[The frequency of gliovascular heterotopies (dysplasias)].
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Brain↗
Biomedical subjects
H Závodská
Publications and source records attributed to H Závodská.
[Annotation to the mitochondrial genome].
After a brief explanation of the mitochondrial function, especially in the relation to the inner-cell coordination, the study analyzed the mitochondrial hypertroph-dilatative cardiomyopathy, myopathy and scrapie which were recently tied to the "D-loop fragment" of the mtDNA. Any primary connection between viral unconventional slow infections and the mitochondrial genome seems unlikely. It is argued in the study that this category of diseases can be much better explained through the transfer of the so-called mobile retroelements.
Animals↗
[The problem of aneurysma dissecans in an example of an aorta "pseudoduplex"].
Dissecting aneurysms of the aorta, taking a chronic course are rare. We describe a case of a female patient who died at the age of 75 years. The left intercostal and lumbar arteries and the left renal artery originated from the aneurysmatic cleft in the aortic wall whereas the corresponding right hand-side arteries originated from the proper aortic lumen. The proximal part of the left common carotid artery was compressed by the aneurysm. The dissecting aneurysm extended into the left common and external iliac arteries. It became obvious from the history of the patient and repeated x-rays of the chest, that the aneurysm had existed 35 months before death. Our case is similar to that published by Busse in 1906. No such cases have been published in recent years. This is an indication of the acute fatal outcome of most of the dissecting aneurysms of the aorta.
Aged↗
[Creutzfeldt-Jakob disease and Gerstmann-Sträussler syndrome with reference to their differential diagnosis].
Differential diagnosis has quite often proved to be difficult between CREUTZFELDT-JAKOB disease (CJD) and GERSTMANN-STRAUSSLER syndrome. Clinical and morphological aspects as well as the pathological course and differential diagnosis of CJD and Gerstmann-Strüssler syndrome are discussed in some detail. Slow influence can be morphologically diagnosed with reference to the overall pattern of individual alterations with inclusion of scrapie-associated fibrils. This morphological diagnosis can be verified by experimental inoculation to susceptible animals.
Animals↗
[The effect of formalin fixation on several properties of the brain].
Described in this paper are the effects on formalin fixation of 106 human brains autopsied at a large neuropsychiatric hospital. Mass and volume of the brains changed considerably during a 3-week fixation period. On average mass increased by 50 g, volume by 57 ml. Mean brain density decreased during the first week and attained its final value almost at the end of the third week. Most changes seem to happen during the first week. Male and female brains behaved differently. Formulas to predict at autopsy time mass and volume of the brain after 3-week fixation in formalin are presented. Possible interpretations of our findings are discussed and compared with results reported by previous workers on this subject.
Adolescent↗
[Changes in dendrites in disease and aging].
Just like in other organs, e.g. in the skin or in the supporting tissue, in the CNS too the reactions to diverse noxae or causes are always stereotyped. The mental or motor retardations are distinguished by a deterioration of the neuropils, especially by the recession of the dentritic branching and their spinae. Also during physiological aging the dendritic apparatus decreases. The neuropilic differences during physiological and pathological aging, however, are rather quantitative than qualitative ones. Dendritic proliferation as a compensation mechanism for neuron recession in old age is of particular importance.
Aged↗