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Biomedical subjects

J Mach

Publications and source records attributed to J Mach.

At least 19 recordsLinked to original sources
PubMed1990

[Patellar changes following knee joint endoprosthesis-plasty].

We analysed the postoperative position of the patella and the degenerative changes in 80 implanted Guepar, Tillmann and GSB-prostheses of the knee. We did not found differences between the types of endoprostheses. We saw many dislocations of the patella, especially in patients with rheumatism, but only some patients had trouble with the operated knee. We can diminish these problems by modelling of the patella and by careful reconstruction of the muscle strength.

Adult↗
PubMed1990

[The significance of concomitant internal diseases for hip endoprosthesis in elderly patients].

We studied the importance of internal diseases in 110 patients with an age of more than 70 years. Most of the patient (85%) were handicapped by several diseases with an increasing of the operative risk. Special importance has the prophylaxis of thromboembolism and the elimination of essential local infect. In this way it is possible to avoid essential postoperative complications. There is nearly no restriction of the older patients in reference to the endoprosthesis of the hip if we notice the risk factors and the optimal pre-, intra- and postoperative treatment of the patients.

Aged↗
PubMed1979

Effect of colicin E3 on leukemia cells P388 in vitro.

Proliferation of murine leukemia cells P388 is stimulated by less than 1.0 mg/ml colicin E3, while being inhibited by higher concentrations. By 1.6 mg/ml colicin E3, uptake of thymidine into cold TCA-precipitable fraction is decreased by 59% during 24 h, uptake of uridine by 29%.

Animals↗
PubMed1979

The cytoplasmic membrane as a site of the primary effect of colicins on eukaryotic cells.

The effect of colicins E3 and K on mouse lymphoid cells was studied. Low doses of colicin E3, which were not toxic, inhibited profoundly the Con A-induced activation of lymphocytes while the Con A binding capacity of the treated cells was retained. The inhibitory effect of colicin E3 was also found when the homing of 51Cr-labelled lymph node cells to the lymph nodes of syngeneic recipients was studied, while the rate of redistribution of H-2 antigens on the surface of the lymphocytes was not changed by colicin E3 or colicin K. The findings suggest that the cytoplasmic membrane is the site of the primary effect of colicin E3 on eukaryotic cells.

Animals↗