Biomedical subjects

J H Doust

Publications and source records attributed to J H Doust.

At least 19 recordsLinked to original sources

PubMed2006

Oxygen uptake kinetics during supra VO2max treadmill running in humans.

Accurate classification of VO2 kinetics is essential to correctly interpret its control mechanisms. The purpose of this study was to examine VO2 kinetics in severe and supra-maximal intensity running exercise using two modelling techniques. Nine subjects (mean +/- S.D: age, 27 +/- 7 years; mass, 69.8 +/- 9.0 kg; VO2max, 59.1 +/- 1.8 mL x kg x min(-1)) performed a series of "square-wave" exercise transitions to exhaustion at running speeds equivalent to 80% of the difference between the VO2 at LT and VO2max (delta), and at 100%, 110% and 120% VO2max. The VO2 response was modelled with an exponential model and with a semi-logarithmic transformation, the latter assuming a certain steady state VO2. With the exponential model there was a significant reduction in the "gain" of the primary component in supra-maximal exercise (167 +/- 5 mL x kg(-1) x km(-1) at 80% delta to 142 +/- 5 mL x kg(-1) x km(-1) at 120% VO2max, p = 0.005). The time constant of the primary component also reduced significantly with increasing intensity (17.8 +/- 1.1 s at 80% delta to 12.5 +/- 1.2 s at 120% VO2max, p < 0.05). However, in contrast, using the semi-log model, the time constant significantly increased with intensity (30.9 +/- 13.5 s at 80% delta to 72.2 +/- 23.9 s at 120% VO2max, p < 0.05). Not withstanding the need for careful interpretation of mathematically modelled data, these results demonstrate that neither the gain nor the time constant of the VO2 primary component during treadmill running are invariant across the severe and supra-maximal exercise intensity domains when fit with an exponential model. This suggests the need for a reappraisal of the VO2/work rate relationship in running exercise.

Adult↗

PubMed2001

Effects of prior exercise and recovery duration on oxygen uptake kinetics during heavy exercise in humans.

Prior heavy exercise (above the lactate threshold, LT) reduces the amplitude of the pulmonary oxygen uptake (VO2) slow component during heavy exercise, yet the precise effect of prior heavy exercise on the phase II VO2 response remains to be established. This study was designed to test the hypotheses that (1) prior heavy exercise increases the amplitude of the phase II VO2 response independently of changes in the baseline VO2 value and (2) the effect of prior exercise depends on the amount of external work done during prior exercise, irrespective of the intensity of the prior exercise. Nine subjects performed two 6 min bouts of heavy cycling exercise separated by 6 min baseline pedalling recovery (A), two 6 min heavy exercise bouts separated by 12 min recovery (6 min rest and 6 min baseline pedalling, B), and a bout of moderate exercise (below the LT) in which the same amount of external work was performed as during the prior heavy exercise, followed by 6 min heavy exercise (C). In both tests A and B, prior heavy exercise significantly increased the absolute VO2 amplitude at the end of phase II (by approximately 150 ml x min(-1)), and reduced the amplitude of the VO2 slow component by a similar amount. Following 12 min of recovery (B), baseline VO2, but not blood [lactate], had returned to pre-exercise levels, indicating that these effects occurred independently of changes in baseline VO2. Prior moderate exercise (C) had no effect on either the VO2 or blood [lactate] responses to subsequent heavy exercise. The VO2 response to heavy exercise was therefore dependent on the intensity of prior exercise, and the effects on the amplitudes of the phase II and slow VO2 components persisted for at least 12 min following prior heavy exercise.

Adult↗

PubMed2001

Oxygen uptake kinetics during treadmill running in boys and men.

