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J Lusins

Publications and source records attributed to J Lusins.

18 recordsLinked to original sources

Reversible autonomic neuropathy and hypertrophic osteoarthropathy in a patient with bronchogenic carcinoma.

We report a case of adenocarcinoma of the lung with pandysautonomia and pulmonary osteoarthropathy. Surgical resection of the tumor followed by radiation therapy for the residual tumor resulted in complete resolution of arthropathy and almost complete recovery from autonomic dysfunction. The symptoms of arthropathy and autonomic neuropathy recurred simultaneously several months later with increase in the tumor mass as confirmed at second surgical exploration. The second thoracotomy revealed an inoperable tumor. Following the second thoracotomy, while the arthropathy was relieved, the autonomic neuropathy persisted. We concluded that autonomic neuropathy is related to the tumor mass, and pulmonary osteoarthropathy and autonomic neuropathy are probably caused by different mechanisms, as yet undefined.

Autonomic Nervous System Diseases

Alcoholism and cerebral atrophy: a study of 50 patients with CT scan and psychologic testing.

A study of 50 alcoholic patients was carried out with CT scans of their brains and psychologic testing. These patients were alcohol and drug free, and without neurologic or psychiatric disorders at the time of the examinations. Fifty-eight percent of the patients had cerebral atrophy. There was no correlation between the results of the psychologic testing and cerebral atrophy. The only factor that correlated to a significant degree with cerebral atrophy was the duration of problem drinking. Only the mean duration of problem drinking was statistically significantly longer in the patients with cerebral atrophy compared to those patients with normal CT scans. Implications of this finding of high prevalence of cerebral atrophy in alcoholics is discussed.

Adult

Computer assisted tomography of unoperated subdural hematoma: short- and long-term follow-up.

Six recent and six long-term follow-up patients with subdural hematoma studied by computer assisted tomography are reported. This method was used to demonstrate progressive resolution of the hematoma in five of the recent patients as well as enlargement of the hematoma in one of the patients who subsequently was sent to surgery. Computer assisted tomography was also used to demonstrate absence of subdural hematoma in long-term follow-up of unoperated patients. The possible difficulty in visualizing an isodense subdural hematoma is discussed, as is the use of additional coronal views to improve subdural detection.

Aged

Unoperated subdural hematomas. Long-term follow-up study by brain scan and electroencephalography.

The authors report nine patients selected from over 100 patients with subdural hematomas successfully treated without surgery. These patients were followed for as long as 5 years. All had angiographically demonstrated subdural hematomas. Electroencephalograms (EEG) documented well the clinical improvement of the patient, but were poor guides to the true size of the hematoma, since EEG returns to normal early in the patient's course. Static scans are a better guide to the presence of a subdural hematoma, but they lag behind clinical improvement and usually remain abnormal for considerable periods of time after a major portion of the hematoma has been reabsorbed, and the patient is asymptomatic.

Aged