Biomedical subjects

J Lindemuth

Publications and source records attributed to J Lindemuth.

3 recordsLinked to original sources

PubMed2004

Comment on "Mobility spectrum computational analysis using a maximum entropy approach".

We point out that the comparison in Fig. 1 of the recent publication by S. Kiatgamolchai et al. [Phys. Rev. E 66, 036705 (2002)] of the proposed maximum entropy-mobility spectrum analysis (ME-MSA) with our quantitative mobility spectrum analysis (QMSA) is misleading. Rather than comparing with the more recent "improved" version of QMSA [Vurgaftman et al., J. Appl. Phys. 84, 4966 (1998)], a preliminary version that was three years older and demonstrably inferior was employed. We show that ME-MSA and the improved QMSA give quite similar results.

Comment↗

PubMed1991

Variability of clinical dentin substrates.

Clinical trials remain the only conclusive research of dentin adhesive performance, but variables unique to the oral environment may influence the results. Dentin variability is one factor that may account for extreme variations in clinical evaluations. Despite the encouraging results of laboratory studies, clinical trials with adhesive systems have been disappointing. Current adhesives are reported to react with dentin surfaces by chemical and/or micro-mechanical mechanisms. Dentin surfaces, such as cervical abrasions, are subject to intraoral changes and dentin tubules commonly become obturated by the growth of peritubular dentin or by the precipitation of calcific deposits within the tubules. The resulting sclerotic dentin may be less receptive to current dentin adhesives. An examination of clinically aged dentin surfaces has revealed an inordinate variability in tubular morphology. The more sclerotic dentin present, the less effective was dentin conditioning and resin composite adaptation. Early correlations with current clinical trials appear to substantiate that the greatest failure of restorations occurred in sclerotic lesions.

Aging↗

PubMed1990

Polymeric adhesion to dentin: contrasting substrates.

Clinical research has shown that resin dentin adhesive materials often fail prematurely despite encouraging laboratory-derived data. Current adhesives are reported to react with dentin surfaces by chemical and/or micro-mechanical mechanisms. The ionic bonding to calcium and the covalent coupling to collagen are representative chemical strategies employed today. Recent findings suggest that the mechanical interlocking of resins into open dentinal tubules may play an even more important role with present adhesive systems. Dentin surfaces, such as cervical abrasions, undergo changes in the oral cavity and dentinal tubules may become partially or completely obturated by the growth of peritubular dentin or by the precipitation of mineral salts within the tubules. The resulting sclerotic dentin may be less receptive to current dentin adhesives. An examination of clinically aged dentin surface types revealed extreme variability in tubular morphology. The more sclerotic dentin presented, the less effective was dentin conditioning and resin adaptation. Early correlations with ongoing clinical trials appear to substantiate this finding with the greatest failure of restorations occurring in more sclerotic lesions.

Adhesives↗