Biomedical subjects

Iu D Kalashnikov

Publications and source records attributed to Iu D Kalashnikov.

4 recordsLinked to original sources

PubMed1979

[Pathogenesis of symptomatic facial hemiatrophy].

The data on clinical and electroencephalographic examination of 19 patients are presented. The patients developed facial hemiatrophy after they had sustained ganglionitis of the superior cervical sympathetic ganglion, stem encephalitis, trifacial neuralgia (neuritic stage), tumours of the Gasserian ganglion and syringobulbia. A pathogenetic relationship between the development of symptomatic facial hemiatrophy and lesions of the vegetative and somatic apparatus in the cerebral (mesencephalotruncal area) and extracerebral (superior cervical sympathetic ganglion) structures of the nervous system is noted.

Arachnoiditis↗

PubMed1978

[Monosynaptic spinal reflex in epilepsy].

In 33 patients with generalized convulsive seizures a monosynaptic spinal reflex was studied. In an interattack period the author observed its clear asymmetry which was combined with a contralateral situation of epileptogenic focus on the EEG. In the majority of cases in patients with diffusive paroxysmal activity on the EEG asymmetry in the quantity of H-reflex was marked as well. The obtained data permit to recommend a study of H-reflex in an interattack period of epilepsy for specification of the topics of an epileptogenic focus.

Adolescent↗

PubMed1977

[Diagnosis of seizures of ischemic origin in outpatient practice].

Among the group of 216 patients with cerebral attacks, 40 patients were selected with different syncope-like attacks, epileptic nature of which was rejected or was dubious. On the basis of clinical examinations and analysis of the phenomenology of paroxysms, a group of patients was distinguished where there were attacks of sudden falling in cervical osteochondrosis and a syncopal vertebrogenic syndrome, patients with simple and convulsive syncopes, as well as patients with cerebral attacks of an uncertain origin. It is assumed that there may be an ischemic genesis of syncope-like attacks, on the background of which an inclusion of secondary epileptic mechanisms is possible, especially in some individuals with constitutional or acquired predisposition to epilepsy. This may cause a development of convulsive phenomena and a corresponding EEG picture. Consequently the authors discuss the expediency of including anti-epileptic preparations into the scheme of such patients.

Adolescent↗

PubMed1976

[Pterygopalatine ganglion syndrome].

An examination of 75 patients with the syndrome of pterygopalatine ganglian established that in out-patient practice such disorders are frequently incorrectly diagnosed. As a rule the disorder is seen at young and middle age after general infections and local inflammatory processes. Acute and subacute development is expressed more frequently in a neuralogical form of the disease while the chronic development corresponds more commonly to a neurotic affection of the ganglion. Clinical criteria in the diagnosis of the pterygopalatine ganglion syndrome are pain attacks with a certain localization and its irraidiation, accompanied by vegetative vascular and secretory changes of a parasympathical character. An important sign in the differential diagnosis is an arrest of the attack by a cocainization of the mucous in certain parts of the middle nasal passage.

Adolescent↗