The purpose of this study was to compare the kinetics of the oxygen uptake (VO(2)) response of boys to men during treadmill running using a three-phase exponential modeling procedure. Eight boys (11-12 yr) and eight men (21-36 yr) completed an incremental treadmill test to determine lactate threshold (LT) and maximum VO(2). Subsequently, the subjects exercised for 6 min at two different running speeds corresponding to 80% of VO(2) at LT (moderate exercise) and 50% of the difference between VO(2) at LT and maximum VO(2) (heavy exercise). For moderate exercise, the time constant for the primary response was not significantly different between boys [10.2 +/- 1.0 (SE) s] and men (14.7 +/- 2.8 s). The gain of the primary response was significantly greater in boys than men (239.1 +/- 7.5 vs. 167.7 +/- 5.4 ml. kg(-1). km(-1); P < 0.05). For heavy exercise, the VO(2) on-kinetics were significantly faster in boys than men (primary response time constant = 14.9 +/- 1.1 vs. 19.0 +/- 1.6 s; P < 0.05), and the primary gain was significantly greater in boys than men (209.8 +/- 4.3 vs. 167.2 +/- 4.6 ml. kg(-1). km(-1); P < 0.05). The amplitude of the VO(2) slow component was significantly smaller in boys than men (19 +/- 19 vs. 289 +/- 40 ml/min; P < 0.05). The VO(2) responses at the onset of moderate and heavy treadmill exercise are different between boys and men, with a tendency for boys to have faster on-kinetics and a greater initial increase in VO(2) for a given increase in running speed.

Adult↗

PubMed2000

Evaluation of the reliability and validity of a soccer-specific field test of repeated sprint ability.

The reliability and validity of a soccer-specific field test of repeated sprint ability was assessed. Seven male games players performed the repeated sprint test on six separate occasions. The temporal pattern of the mean sprint time was analysed by using coefficient of variation with confidence intervals (CI), and repeated measures ANOVA. A within subject mean coefficient of variation of 1.8% (95% CI, 1.5-2.4) was found for performance in the repeated sprint test across all six trials. The mean coefficient of variation across trials 2-4 was found to be 1.9% (95% CI, 1.3-3.1), compared to trials 4-6, where it was 1.4% (95% CI, 1.0-2.3). The ANOVA showed that a significant difference was present between the trials (F6,30 9.8. P<0.001). A Tukey post-hoc test showed that significant differences were present between trial I and trials 3-6, and trial 2 and trial 5. The learning effect was complete by trial 3. Performance in the repeated sprint test was compared to total running time averaged from two repeats of the maximal anaerobic running test laboratory protocol. Mean sprint time in the repeated sprint test and total running time in the laboratory protocol had a correlation coefficient of r = -0.298 (P = 0.516, n = 7), suggesting that the energetics of the two tests are not closely related. In conclusion, this soccer-specific field test demonstrated high reliability.

Adult↗

PubMed2000

Time to exhaustion during severe intensity running: response following a single bout of interval training.

The primary aim of this study was to examine any change in performance caused by a fatiguing interval training session (TS). A secondary aim of this study was to examine the change in oxygen uptake (VO(2)) during moderate and severe intensity running, and the relationship with the change in performance. Seven male runners [mean age 24 (SD 6) years, height 1.79 (SD 0.06) m, body mass 67.9 (SD 7.6) kg, maximal oxygen uptake (VO(2max)) 4.14 (SD 0.49) l. min(-1)] were studied. The VO(2) during moderate and severe intensity running and running performance were studied immediately prior to, 1 h following, and 72 h following TS. The TS was performed on a treadmill, and consisted of six bouts of 800 m at 1 km. h(-1) below the velocity at VO(2max) (v(V)O(2max)), with 3-min rest intervals. Performance was also assessed at 1 km. h(-1) below v(V)O(2max), in the form of time to exhaustion (t(lim)). The VO(2) and heart rate (f(c)) were assessed both during the severe intensity performance trial, and the moderate intensity run at 50% v(V)O(2max). Whilst a significant change was observed in running performance and the VO(2) during both moderate and severe intensity running prior to and following TS, no relationship was observed between the magnitude of change in these variables. At 1 h following TS, t(lim) had decreased by 24%, VO(2) during moderate intensity running had increased by 2%, and the difference in VO(2) between 2 min 45 s and the end of severe intensity running had increased by 91% compared with values recorded prior to TS. At 1 h following TS, f(c) had also increased significantly during moderate intensity running by 5% compared to the value recorded prior to TS. These findings demonstrated that TS resulted in a reduction in performance, and that the relationship between running performance and VO(2) during running may be altered under conditions of prolonged fatigue.

