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Biomedical subjects

J Guerrero

Publications and source records attributed to J Guerrero.

15 recordsLinked to original sources
PubMed1979

[Urea half-live as index of renal failure in hypertonic dehydration (author's transl)].

Evolution of uremic half-time in 27 infants with hypertonic dehydration is studied. Authors establish three groups in accordance with duration of uremic half-time: 1st: less than 24 hours: 5 infants. 2nd: More than 24 hours without renal or uremic pathology: 12 infants. 3rd: More than 24 hours with renal or uremic pathology: 10 infants. Utility of this parameter is discussed, as well as its physiologic bases. The existence of 2nd group makes the value of this control as an organic of functional renal index uncertain.

Acute Kidney Injury↗
PubMed1978

Experimental muscular amebiasis in hamsters as a biological model.

Trophozoites of Entamoeba histolytica maintained in vitro in Pavlova's medium were inoculated by deep intramuscular injection into the proximal left hindleg of hamsters. Thioglycollate medium was utilized as a successful vehicle to induce the infection. The invasion of the muscular tissue by the vegetative forms caused the formation of abscesses with great destruction of muscular fibers. The lesions were limited to the muscular tissue of the femoral area. The number of trophozoites, the medium of thioglycollate as a vehicle, the volume of the inoculum and the trauma caused by the needle were important elements in the evolution of the muscular amebic abscesses. A limited trial of the amebicidal activity of metronidazole utilizing the amebic intramuscular infection was also performed.

Abscess↗
PubMed1978

Normotensive hyperreninemia in systemic lupus erythematosus. An indicator of tubular dysfunction.

High plasma renin activity (PRA) was found in 16 of 42 randomly selected nonuremic systemic lupus erythematosus (SLE) patients. Mild hypertension was present in 3 of the 16.6 high-PRA and 10 normal-PRA patients were admitted to a metabolic ward. Salt restriction produced a disproportionate rise in both PRA and aldosterone, a decrease in glomerular filtration rate (GFR) and a slightly greater negative sodium balance in the group with high PRA. Potassium excretion was less than intake in both groups. Balance studies were performed in 6 additional high-PRA patients before and during indomethacin administration (150 mg/24 h). PRA and aldosterone were markedly suppressed by indomethacin. UnaV was significantly greater than in the control period despite of the 28% reduction in GFR. These results suggest that high PRA is secondary to impaired distal tubular sodium reabsorption. Such a defect could be responsible for the relatively low frequency of hypertension in lupus nephritis.

Adolescent↗
PubMed1978

[Pancreatic lipomatous hypoplasia associated with skeleton anomalies. Shwachman's syndrome (author's transl)].

A five month old girl with exocrine pancreatic insufficiency, skeletal deformities and a non constant neutropenia (Shwachman's syndrome) is presented. Infectious complications, mainly septic and pulmonary, which caused her death, were frequent. The pathologic findings showed a pancreatic lipomatous hypoplasia and absence of zymogen granulos, as well as anomalies of the growing cartilage where no radiological abnormalities could be seen.

Abnormalities, Multiple↗
PubMed1978

[Hyperglycemia in hypertonic dehydration (author's transl)].

Fourteen patients who developed hyperglycemia with hypertonic dehydration are reviewed, and compared to a control group of twenty-four dehydrated infants without hyperglycemia. The threshold of hyperglycemia was considered at 125 mg. %. The relationship between hyperglycemia and degree of formula concentration, incidence of shock and water and solute metabolism chemistry are analysed. The different theories about hyperglycemia in the course of hypertonic dehydration are discussed. Because of the negativity of other factors, authors suggest that the most important factor in hyperglycemia may be the ionic alteration of the cell.

Dehydration↗
PubMedDate not supplied

[Myocardial fibrosis in Shwachman's syndrome (author's transl)].

A case of a child affected by Shwachman's syndrome with a chronic diarrhea, cyclic neutropenia and bone defects is described. The child died at the age of eight months from an acute cardiac failure which showed analytical alterations compatible to an acute myocardial infarction. Pathologic study showed a pancreatic lipomatous hypoplasia, myocardial fibrosis and chondrometaphyseal dysplasia. Main hypothesis that relate pancreatic pathology and development of myocardial fibrosis are discussed, although there is a lack of definitive evidence for confirming such relation.

Bone Diseases, Developmental↗