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Biomedical subjects

J Griffe

Publications and source records attributed to J Griffe.

12 recordsLinked to original sources

[Peritonitis encapsulans].

Encapsulating peritonitis is characterized by the formation of a membrane which encases all or part of the abdominal viscera. It is a fibrous perivisceritis. It may answer to many different causes. Tuberculosis, though often considered, is only infrequently ascertained. Other etiologies are: trauma, colitis, jejuno-ileitis, appendicitis, etc. The clinical presentation is a subocclusive syndrome with, in some instances, a fixed abdominal mass giving a paradoxically resonant percussion note. Roentgenograms show gastroduodenal displacement, signs of stenosis, and agglutinated small bowel loops. The best procedure is excision of the membrane with complete decortication. Subsequently, the main risk is recurrence of bowel obstruction.

Abscess

[A new case of carcinoma arising in Caroli's disease (author's transl)].

A new case of carcinoma involving Caroli's disease in a 51 years old woman is reported. This papillary carcinoma is developed in left hepatic duct. The type is quite different from the previous cases already described. It is not a cholangiocarcinoma but a papillary mucoid carcinoma.

Adenocarcinoma, Mucinous

[Annular pancreas in the adult (author's transl)].

Annular pancreas--a congenital malformation consisting of a ring of pancreatic tissue around the second part of the duodenum--is a rare surgical condition in adults. It may be responsible for occlusion in the newborn, but frequently remains latent or does not reveal itself until adulthood, usually around the age of 40. The only symptoms are ulcer-like epigastric pains; vomiting occurs at a late stage and jaundice irregularly. Hypotonic duodenography shows, at best, duodenal stenosis, the extrinsic origin of which is disclosed by fiberoscopy. Pre-operative investigations should aim at detecting a gastro-duodenal ulcer or an associated chronic pancreatitis. Owing to the presence of an excretory duct within the ring, section of the latter entails a risk of fistulisation. The best prospects of cure are offered by digestive tract derivation procedures: latero-lateral duodeno-duodenostomy, gastro-enterostomy and duodeno-jejunostomy on a Y-shaped excluded jejunal loop.

Adult

[Primary hyperaldosteronism with paroxysmal arterial hypertension. Apropos of 2 operated cases].

Primary hyperaldosteronism usually causes moderate hypertension. It is rare to note as in our two patients intermittent attacks of paroxysmal hypertension. The diagnosis of aldosteronism will be suspected on the finding of persistent hypokalemia with acidosis. It will be confirmed by laboratory examinations severe fall in plasma renin activity and rise in aldosterone in the adrenal veins. To determine the affected side, one may carry out adrenal phlebography which is a difficult technic, and/or a scan using iodine cholesterol which is benign and precise. Surgery with removal of the adenomatous hyperplasia in one case and of an adenoma in the other, gave one very good result.

Adrenal Glands

[Abdominal manifestations of hereditary angioneurotic edema. Importance of the exploration of the complement system (apropos of 29 families)].

Abdominal manifestations are almost constantly present (85% of cases) in the current form of hereditary Quincke's disease. In some cases, these abdominal manifestations occur even when cutaneomucosal edema is not present which leads to unwarranted often repeated and sometimes dangerous surgery. Apart from a story of heredity diagnosis of such troubles is possible, provided the total complement has been assayed to note its sharp fall. It can be subsequently explained by a functional defect of the C1 esterase inhibitor or alpha2-neuraminoglycoprotein.

Adolescent