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J Gehring

Publications and source records attributed to J Gehring.

22 records · Page 2Linked to original sources
PubMed1982

[Electrocardiography and quantitative levocardiography in chronic transmural infarct. A correlative study].

For evaluation of a connection between the electrocardiographic diagnosis and localisation of chronic infarction and the angiographic results left ventriculography data of 97 patients with coronary heart disease were assessed quantitatively according to the method of Mathes. According to electrocardiographic criteria of infarction six subgroups were distinguished: anteroseptal, anterior, anterolateral, inferior, inferiolateral, and posterolateral. Asynergies were demonstrable in 87% of patients with infarct electrocardiograms, normal ventricular function was seen in 80% of patients without infarct ECG. Asynergies were seen in 75% of electrocardiographically diagnosed anterior wall infarcts and in 92% of posterior wall infarcts. The sensitivity of the ECG in chronic infarction was 83%, the specificity 84%. Pronounced differences in the number of involved segments were found between inferior and inferiolateral infarction. In contrast, no clear-cut differentiation of electrocardiographic findings judged by segmental involvement in the left ventriculography was seen for the anterior wall. The extent of the anterior wall infarction diagnosed by electrocardiography did not correlate with results of left ventriculography. The ECG is a suitable means for detection of segmental disturbances of contraction also in the chronic infarction stage. However, localisation and extent of the lesion can only be assessed within limits.

Angiocardiography↗
PubMed1981

[Reversible asymmetric septal thickening in the echocardiogram in a case with suspected perimyocarditis (author's transl)].

A 42-year-old male patient, previously in good health, developed signs of pericarditis, pericardial effusion and possible myocarditis 3 weeks after a virus infection of the upper respiratory tract. Because of enlargement of the previously normal cardiac silhouette, an M-mode-echocardiogram was performed. A pericardial effusion and pericardial thickening was diagnosed. Disproportionate septal thickening was noted (septum/posterior wall = 1.4). 3 days after institution of cortisone therapy gradual clinical improvement started. on day 6 a repeat M-mode-echocardiogram showed regression of the pericardial effusion of the septal thickening. Consecutive echocardiograms showed complete disappearing of the pericardial effusion, regression of the pericardial thickening, and complete normalisation of the left ventricular dimensions. Computer-assisted analysis of the first echocardiogram revealed reduced rate of septal and posterior wall thinning and prolongation of the early diastolic period of rapid filling, while peak VCF remained within normal limits. These changes were much less apparent on day 6. On day 26 all relaxation- and contraction parameters were within normal limits. It is concluded that in this case transient disproportionate thickening of the interventricular septum, prolongation of the early diastolic period of rapid left ventricular filling and reduced rate of diastolic septal and posterior wall thinning may have represented edematous and/or inflammatory changes of the myocardium. It is supposed that these findings may represent early changes in acute peri-myocarditis.

Adult↗