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I Coma Canella

Publications and source records attributed to I Coma Canella.

At least 19 recordsLinked to original sources

[Clinical practice guidelines of the Spanish Society of Cardiology on unstable angina/infarction without ST elevation].

This paper up-dates the Clinical Guidelines for Unstable Angina/Non Q wave Myocardial Infarction of the Spanish Society of Cardiology. Due to the increased efficacy of adequate management in the early phases, it has been considered necessary to include recommendations for the pre Hospital and Emergency department phase. Prehospital management. Patients with thoracic pain compatible with myocardial ischemia should be transferred to Hospital as quickly as possible and an ECG tracing performed. Initial management includes rest, sublingual nitroglycerin and aspirin. In the Emergency department. Immediate clinical attention and accessibility to a defibrillator should be available. If ECG tracing discloses ST elevation reperfusion strategy is to be implemented immediately. If no ST elevation is present, the probability of myocardial ischemia and risk factor evaluation is essential for adequate management. A simplified risk stratification classification is presented, that also determines the most adequate site for admission: Coronary Care Unit if high risk factors are present, Cardiology ward for the intermediate risk patient and ambulatory treatment if low risk. Management in Coronary Care Unit. Includes routine ECG monitoring and analgesia. Antithrombotic and anti ischemic treatment include new indication for GP IIb-IIIa and Low molecular weight heparins. Coronary arteriography and revascularisation are recommended, if refractory or recurrent angina, left ventricles dysfunction or other complications are present. Management in the ward is based on adequate chronic medical treatment, risk stratification, and secondary prevention strategy. Coronary arteriography before discharge must be considered in the light of the result of non-invasive tests.

Angina, Unstable↗

[Study of myocardial viability using single-photon emission computed tomography with 201-thallium and re-injection in 4 series (early and late images].

OBJECTIVE: The present study was designed to determine whether 24-hour imaging after thallium reinjection or imaging obtained shortly after reinjection provides better results regarding reversibility of fixed perfusion defects observed in conventional stress-redistribution imaging. PATIENTS AND METHODS: We studied 24 patients undergoing stress-redistribution thallium tomography with fixed defects (15 exercise, 6 adenosine, 3 dobutamine). All patients had coronary angiography and 17 a history of myocardial infarction. After obtaining the redistribution images, 1 mCi thallium was injected at rest, and images were acquired at 30 minutes and 24 hours after reinjection. The tomograms obtained were divided into 12 segments and analyzed quantitatively. RESULTS: Of the 190 abnormal segments on the stress images, 53 (28%) demonstrated improved thallium uptake on redistribution images and 137 had persistent defects. Shortly after reinjection, 33 (24%) segments had improved thallium uptake and 104 had persistent defects, 29 (28%) of which showed further improvement in the 24-hour study. In patients with myocardial infarction, of the 36 fixed severe defects, 9 (25%) had improved thallium uptake shortly after reinjection, increasing activity from 36 +/- 10% to 53 +/- 8%, and 22 (61%) defects improved at 24 hours, increasing activity from 37 +/- 8% to 56 +/- 6%. Therefore, 13 irreversible segments in the short-term study after reinjection were reversible on 24-hour images. CONCLUSION: These data indicate that 24-hour imaging after thallium reinjection provides better results regarding reversibility of fixed perfusion defects observed in conventional stress-redistribution imaging than imaging obtained shortly after reinjection.

Adult↗

[Role of noninvasive examinations in the management of ischemic heart disease. III. Assessment of myocardial viability].

Assessment of myocardial viability is a field of growing interest. This article summarizes the pathophysiology of myocardial stunning and hibernation; both phenomena are associated with the presence of dysfunctional, viable myocardium. The techniques that are currently available for the assessment of viability, and the clinical situations in which these assessments may be more useful are discussed.

Animals↗

[Shock secondary to right ventricular infarction (author's transl)].

The diagnosis of right ventricular infarction was made in a patient with acute inferior myocardial infarction on the basis of: right atrial pressure higher than pulmonary capillary pressure, increase of echocardiographic diameter, and abnormal technetium pyrophosphate deposit on the right ventricle. The patient developed a severe shock syndrome of 30 hours duration, which was successfully corrected with massive fluid volume replacement. Study of ventricular functions suggested right ventricular infarction as the cause of shock, since this ventricle showed a greater degree of functional impairment.

Aged↗