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J Ruiz Galiana

Publications and source records attributed to J Ruiz Galiana.

At least 19 recordsLinked to original sources
PubMed1997

[Clenbuterol poisoning. Clinical and analytical data on an outbreak in Móstoles, Madrid].

OBJECTIVE: To report the clinical manifestations and analytical findings in an epidemic outbreak of acute food poisoning with clenbuterol. MATERIALS AND METHODS: The clinical manifestations, physical examination findings and results of complementary tests are reported of fifteen patients performed by veal liver contaminated with clenbuterol. The clinical course of patients at 72 hours is reported. A quantitative measurement of clenbuterol in urine specimens from patients and in a veal liver specimen was performed by high pressure liquid chromatography (HPLC). RESULTS: The male/female distribution of patients was 7/8 respectively, with age ranging from 6 to 44 years. Symptoms appeared after 30 minutes to 2 hours of having ingested veal liver in 93% of cases. Patients presented at the Emergency Department with tremors, palpitations, anxiety, malaise, nausea, and pruritus as the most common complaints. On physical examination tachycardia was noted in 100% of cases. The analytical data included mild hypokaliemia (66%) and leukocytosis (28%). Only one patient required hospital admission on account of an hypertensive crisis. After 72 hours, 67% of patients were asymptomatic. The remaining patients had mild symptoms which included headache, myalgia, asthenia and anorexia. Serum potassium values returned to normality (p < 0.05). Urine measurements of clenbuterol were positive for all analyzed cases (50 +/- 42 ng/ml). The concentration of clenbuterol in a veal liver sample was 500 ppb. CONCLUSION: Clenbuterol poisoning should be suspected when symptoms of adrenergic hyperstimulation occur after the ingestion of meal, usually liver. Common symptoms include tachycardia and mild hypopotasemia. Diagnosis is confirmed by quantitative measurement of clenbuterol in urine. Most patients improve spontaneously shortly afterwards.

Adolescent↗
PubMed1997

[Plasma levels of vitamins A and E and the risk of acute myocardial infarct].

INTRODUCTION AND OBJECTIVE: The incidence of myocardial infarction in Spain is low in spite of the high prevalence of smoking and hypertension. Natural antioxidants as vitamin E from a diet rich in fruit, oils and vegetables may protect against the development of this disease. In this study we have examine plasma levels of vitamin A and E as risk factors in the development of myocardial infarction. STUDY DESIGN: 62 cases were selected among patients 30-70 years old admitted to the hospital with a first episode of myocardial infarction, or diagnosed by arteriography of coronary heart disease. 62 controls were selected among patients with minor surgical conditions, and free of coronary heart disease according to the Rose questionnaire and ECG. LABORATORY ANALYSIS: Plasma samples for vitamin assays were taken in the first 24 hours after admission (62 cases) Plasma was separated by centrifugation and stored at -80 degrees C until plasma vitamins were determined by HPLC. RESULTS: Lipid adjusted blood levels of vitamin E were lower in cases, mean: 1092.0 +/- standard deviation 165.8 micrograms/dl (mean +/- SD) than in controls (1220.1 +/- 274.8 micrograms/dl p = 0.002). The multivariate adjusted odds ratio between extreme quintiles of blood vitamin E were 0.06 (95% confidence intervals (CI): 0.01-0.5; p for trend 0.0027. Adjusted blood levels of vitamins A were also lower in cases 44.0 +/- 15.1 micrograms/dl than in controls: (57.5 +/- 15.2 micrograms/dl; p < 0.001). Adjusted odds ratio between extreme quintiles of blood vitamin A adjusted for confounding factors were 0.06 (95% CI: 0.01-0.36; p for trend < 0.00001). CONCLUSIONS: There is an association between acute of myocardial infarction and reduce blood levels of vitamin A and E, that is not explained by the decrement in blood lipids levels. Therefore low blood levels of fat soluble vitamins may be a risk factor for the development of this disease.

Adult↗
PubMed1997

[Spontaneous peritonitis in a cirrhotic patient with a cat: Pasteurella multocida infection of the ascitic fluid].

Spontaneous peritonitis due to Pasteurella multocida is exceptional. As far as we know only 11 other cases have been reported. We describe a 45 year old patient who presented with a spontaneous Pasteurella multocida peritonitis as the first complication of a previously undiagnosed cirrhosis. The patient used to play with his pet cat, not recalling having ever sustained any injury. Cultures of the cat's mouth grew the same strain of Pasteurella multocida than was found in the patient's ascitic fluid. The clinical findings of the previous cases, most of which were also related to non traumatic exposure to domestic animals, are here described. Pasteurella multocida in one potential agent in the cirrhotic patient presenting with spontaneous peritonitis, especially if in close contact with animals, cats being the most often carriers.

Animals↗
PubMed1993

["Nutrition": a program to facilitate the calculations of the composition of menus and diets].

The methodology used to build a software program to compute the nutrient composition of menus and diets is discussed. Topics as the variability of food composition, the scarcity of local food composition data, the bias that may be incurred in nutrient data of food consumption studies are extensively discussed and also the possible solution through the software program. All of these topics must be taken into account in epidemiological studies as well as in computing diet composition.

Databases, Factual↗
PubMed1991

[Chronic eosinophilic pneumonia. Presentation of 2 cases].

Two cases of patients with chronic eosinophilic pneumonia are presented. We highlight the possibility to obtain the diagnosis without lung biopsy, as well as the spectacular response to steroid treatment which has to be continued for a long period in order to avoid relapses.

Adult↗