Adult↗

PubMed2000

Changes in blood lactate and pyruvate concentrations and the lactate-to-pyruvate ratio during the lactate minimum speed test.

The aim of this study was to assess the responses of blood lactate and pyruvate during the lactate minimum speed test. Ten participants (5 males, 5 females; mean +/- s: age 27.1+/-6.7 years, VO2max 52.0+/-7.9 ml x kg(-1) x min(-1)) completed: (1) the lactate minimum speed test, which involved supramaximal sprint exercise to invoke a metabolic acidosis before the completion of an incremental treadmill test (this results in a 'U-shaped' blood lactate profile with the lactate minimum speed being defined as the minimum point on the curve); (2) a standard incremental exercise test without prior sprint exercise for determination of the lactate threshold; and (3) the sprint exercise followed by a passive recovery. The lactate minimum speed (12.0+/-1.4 km x h(-1)) was significantly slower than running speed at the lactate threshold (12.4+/-1.7 km x h(-1)) (P < 0.05), but there were no significant differences in VO2, heart rate or blood lactate concentration between the lactate minimum speed and running speed at the lactate threshold. During the standard incremental test, blood lactate and the lactate-to-pyruvate ratio increased above baseline values at the same time, with pyruvate increasing above baseline at a higher running speed. The rate of lactate, but not pyruvate, disappearance was increased during exercising recovery (early stages of the lactate minimum speed incremental test) compared with passive recovery. This caused the lactate-to-pyruvate ratio to fall during the early stages of the lactate minimum speed test, to reach a minimum point at a running speed that coincided with the lactate minimum speed and that was similar to the point at which the lactate-to-pyruvate ratio increased above baseline in the standard incremental test. Although these results suggest that the mechanism for blood lactate accumulation at the lactate minimum speed and the lactate threshold may be the same, disruption to normal submaximal exercise metabolism as a result of the preceding sprint exercise, including a three- to five-fold elevation of plasma pyruvate concentration, makes it difficult to interpret the blood lactate response to the lactate minimum speed test. Caution should be exercised in the use of this test for the assessment of endurance capacity.

Adaptation, Physiological↗

PubMed2000

Changes in cycling efficiency and performance after endurance exercise.

PURPOSE: This study was designed to examine the effects of moderate-intensity endurance exercise on cycling performance, gross efficiency, and 30-s sprint power output. METHODS: Two separate experiments were conducted. After a controlled warm-up, subjects completed as much work as possible in a 5-min performance test (EXP1) or a maximal 30-s sprint test (EXP2). These initial exercise bouts were followed by approximately 60 min of cycling at approximately 60% VO2peak or an equivalent period of rest (control) before repeating the warm-up exercise and either the 5-min performance or 30-s sprint test. Expired gas for calculation of cycling gross efficiency was collected over the last minute of each warm-up period. RESULTS: Average 5-min performance power output was significantly reduced (12 W) after exercise in EXP1, and in EXP2 both peak and mean power output were significantly lower (26 and 35 W, respectively). Gross efficiency decreased significantly with exercise in both EXP1 and EXP2. Moreover, the change in gross efficiency was correlated with the change in 5-min performance (r = 0.91, P < 0.01), but not with the change in mean or peak 30-s sprint power output. CONCLUSIONS: After sustained moderate-intensity cycling significant reductions in 5-min performance, gross efficiency and sprint power output were observed in endurance trained cyclists. The reduction in 5-min performance was related to the exercise induced decrease in gross efficiency.

Bicycling↗

PubMed2000

Oxygen uptake kinetics in treadmill running and cycle ergometry: a comparison.

