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Biomedical subjects

J J van Overbeeke

Publications and source records attributed to J J van Overbeeke.

30 records · Page 2Linked to original sources
PubMed1997

Charles Labbé (1851-1889).

The vein of Labbé is a very important structure and every neurosurgeon is acquainted with its anatomy. Because of the recent increasing interest and experience in skull base surgery, the vein of Labbé has received a great deal of attention. Intraoperative damage to this vein should be avoided and several methods to prevent this have been described. Despite these developments, nothing is written in the neurosurgical literature about the man who described this vein for the first time: Charles Labbé. The authors therefore conducted an extensive search of the literature and uncovered several public records in France to learn more about Charles Labbé.

France↗
PubMed1996

Intracranial nerve repair: a review of experimental and clinical studies.

With recent developments in neurosurgery and related disciplines, more aggressive approaches are being made for various lesions of the skull base, and, as a consequence, cranial nerves are more frequently damaged, which causes significant morbidity. The authors review experimental and clinical studies involving surgical repair of severed cranial nerves and provide evidence that some degree of functional regeneration occurs. Functional recovery after repair is mainly dependent on the preoperative function of the muscle--nerve unit and the morphological organization of the nerve; the more complex the organization, the lesser the degree of functional recovery. The beneficial effect of surgical repair on postoperative morbidity is outlined together with suggestions for future research.

Journal Article↗
PubMed1996

The Dutch 'Benefit-II' project: do physicians benefit from using an electronic medical dossier?

In 1992 the Dutch College of General Practitioners (NHG) started a project, which lasted until May 1994. The project had three major objectives: establishing as exactly as possible what benefits we gain from using an Electronic Medical Dossier (EMD) can we find an influence on the quality of care? Which drawbacks or problems are there, advising on the optimal use of an EMD; What is the best place to record certain data? advising the Working Committee on Informatics (WCIA), of Dutch National Association of General Practitioners (LHV) and NHG on the directions for the new Reference Model 1995. To obtain these goals panel discussions and a questionnaire were used. The project resulted in advice on how to use the EMD in the most efficient way. Also we have learned about the use of GP-information systems and the perception about this usage by the Dutch GPs.

Computer Literacy↗
PubMed1996

Polyposis coli, craniofacial exostosis and astrocytoma: the concomitant occurrence of the Gardner's and Turcot syndromes.

BACKGROUND: Up to 60% of the patients with known adenomatous polyposis coli may present hyperostosis of the skull and facial bones, and/or a susceptibility to fibromas. This is known as the Gardner's syndrome, and is considered as an allelic variant of familial adenomatous polyposis (FAP). Also, although very rare, an adenomatous polyposis coli may occur with malignant tumors of the central nervous system, known as Turcot syndrome. If both syndromes are different phenotypic presentation of FAP, this would explain a simultaneous occurrence. METHOD: We report the history of a patient who showed clinical signs of the simultaneous occurrence of both Gardner's and Turcot syndromes. The syndromes are compared, and in view of the literature, a genetic explanation for the concomitant occurrence is discussed. RESULTS: Evidence obtained from the literature to consider Turcot syndrome as a phenotype of FAB is as follows: (1) The occurrence of Gardner's and Turcot syndromes in one family, but in different members; (2) The presence of congenital hypertrophic retinal pigmented epithelium (CHRPE), which correlates with the expression of polyps in FAP patients, in both syndromes; (3) Linkage of the Turcot phenotype to the adenomatous polyposis coli locus by genetic markers. Evidence obtained from this case report indicates that there is a manifestation of both syndromes in one patient together with a positive family history for FAP. CONCLUSION: This concomitant occurrence of both Gardner's and Turcot syndromes in one patient clinically supports genetic and ophthalmic investigation to consider Turcot syndrome (like Gardner's syndrome) as a phenotypic variant of FAP. Patients with FAP should be examined for the presence of Gardner's syndrome. In case a Gardner's syndrome is suspected, a computed tomography scan of the brain is recommended because of the possible existence of a simultaneous Turcot syndrome.

Adenomatous Polyposis Coli↗
PubMed1995

Anatomy of the sympathetic pathways in the cavernous sinus.

We studied sympathetic fibres in the cavernous sinus in 40 unfixed specimens obtained from human cadavers. Sympathetic fibres in the cavernous sinus are understood to be grouped in a plexiform configuration surrounding the internal carotid artery and have a diffuse distribution among the sympathetic nerves. Our study, however, suggests that a more systematic arrangement of sympathetic pathways exists in the cavernous sinus. A detailed anatomical description of intracavernous sympathetic fibres contributes to academic anatomical knowledge and may have practical applications for 1. diagnostic interpretation of pathologic conditions involving the cavernous sinus, 2. recognition and orientation of anatomical structures during intracavernous surgical procedures, and 3. a deeper understanding of the sympathetic nerve supply to cerebral vasculature.

Carotid Artery, Internal↗
PubMed1994

The arterial pattern at the base of arhinencephalic and holoprosencephalic brains.

The mechanisms by which the anatomical variations of the circle of Willis develop is considered to be related to haemodynamic factors, i.e. the differential growth of the various parts of the brain will continuously change the haemodynamic demands and consequently the flow patterns in the cerebral arteries. It is therefore to be expected that, if a selected part of the brain does not develop, the change in the haemodynamic demand will affect the development of some cerebral arteries. Consequently the arteries at the base of 2 arhinencephalic and 8 holoprosencephalic brains were studied in conjunction with the brain malformations. The defects of holoprosencephaly are believed to arise from a failure of the prosencephalon to separate fully into the telencephalon and diencephalon and become manifest at the time that the prosencephalon normally starts to separate into the hemispheres, i.e. 28-34 d p.c. Arhinencephalic brains are fully diverticulated. There is only a partial or complete agenesis of the olfactory tracts and bulbs. The defect causing arhinencephaly starts at 43 d p.c. In the arhinencephalic brains no particular vascular abnormalities were found. However, at the base of the holoprosencephalic brains no complete circle of Willis was present; the anterior part was lacking and was replaced by anterior branches which emerged unilaterally or bilaterally from the internal carotid artery. The choroidal arteries were of very large calibre and ran to the highly vascularised wall of the dorsal cyst which is usually present in holoprosencephalic brains. In contrast to the anterior part, the posterior arterial pattern was almost identical to the posterior part of the circle of Willis of normal brains. The basic vascular patterns found in the holoprosencephalic brains displayed the features of Padget's developmental stages 2 and 3 of the cerebral vasculature, i.e. the pattern that has normally developed within 28-40 d p.c. The further modification of this pattern could largely be understood from the functional demand imposed on the circulation by the enlarged anterior choroidal arteries. Because the development of the anterior part of the circle of Willis precedes the developmental derangement causing arhinencephaly, a complete circle was found in these brains.

Brain↗
PubMed1991

Surgical repair of a cranial meningocele in a calf.

A meningocele at the anterior fontanelle was diagnosed in a two-and-a-half month old Meuse-Rhine-Yssel calf. After radiographic and ultrasonographic evaluation, the meningocele was successfully repaired surgically.

Animals↗
PubMed1988

The cavernous sinus syndrome. An anatomical and clinical study.

The cavernous sinus is often involved pathologically, which can cause ocular motor nerve palsies with or without facial sensory disturbances. Consequently several clinical features of ocular motor nerve palsies have been described. In this article we present a study of the cavernous sinus syndrome, and compare this syndrome with other nerve palsy syndromes caused by lesions in or adjacent to the cavernous sinus. The clinical features are explained by means of an anatomical study of the cavernous sinus.

Cavernous Sinus↗