[Anetoderma--a clinical and therapeutic problem].
Current views on etiopathogenesis of anetoderma have been presented on the grounds of literature data. Disorders of elastin metabolism are distinctive for the disease causing elastin fibres becoming disintegrated and atrophied. Apart from mechanism of anetoderma plaques evolution clinical observation and full diagnostication of systemic collagenosis seems to be important. Despite of numerous therapeutic trials it appears that medication of anetoderma lesions is inefficacious.
Atrophy↗