The purpose of the present study was to comprehensively examine oxygen consumption (VO(2)) kinetics during running and cycling through mathematical modeling of the breath-by-breath gas exchange responses to moderate and heavy exercise. After determination of the lactate threshold (LT) and maximal oxygen consumption (VO(2 max)) in both cycling and running exercise, seven subjects (age 26.6 +/- 5.1 yr) completed a series of "square-wave" rest-to-exercise transitions at running speeds and cycling power outputs that corresponded to 80% LT and 25, 50, and 75%Delta (Delta being the difference between LT and VO(2 max)). VO(2) responses were fit with either a two- ( LT) exponential model. The parameters of the VO(2) kinetic response were similar between exercise modes, except for the VO(2) slow component, which was significantly (P < 0.05) greater for cycling than for running at 50 and 75%Delta (334 +/- 183 and 430 +/- 159 ml/min vs. 205 +/- 84 and 302 +/- 154 ml/min, respectively). We speculate that the differences between the modes are related to the higher intramuscular tension development in heavy cycle exercise and the higher eccentric exercise component in running. This may cause a relatively greater recruitment of the less efficient type II muscle fibers in cycling.

Adult↗

PubMed2000

Effects of prior heavy exercise on phase II pulmonary oxygen uptake kinetics during heavy exercise.

We tested the hypothesis that heavy-exercise phase II oxygen uptake (VO(2)) kinetics could be speeded by prior heavy exercise. Ten subjects performed four protocols involving 6-min exercise bouts on a cycle ergometer separated by 6 min of recovery: 1) moderate followed by moderate exercise; 2) moderate followed by heavy exercise; 3) heavy followed by moderate exercise; and 4) heavy followed by heavy exercise. The VO(2) responses were modeled using two (moderate exercise) or three (heavy exercise) independent exponential terms. Neither moderate- nor heavy-intensity exercise had an effect on the VO(2) kinetic response to subsequent moderate exercise. Although heavy-intensity exercise significantly reduced the mean response time in the second heavy exercise bout (from 65.2 +/- 4.1 to 47.0 +/- 3.1 s; P < 0.05), it had no significant effect on either the amplitude or the time constant (from 23.9 +/- 1.9 to 25.3 +/- 2.9 s) of the VO(2) response in phase II. Instead, this "speeding" was due to a significant reduction in the amplitude of the VO(2) slow component. These results suggest phase II VO(2) kinetics are not speeded by prior heavy exercise.

Adult↗

PubMed2000

Effect of endurance training on oxygen uptake kinetics during treadmill running.

The purpose of this study was to examine the effect of endurance training on oxygen uptake (VO(2)) kinetics during moderate [below the lactate threshold (LT)] and heavy (above LT) treadmill running. Twenty-three healthy physical education students undertook 6 wk of endurance training that involved continuous and interval running training 3-5 days per week for 20-30 min per session. Before and after the training program, the subjects performed an incremental treadmill test to exhaustion for determination of the LT and the VO(2 max) and a series of 6-min square-wave transitions from rest to running speeds calculated to require 80% of the LT and 50% of the difference between LT and maximal VO(2). The training program caused small (3-4%) but significant increases in LT and maximal VO(2) (P<0.05). The VO(2) kinetics for moderate exercise were not significantly affected by training. For heavy exercise, the time constant and amplitude of the fast component were not significantly affected by training, but the amplitude of the VO(2) slow component was significantly reduced from 321+/-32 to 217+/-23 ml/min (P<0.05). The reduction in the slow component was not significantly correlated to the reduction in blood lactate concentration (r = 0. 39). Although the reduction in the slow component was significantly related to the reduction in minute ventilation (r = 0.46; P<0.05), it was calculated that only 9-14% of the slow component could be attributed to the change in minute ventilation. We conclude that the VO(2) slow component during treadmill running can be attenuated with a short-term program of endurance running training.

