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Biomedical subjects

J A De Oliveira

Publications and source records attributed to J A De Oliveira.

8 recordsLinked to original sources
PubMed2003

Morphometry and histochemistry of the semitendinosus muscle of tufted capuchin monkeys (Cebus apella Linnaeus, 1758).

Considering monkeys are animals closely related to the human, and semitendinosus muscle has been used in many postural research experiments, we have decided to study its histochemical characteristics. Samples were removed from the proximal, middle and distal regions of the semitendinosus muscles of five adult male tufted capuchin monkeys and observed for reaction with m-ATPase (with alkaline and acid pre-incubation), nicotinamide adenine dinucleotide tetrazolium reductase, and haematoxylin and eosin. The muscle fibres were classified as fast glycolytic (FG), fast oxidative glycolytic (FOG) and slow oxidative (SO) and quantified in terms of frequency and area. The three regions of the semitendinosus muscle showed no significant differences in frequency or area of the respective fibre types, and therefore the muscle can be considered histoenzymologically homogeneous. FG fibres presented higher frequencies and larger areas. The sum of FOG and SO fibres was 57.5%, suggesting that the semitendinosus muscle of the tufted capuchin monkey is adapted to an action involved in posture maintenance and in long arboreal dislocation. The present data agrees with the notion of differentiated quadrupedalism in some primates, which support a lower percentage of their weight on the fore limbs.

Analysis of Variance↗
PubMed1995

An infarct-like myocardial lesion experimentally induced in Wistar rats with Africanized bee venom.

There is evidence in the literature that death following a bee or wasp sting may result from cardiac involvement. This study describes acute cardiac lesions experimentally induced in Wistar Rats submitted to intravenous inoculation of Africanized bee venom (ABV) and killed 1, 4, and 24 h after inoculation. Significant increases in serum enzyme levels were detected; light microscopy showed necrosis of the myocardium; and enzyme histochemistry showed inactivation of enzymes in and around the areas of necrosis. This is the first report of an acute necrotizing cardiac lesion, similar to human myocardial infarction, produced by the inoculation of ABV.

Animals↗
PubMed1992

[Recurrent bacterial meningitis].

The study of CSF fistulae, and especially those involving otolaryngological anomalies, must be based on the search for the causative problem of recurrent meningitis. Congenital malformations, post-traumatic and post-operative situations or even diseases involving the cranial bones are basic causes that should be studied. Currently, cranial trauma is the most usual cause of CSF fistulae, with the possibility of recurrent bacterial meningitis.

Brain Injuries↗
PubMed1975

[Stress due to immobilization and low protein intake in rats. Biochemical changes].

Rats fed a control casein diet, when exposed to immobilization, loose more nitrogen, urea and vitamin C in urine. Animals receiving a protein deficient diet excrete less nitrogen, urea and ascorbic acid in urine than their controls. When protein deficiency and immobilization are associated, there is not an increase on the urinary excretion of those substance compared to the deficient animals.

Animals↗
PubMed1975

[Stress caused by immobilization and low protein intake in rats. Morphological changes].

The periodic immobilization of the animals receiving a control diet resulted in a smaller weight gain than in controls. The animals fed a protein deficient diet gained also less weight as compared to controls. The association of the deficiency and the stress of immobilization further decreases the weight gain, but the differences were not significant. The animals submitted to restraint or protein deficiency stress showed an increase in their heart weight in relation to body weight. This increase in more significant when immobilization and protein deficiency were associated. The degree of cardiac muscle fibers damage was also found more pronounced in this group of animals, when analysed through histochemical methods. Liver weight was not found different in the animals submitted to the experimental program. Histological examination of this organ showed fatty infiltration in the protein deficient animals. The picture was not found to be agravated when the animals were further stimulated by the restraint mechanism. Spleen weight was found to be similar in all groups. Histological study of the protein deficient group showed small lesions, that were increased when the deficiency was associated to immobilization. The relative testis weight of the restraint animals receiving the control diet was found to be similar to the animals not immobilized. The protein deficient animals have significantly smaller testis weight than the well nourished group. The histologic study showed that restraint or protein deficiency are responsible for a sexually retarded animal, but the lesions increased with the association of restraint and deficiency.

Animals↗