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Biomedical subjects

J S Ruiz

Publications and source records attributed to J S Ruiz.

6 recordsLinked to original sources
PubMed1983

Observation on peritumoural oedema in meningioma. Part I: Distribution, spread and resolution of vasogenic oedema seen on computed tomography.

The distribution of oedema detected by computed tomography in 46% of 160 patients with cerebral meningiomas was studied. Oedema occurred most frequently in the centrum semiovale and deep white matter around the ventricular trigones and frontal horns, the distribution reflecting mainly the size of the interstitial spaces adjacent to the tumour. Other theoretically relevant factors such as hydrocephalus and cerebral atrophy did not influence oedema distribution. Cyst like spaces of various types were observed in 20% and their aetiology is discussed. Oedema usually resolved after surgery, but in at least 13% it persisted after 3 months. Atrophy related to tumour size and not to presence or extent of oedema. Increased oedema after surgery was never of vasogenic type and could nearly always be ascribed to a definable complication.

Brain Diseases↗
PubMed1983

Observations on peritumoral oedema in meningioma. Part II: Mechanisms of oedema production.

Meningiomas occurring in 160 patients were studied in relation to oedema production using computerised tomography. Statistically significant positive correlations with oedema production were found with tumours of large surface area, anterior parasagittal position, prominent vascularity, involving dural sinuses, and with short clinical history. Significant negative correlations were found with small tumour size and the presence of calcification. It is considered that hydromechanical factors predominate in oedema production with meningioma.

Blood-Brain Barrier↗
PubMedDate not supplied

Chaos theories and therapeutic commonalities among depression, Parkinson's disease, and cardiac arrhythmias.

This report reviews and compares all therapies that have shown efficacy in depression and Parkinson's disease, although some are not in current use and others are at the experimental stage. They include pharmacological modification of neurotransmitter pathways, electroconvulsive therapy (ECT), sleep deprivation, psychosurgery, electrical stimulation through cerebral electrodes, and transcranial magnetic stimulation. Stemming from a pathophysiological model that stresses the brain as an open, complex, and nonlinear system, all therapies have been attributed a common mechanism of action. This report suggests that the therapeutic isomorphism is related to their ability to help the CNS deactivate cortical-subcortical circuits that are dysfunctionally coupled. These circuits are self-organized among neurons of their informational subsystem (rapid conduction) and modulating subsystem (slow conduction). Finally, this report extends the analysis and comparison of these therapies to some cardiac arrhythmias.

Arrhythmias, Cardiac↗
PubMedDate not supplied

CT changes associated with migraine.

In order to determine further the correlation between severe migraine and cerebral atrophy, a group of patients attending either the Charing Cross Hospital migraine clinic or outpatient clinics at the National Hospital, Queen Square, was investigated by computed tomography. The results confirm an association between atrophy and migraine of more than 5 years duration, probably more severe in men than women. The most striking changes were seen in those men who suffered from attacks lasting less than 6 hr.

Adolescent↗