Adult↗

PubMed1999

Oxygen uptake during high-intensity running: response following a single bout of interval training.

Elevated oxygen uptake (VO2) during moderate-intensity running following a bout of interval running training has been studied previously. To further investigate this phenomenon, the VO2 response to high-intensity exercise was examined following a bout of interval running. Well-trained endurance runners were split into an experimental group [maximum oxygen uptake, VO2max 4.73 (0.39)l x min(-1)] and a reliability group [VO2max 4.77 (0.26)l x min(-1)]. The experimental group completed a training session (4 x 800 m at 1 km x h(-1) below speed at VO2max, with 3 min rest between each 800-m interval). Five minutes prior to, and 1 h following the training session, subjects completed 6 min 30 s of constant speed, high-intensity running designed to elicit 40% delta (where delta is the difference between VO2 at ventilatory threshold and VO2max; tests 1 and 2, respectively). The slow component of VO2 kinetics was quantified as the difference between the VO2 at 6 min and the VO2 at 3 min of exercise, i.e. deltaVO2(6-3). The deltaVO2(-3) was the same in two identical conditions in the reliability group [mean (SD): 0.30 (0.10)l x min(-1) vs 0.32 (0.13)l x min(-1)]. In the experimental group, the magnitude of the slow component of VO2 kinetics was increased in test 2 compared with test 1 by 24.9% [0.27 (0.14)l x min(-1) vs 0.34 (0.08)l x min(-1), P < 0.05]. The increase in deltaVO2(6-3) in the experimental group was observed in the absence of any significant change in body mass, core temperature or blood lactate concentration, either at the start or end of tests 1 or 2. It is concluded that similar mechanisms may be responsible for the slow component of VO2 kinetics and for the fatigue following the training session. It has been suggested previously that this mechanism may be linked primarily to changes within the active limb, with the recruitment of alternative and/or additional less efficient fibres.

Adult↗

PubMed1999

Effect of 6 weeks of endurance training on the lactate minimum speed.

The aim of this study was to assess the sensitivity of the lactate minimum speed test to changes in endurance fitness resulting from a 6 week training intervention. Sixteen participants (mean +/- s: age 23+/-4 years; body mass 69.7+/-9.1 kg) completed 6 weeks of endurance training. Another eight participants (age 23+/-4 years; body mass 72.7+/-12.5 kg) acted as non-training controls. Before and after the training intervention, all participants completed: (1) a standard multi-stage treadmill test for the assessment of VO2max, running speed at the lactate threshold and running speed at a reference blood lactate concentration of 3 mmol x l(-1); and (2) the lactate minimum speed test, which involved two supramaximal exercise bouts and an 8 min walking recovery period to increase blood lactate concentration before the completion of an incremental treadmill test. Additionally, a subgroup of eight participants from the training intervention completed a series of constant-speed runs for determination of running speed at the maximal lactate steady state. The test protocols were identical before and after the 6 week intervention. The control group showed no significant changes in VO2max, running speed at the lactate threshold, running speed at a blood lactate concentration of 3 mmol x l(-1) or the lactate minimum speed. In the training group, there was a significant increase in VO2max (from 47.9+/-8.4 to 52.2+/-2.7 ml x kg(-1) x min(-1)), running speed at the maximal lactate steady state (from 13.3+/-1.7 to 13.9+/-1.6 km x h(-1)), running speed at the lactate threshold (from 11.2+/-1.8 to 11.9+/-1.8 km x h(-1)) and running speed at a blood lactate concentration of 3 mmol x l(-1) (from 12.5+/-2.2 to 13.2+/-2.1 km x h(-1)) (all P < 0.05). Despite these clear improvements in aerobic fitness, there was no significant difference in lactate minimum speed after the training intervention (from 11.0+/-0.7 to 10.9+/-1.7 km x h(-1)). The results demonstrate that the lactate minimum speed, when assessed using the same exercise protocol before and after 6 weeks of aerobic exercise training, is not sensitive to changes in endurance capacity.

