus lesions is reported with reference to two cases: plexus lesion after combined therapy a) of a malignant testicular tumor and b) of a breast cancer.

Brachial Plexus↗
PubMed1982

[Migraine accompagnée].

The symptomatology of migraine accompagnée is described. The order of the symptoms, the suspected localisation and the possibility of permanent defects, heredity and treatment are discussed. The results obtained by traditional methods of examination such as blood flow measurements, arteriography and EEG are presented, as are those of more modern techniques such as skull rheography and computed tomography.

Aphasia↗