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Biomedical subjects

J J Feldmeyer

Publications and source records attributed to J J Feldmeyer.

11 recordsLinked to original sources
PubMed2000

[Cervical myelopathy: diagnostic problems].

Non traumatic cervical myelopathy is a common disease. Spondylosis with or without discopathy is a major cause aside meningioma or myelitis. Most cases can get some benefit from neurosurgery. But in current practice the diagnosis encounters difficulties because of insidious course and non specific initial symptoms, so that we can think the disease remains a long time unknown. The review of 18 patients allows me to recall the criteria of diagnosis and to confirm the importance of neurological examination: thanks to it we can better focus on complementary exams (MRI, lumbar puncture, electrophysiology) and if necessary go round behind a deceptive radiologic "evidence".

Adult↗
PubMed1990

[Diabetic neuropathy].

Neuropathy is a frequent late complication of diabetes. The severity and duration of hyperglycaemia are probably the principal causal factor. The consequences of the effects of neuropathy on the lower-limb and the autonomic nervous system are major causes of morbidity. Apart from glycaemic control, no specific treatment is yet available, but measures for symptomatic alleviation of certain painful and visceral manifestations of diabetic neuropathy exist and are outlined in these guidelines. The guidelines also describe simple diagnostic strategies for detecting potentially threatening neurological signs, notably reduction or loss of lower-limb sensation which expose the patient to the risk of ulceration, unnoticed trauma and amputation. The guidelines also summarize the preventive educational measures required to minimize these risks.

Diabetic Neuropathies↗
PubMed1984

[Unilateral or bilateral asterixis in cases of thalamic or parietal lesions: an afferent motor disorder?].

Four cases are described in which asterixis was the main symptom of a focal brain lesion: controlateral asterixis from thalamic hemorrhage or infarction, and bilateral asterixis from posterior parietal hematoma or glioblastoma. The features of this symptom and the localization of the causal lesions make it possible to consider asterixis as a focal and transitory afferent motor dysfunction.

Aged↗
PubMed1984

Painful ataxic hemiparesis.

Right hemiparesis with right-sided pain and ataxia developed in a 68-year-old man. Sensation, neuropsychological function, and somatosensory evoked potentials were normal. Computed tomography showed an isolated fresh infarct in the left part of the thalamus. The pain and ataxic disturbances were related to involvement of the thalamus itself, but the hemiparesis with hyperactive tendon reflexes and Babinski's sign was probably due to associated dysfunction in the adjacent internal capsule from compression or edema. In the available clinicopathological reports of cases with hemiparesis and thalamic infarction, contiguous involvement of the internal capsule or no associated lesion has been reported. Because of the occurrence of pain, which is not present in pontine, mesencephalic, or capsular ataxic hemiparesis, we suggest that the syndrome seen in our patient be called "painful ataxic hemiparesis."

Ataxia↗
PubMed1983

[Symptomatic stenoses of the middle cerebral artery].

The clinical angiographic and prognostic features of 13 patients with symptomatic stenosis of the middle cerebral artery (MCA) admitted between 1970 and 1981 have been reviewed. The etiology of the stenosis was probably atherosclerosis, except in 1 case due to fibro-muscular dysplasia, 1 with post-radiation angiopathy and 2 related to oral contraceptives. Two main clinical groups were delineated: 1) transitory and minor ischemic accidents always arising in the same arterial area; these occurred in older patients with multiples atheromatous lesions along the carotid artery. 2) progressive--or stepwise--strokes occurring in younger patients, mainly females, with isolated MCA stenosis. These cerebrovascular symptoms and signs, in the absence of carotid bruit, Doppler ultrasonography abnormality or evidence of cardiac embolism should suggest intracranial stenosis and require appropriate therapy to avoid complete occlusion. Our study shows that early medical treatment--anti-coagulation, then antiaggregation--provides a suitable alternative to EC/IC bypass. This is suggested by evidence for micro-embolization in addition to hemodynamic mechanisms.

Adult↗
PubMed1980

[Acute alcoholic polyneuropathies as an unusual clinical and electromyographic entity].

The clinical and EMG aspects of a particular form of polyneuropathy encountered in seven young alcoholics are presented. This form is characterized by an acute, severe and extensive paralysis with motor, sensitive and vegetative deficit. The reasons of the sudden aggravation of a previous chronic PN are thiamine deficiency -- wich realise the BERI-BERI with Wernicke-Korsakoff encephalopathy -- and a probable poisoning due to the multiples toxics included in the alcoholised beverages. The remarkable clinical features of this PN are: the quickness of the onset, the extension and the severity of the paralysis, the young age of the patients and the relative prevalence of the women -- these two points suggesting a possible congenital predisposition -- and the fast improvement under vitamino-therapy and abstinence. Our EMG findings, in accord with some recent electro-physiological studies with histological confrontations, show up the indirect signs of a massive axonal degeneration in contrast to a mild demyelination. The EMG examen was a good contribution to exclude other possibilities like Guillain-Barré syndrome or alcoholic myopathy, and to follow the evolution.

Acute Disease↗