Adult↗

PubMed1999

Effect of incremental test protocol on the lactate minimum speed.

PURPOSE: The purpose of this study was to investigate the effect of altering the initial running speed (RS) in the incremental portion of the lactate minimum test on the lactate minimum speed (LMS). METHODS: Eight well-trained endurance runners (mean +/- SD age 29.0 +/- 5.4 yr, body mass 72.0 +/- 5.6 kg, VO2max 63.1 +/- 3.8 mL x kg(-1) min(-1)) completed a standard incremental treadmill test for the assessment of the lactate threshold (LT) and VO2max, and eight lactate minimum tests. Following a period of supramaximal exercise, subjects were allowed 8 min of recovery to allow blood [lactate] to peak. Subjects then undertook eight randomly-assigned incremental treadmill tests from different initial running speeds (3.0, 2.5, 2.0, 1.5, 1.0, and 0.5 km x h(-1) below the predetermined RS-LT, at the RS-LT, and at 1.0 km x h(-1) above the RS-LT) with RS increased by 1.0 km x h(-1) every 5 min until volitional fatigue. Blood samples for the determination of blood [lactate] were taken at the end of each stage and the LMS was determined by fitting a spline function to the data. RESULTS: No LMS could be determined for the two highest initial RS conditions. For the other conditions, the LMS was significantly affected by the initial RS used in the incremental test and varied from 13.8 +/- 0.7 km x h(-1) with an initial RS of 3.0 km x h(-1) below the RS-LT, to 15.8 +/- 0.8 km x h(-1) with an initial RS of 0.5 km x h(-1) below the RS-LT. The LMS was significantly different from the RS-LT (15.4 +/- 0.8 km x h(-1)) (P < 0.05), except when the incremental test started at 1.0 or 1.5 km x h(-1) below the RS-LT. CONCLUSIONS: These results suggest that the LMS test is not a valid method for estimation of the LT since it is profoundly influenced by the starting speed selected for the incremental portion of the test.

Adult↗

PubMed1999

A disproportionate increase in VO2 coincident with lactate threshold during treadmill exercise.

PURPOSE: The purpose of this study was to assess the relationship between pulmonary VO2 and running speed over a range of exercise intensities. During constant-load cycle exercise above the lactate threshold (Tlac), it has been shown that VO2 does not attain a steady state within 3 min but continues to rise until either a delayed but elevated steady-state VO2 is attained or exhaustion occurs. Since this greater oxygen cost of exercise (V02 slow component) has only been demonstrated at discrete exercise intensities above Tlac, it was hypothesised that the onset of the VO2 slow component would coincide with Tlac during an incremental test if the stage durations were of sufficient length. METHODS: Five male subjects (mean +/- SD age 31 +/- 2 yr: VO2peak 60.1 +/- 5.8 mL x kg(-1) x min(-1)) performed four identical treadmill tests within an 8-d period. The tests involved the completion of six stages of 7-min duration. Running speed was increased by 0.5 km x h(-1) between stages. In the first test, fingertip capillary blood was sampled at the end of each stage for determination of Tlac. For all tests expired air was collected into Douglas bags from 3.0 to 3.75 min and from 6.0 to 6.75 min of each stage to determine any increase in V02 (deltaVO2) over the duration of the stage. RESULTS: The mean deltaVO2 for each stage over the four tests was determined for each subject. Repeated measures ANOVA with post-hoc Tukey tests revealed a significant increase in deltaVO2 at running speeds above, but not below, Tlac. CONCLUSIONS: The results of this study confirm the close association between the VO2 slow component and the onset of lactic acidosis and demonstrate alinearity in the VO2-exercise intensity relationship above Tlac for incremental treadmill exercise.

Adult↗

PubMed1998

Oxygen uptake during moderate intensity running: response following a single bout of interval training.

