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Biomedical subjects

J M Thiron

Publications and source records attributed to J M Thiron.

8 recordsLinked to original sources
PubMed1980

Variations in height of jugular "a" wave in relation to heart rate in normal subjects and in patients with atrial septal defect.

Analysis of jugular tracings in seven normal subjects in sinus rhythm whose heart rate varied spontaneously from one moment to another during expiratory apnoea, showed that a pronounced variation occurred in the size of the "a" waves in relation to that of the "v" waves and in accordance with the corresponding RR interval. In the 53 measurements which were carried out, the "a/v" ratio had a mean value of 0.9 for a heart rate above 87, 1.4 for a heart rate between 87 and 68, and 1.1 for a heart rate less than 68. The variations in the size of the "a" waves in relation to heart rate are explained by the place of atrial systole during the different phases of ventricular filling. This variation in the height of the "a" wave has also been found in patients with an atrial septal defect for heart rates between 62 and 86, and this could lead to misdiagnosis, either dismissing the diagnosis of atrial septal defect when the heart rate is around these values, or suspecting it wrongly when the heart rate is either slower or faster. When examining jugular tracings, one should therefore be cautious about interpreting the size of the "a" wave which should be evaluated as a function of heart rate.

Adolescent↗
PubMed1976

[Study of systolic time by external phonomechanocardiography in myocardial infarction. 1. Value of systolic times and their course during the 1st 3 weeks].

A phonomechanographic study was carried out on 38 patients with recent myocardial infarction both during the initial phase of necrosis (between the 5th and 7th day) and at the end of the third week. The parameters studied were electromechanical systole (QB2), systolic ejection time corrected for heart rate (CET), the OBl interval, isometric contraction (IC), the pre-ejection periode (PEP), and the haemodynamic coefficient (PEP/ET). The results were compared with the values found in a similar control series. A tendency towards lengthening of the pre-ejection times and shortening of the corrected ejection time was noted. These changes were significant for the second measurement of CET, and for the ratio PEP/ET during the initial phase and on second measurement. The pre-ejection times were generally increased more markedly in the group of patients with left ventricular failure; the ejection time was also markedly shorter in this group, and the haemodynamic coefficient was larger. There was a tendency for the various measurements studied to return to normal by the third week.

Heart Rate↗
PubMed1976

[Study of systolic times by external phonomechanocardiography in myocardial infarction. 2. Results of sensitizing tests: methoxamine, venous tourniquets, isomeric contration].

In an attempt to improve the quality of information provided by measuring the systolic times, the effect of three thpes of sensitising test was studied: the test with venous tourniquets on the lower limbs, the test with methoxamine, and the test under isometric exercise. In none of these tests was there found to be a statistically significant difference between the controls and the patients with myocardial infarction, with or without left ventricular failure. Taken as a whole, however, the sensitising tests studied tended to accentuate the abnormalities in the systolic times established under basal conditions in patients with myocardial infarction more grossly than in controls.

Exercise Test↗
PubMed1975

[Telesystolic mitral insufficiency with systolic click, mitral valve prolapse, and hypertrophic obstructive myocardiopathy].

There are several causes of the syndrome of "Late systolic murmur and mid-systolic click" due to mitral incompetence with ballooning of the valve, and, as numerous recent publications have shown, amongst them must be included abnormalities of left ventricular contraction. Seven cases have been studied by phonomechanocardiographic, cineangiographic, and haemodynamic methods, and analysis of the findings has shown that the abnormalities of contraction may be due to a cardiomyopathy, with a variable degree of left ventricular hypertrophy which is usually of patchy distribution, and leads to a certain degree of left intraventricular obstruction.

Cardiomegaly↗