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Biomedical subjects

J Maess

Publications and source records attributed to J Maess.

At least 19 recordsLinked to original sources
PubMed1996

[Dorsal recumbency in dogs--a circulatory risk during surgical treatment?].

In this study, changes in hemodynamic, blood gas, and metabolic variables recorded during right lateral and dorsal recumbency in beagles anaesthetised with thiopental are presented. Other than reported in human beings, dorsal recumbency in these dogs resulted in an increase (33%) in heart rate, decrease (30%) in systolic, diastolic, and mean systemic arterial pressure, a decrease (17%) in systemic vascular resistance, and a decrease in both right (31%) and left (39%) ventricular work in comparison with lateral recumbency. Furthermore, mixed-venous PO2, oxygen saturation and respiratory quotient were lower in dorsal than in lateral recumbency while O2 consumption and lipolysis were increased in the former. The changes presented may have been caused by beta-adrenergic stimulation in dorsal recumbency. It needs to be studied if capillary perfusion can be maintained adequately during surgery in dorsal recumbency or if this predisposes to cardiovascular shock.

Anesthetics, Intravenous↗
PubMed1990

[Detection of infectious hemorrhagic disease of domestic rabbits (rabbit viral hemorrhagic disease) in wild rabbits (Oryctolagus cuniculus)].

The occurrence of Rabbit Viral Hemorrhagic Disease of domestic rabbits in the population of wild rabbits in Germany was proven through detection of antigen (hemagglutination) in spleen and liver in one animal and through detection of specific antibody in another animal. There was no epizootic connection between the two animals (different locations). The necropsy and histopathologic findings are presented from five wild rabbits which died from RVHD.

Animals↗
PubMed1984

Adenovirus pneumonia in guineapigs: an experimental reproduction of the disease.

The recently described virus-induced pneumonia in guineapigs (Naumann et al., 1981) was experimentally reproducible in newborn animals, though not in preadult animals. Baby hamsters and newborn rats were also not susceptible to infection. 10 of 11 infected newborn guineapigs developed pathological changes identical with those found in spontaneous cases. The incubation period was from 5 to 10 days. The agent could not be cultivated in vitro, and therefore no applicable serological tests could be established. The morphology of the virus, its intranuclear location, the course of the disease and the histopathological and ultrastructural changes strongly suggest that the virus is an adenovirus specific for guineapigs. The virus did not cross-react with human or fowl adenoviruses. It was ether resistant and non-oncogenic in baby rats and hamsters. During a 5-year period we registered a total of 51 spontaneous death cases diagnosed as adenovirus pneumonia in our experimental guineapigs, 4 from own breeding colony.

Adenoviridae Infections↗
PubMed1984

[Rapid serological diagnosis of parvovirus infection in dogs. Fluorescent serological demonstration of virus-specific IgM antibodies].

Rapid diagnostic assay described comprises examination of a single serum sample of the ill dog using indirect immunofluorescence test. The procedure lasts about two hours. The finding of virus-specific humoral IgM antibodies serves as evidence for an acute parvovirus infection. 84 dogs presented as patients with enteritis were examined with this method, 48 of them were seropositive for IgM antibodies.

Animals↗
PubMed1982

Post-mating non-infectious hydrometra in BALB/c: Bom mice.

Post-mating non-infectious hydrometra and hydrovagina of unknown aetiology, leading to a scrotum-like swelling of the perineum, was observed during an experiment on BALB/c: Bom mice. There was a closure of the vulva with accumulation of secretions of uterine glands under high pressure in both uterus and vagina. The mice were otherwise clinically healthy, gained body weight normally, but were incapable of breeding. The morbidity was approximately 1% and the disease could not be transmitted to other females.

Animals↗
PubMed1981

Lethal pneumonia in guinea pigs associated with a virus.

No bacteria were observed in an epizootic of lethal pneumonia in guinea pigs. Necrotic bronchitis and bronchiolitis with basophilic intranuclear inclusion bodies in bronchial epithelial cells were characteristic. Although adenovirus infection of guinea pigs has not previously been reported, histological findings paralleled those found in adenovirus infections of other animals including man. Virus particles found by electron-microscopical examination of the lung tissue closely resembled adenoviruses. The disease seemed to have a low contagiousness, a low morbidity (about 0.7%), but an acute course and a high mortality (100%).

Adenoviridae↗
PubMed1980

[Development of brain lipids in rats receiving a fat-free diet with respect to myelinization].

Wistar rats were fed during the whole time of gestation with a fat-free diet. The diet regime was continued for the pups and mothers after term up to day 56 post partum. Phospholipids, cholesterol, and cerebrosides were estimated in the fetal brain at the 22nd day of gestation and in the brains of the new born animals at the 14th and 56th day post partum. The molar ratios typical for mature of myeline were followed up. Fat-free diet caused a reduction of brain weight by 50 per cent in fetuses at day 22 of gestation if compared with control animals. At day 14 and 56 post partum, however, this differences has been adjusted. Within the main phase of myelinization (14th-56th day) the brains of the diet animals showed an increased accumulation of phospholipids, a lower rise in cholesterol, and a strongly depressed accumulation of cerebrosides if compared with the control group. The results support the view that the fat-free diet may lead to a persistence of early states in mature of the myeline with an enrichment in membrane structures corresponding to early myeline.

Aging↗
PubMed1979

[Effect of fat free diet during the last week of pregnancy upon the linoleic and arachidonic acid content of fetal organ lipids and placenta lipids of the rat].

Pregnant Wistar rats were fed a fatfree diet from day 16--22 of pregnancy. On day 22, the fatty acid components of cholesterol esters, triglycerides, free fatty acids and phospholipids of maternal (brain, muscle, serum, white adipose tissue, liver) and fetal (brain, carcass, serum, liver) tissues, including the placenta, were examined gaschromatographically for the participation of linoleic and arachidonic acid. In all fetal and maternal organs the linoleic acid levels in the fatty acid patterns were strongly reduced. The alterations nearly always involved all the lipid fractions of a tissue and were mostly equal within a tissue. The strongest decreases of linoleic acid occurred in the placenta, and the weakest, in the lipids of maternal muscle and maternal adipose tissue. The linoleic acid alterations were principally similar in fetal and the corresponding maternal tissues, while being less pronounced in case of maternal muscle. The participation of arachidonic acid in the fatty acid pattern is completely retained in the lipids of fetal organs, and is even enhanced in those of the placenta.

Animals↗