Eight male endurance runners [mean+/-(SD): age 25 (6) years; height 1.79 (0.06) m; body mass 70.5 (6.0) kg; % body fat 12.5 (3.2); maximal oxygen consumption (VO2max 62.9 (1.7) ml x kg(-1) x min(-1)] performed an interval training session, preceded immediately by test 1, followed after 1 h by test 2, and after 72 h by test 3. The training session was six 800-m intervals at 1 km x h(-1) below the velocity achieved at VO2max with 3 min of recovery between each interval. Tests 1, 2 and 3 were identical, and included collection of expired gas, measurement of ventilatory frequency (fr), heart rate (Fc)r rate of perceived exertion (RPE), and blood lactate concentration ([La-]B) during the final 5 min of 15 min of running at 50% of the velocity achieved at V02max (50% v-VO2max). Oxygen uptake (VO2), ventilation (VE), and respiratory exchange ratio (R) were subsequently determined from duplicate expired gas collections. Body mass and plasma volume changes were measured preceding and immediately following the training session, and before tests 1-3. Subjects ingested water immediately following the training session, the volume of which was determined from the loss of body mass during the session. Repeated measures analysis of variance with multiple comparison (Tukey) was used to test differences between results. No significant differences in body mass or plasma volume existed between the three test stages, indicating that the differences recorded for the measured parameters could not be attributed to changes in body mass or plasma volume between tests, and that rehydration after the interval training session was successful. A significant (P < 0.05) increase was found from test 1 to test 2 [mean (SD)] for VO2 [2.128 (0.147) to 2.200 (0.140) l x min(-1)], fc [125 (17) to 132 (16) beats x min(-1)], and RPE [9 (2) to 11 (2)]. A significant (P < 0.05) decrease was found for submaximal R [0.89 (0.03) to 0.85 (0.04)]. These results suggest that alterations in VO2 during moderate-intensity, constant-velocity running do occur following heavy-intensity endurance running training, and that this is due to factors in addition to changed substrate metabolism towards greater fat utilisation, which could explain only 31% of the increase in VO2.

Adult↗

PubMed1998

The validity of the lactate minimum test for determination of the maximal lactate steady state.

PURPOSE: The purpose of this study was to investigate the validity of the lactate minimum test ([Lac-]BMIN) in the determination of the velocity at the maximal lactate steady state (V-MLSS), and to identify those physiological factors most closely associated with 8-km running performance. METHODS: Thirteen trained male runners (VO2max range 53-67 mL.kg-1.min-1) took part in an 8-km simulated race on flat roads and completed a comprehensive battery of laboratory tests. RESULTS: Performance velocity was most strongly correlated with the estimated running velocity at VO2max (r = 0.93) and with V-MLSS (r = 0.92) and velocity at lactate threshold (V-Tlac) (r= 0.93). The running velocity at the ventilatory threshold (V-Tvent) (r = 0.81) and the [Lac-]BMIN (r = 0.83) also produced good correlations with performance velocity. Performance running velocity (mean +/- SEM 16.0 +/- 0.3 km.h-1) was not significantly different from V-MLSS (15.7 +/- 0.3 km.h-1). The running velocity at [Lac-]BMIN (14.9 +/- 0.2 km.h-1) was not significantly different from the V-Tlac (15.1 +/- 0.3 km.h-1) or V-Tvent (14.9 +/- 0.2 km.h-1) was not significantly different from the V-Tlac (15.1 +/- 0.3 km.h-1) or V-Tvent (14.9 +/- 0.3 km.h-1) but was significantly lower than the V-MLSS (P < 0.05). The [Lac-]BMIN provided the lowest correlation with V-MLSS (r = 0.61) and the worst estimate of V-MLSS (SEE = 0.75 km.h-1) compared with the other measures of lactate accumulation. The V-Tlac was not significantly different from V-MLSS and provided the highest correlation (r = 0.94) and a close estimate (SEE = 0.33 km.h-1) of the V-MLSS. CONCLUSIONS: It is concluded that of the measures studied relating to blood lactate accumulation during submaximal exercise, V-Tlac provides the best estimate of the V-MLSS and the V-Tlac had equal predictive power for 8-km race performance.

Adult↗

PubMed1997

The Conconi test in not valid for estimation of the lactate turnpoint in runners.

Conconi et al. (1982) reported that an observed deviation from linearity in the heart rate-running velocity relationship determined during a field test in runners coincided with the 'lactate threshold'. The aim of this study was to assess the validity of the original Conconi test using conventional incremental and constant-load laboratory protocols. Fourteen trained male distance runners (mean +/-s: age 22.6 +/- 3.4 years; body mass 67.6 +/- 4.8 kg; peak VO2 66.3 +/- 4.7 ml kg-1 min-1) performed a standard multi-stage test for determination of lactate turnpoint and a Conconi test on a motorized treadmill. A deviation from linearity in heart rate was observed in nine subjects. Significant differences were found to exist between running velocity at the lactate turnpoint (4.39 +/- 0.20 m s-1) and at deviation from linear heart rate (5.08 +/- 0.25 m s-1) (P < 0.01), and between heart rate at the lactate turnpoint (172 +/- 10 beats min-1) and at deviation from linearity (186 +/- 9 beats min-1) (P < 0.01). When deviation of heart rate from linearity was evident, it occurred at a systematically higher intensity than the lactate turnpoint and at approximately 95% of maximum heart rate. These results were confirmed by the physiological responses of seven subjects, who performed two constant-velocity treadmill runs at 0.14 m s-1 below the running velocity at the lactate turnpoint and that at which the heart rate deviated from linearity. For the lactate turnpoint trial, the prescribed 30 min exercise period was completed by all runners (terminal blood lactate concentration of 2.4 +/- 0.5 mM), while the duration attained in the trial for which heart rate deviated from linearity was 15.9 +/- 6.7 min (terminal blood lactate concentration of 8.1 +/- 1.8 mM). We concluded that the Conconi test is invalid for the non-invasive determination of the lactate turnpoint and that the deviation of heart rate from linearity represents the start of the plateau at maximal heart rate, the expression of which is dependent upon the specifics of the Conconi test protocol.

Adult↗

PubMed1996

A 1% treadmill grade most accurately reflects the energetic cost of outdoor running.

When running indoors on a treadmill, the lack of air resistance results in a lower energy cost compared with running outdoors at the same velocity. A slight incline of the treadmill gradient can be used to increase the energy cost in compensation. The aim of this study was to determine the treadmill gradient that most accurately reflects the energy cost of outdoor running. Nine trained male runners, thoroughly habituated to treadmill running, ran for 6 min at six different velocities (2.92, 3.33, 3.75, 4.17, 4.58 and 5.0 m s-1) with 6 min recovery between runs. This routine was repeated six times, five times on a treadmill set at different grades (0%, 0%, 1%, 2%, 3%) and once outdoors along a level road. Duplicate collections of expired air were taken during the final 2 min of each run to determine oxygen consumption. The repeatability of the methodology was confirmed by high correlations (r = 0.99) and non-significant differences between the duplicate expired air collections and between the repeated runs at 0% grade. The relationship between oxygen uptake (VO2) and velocity for each grade was highly linear (r > 0.99). At the two lowest velocities, VO2 during road running was not significantly different from treadmill running at 0% or 1% grade, but was significantly less than 2% and 3% grade. For 3.75 m s-1, the VO2 during road running was significantly different from treadmill running at 0%, 2% and 3% grades but not from 1% grade. For 4.17 and 4.58 m s-1, the VO2 during road running was not significantly different from that at 1% or 2% grade but was significantly greater than 0% grade and significantly less than 3% grade. At 5.0 m s-1, the VO2 for road running fell between the VO2 value for 1% and 2% grade treadmill running but was not significantly different from any of the treadmill grade conditions. This study demonstrates equality of the energetic cost of treadmill and outdoor running with the use of a 1% treadmill grade over a duration of approximately 5 min and at velocities between 2.92 and 5.0 m s-1.

Adult↗