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Biomedical subjects

J S van der Kamp

Publications and source records attributed to J S van der Kamp.

13 recordsLinked to original sources
PubMed1990

Four sporadic cases of congenital swinepox.

Four sporadic cases of congenital swinepox are described. The pigs were born with pox lesions over their entire bodies. Microscopic examination revealed ballooning degeneration of keratinocytes, cytoplasmic inclusions and intranuclear vacuolisation. Electron microscopy detected poxvirus particles in all four pigs. The virus was isolated from one pig and identified by HindIII restriction endonuclease digestion as swinepox virus.

Animals↗
PubMed1986

An outbreak of a herpesvirus infection in harbor seals (Phoca vitulina).

During an outbreak of a herpesvirus infection in juvenile harbor seals, 11 out of 23 seals died. The duration of the disease in these 11 animals varied from 1-6 days. Nasal discharge, inflammation of the oral mucosa, vomiting, diarrhea and fever up to 40 degrees C were observed in the first days of the disease. In later stages coughing, anorexia and lethargy occurred. Severe necrosis of the liver and interstitial pneumonia were the most striking histopathological findings.

Animals↗
PubMed1985

[One man's meat is another man's poison].

A case of rumen acidosis in non-lactating cattle is reported. This was due to the fact that stale bread was fed ad lib, rather than concentrates. Of seventy animals, fourteen died or were slaughtered. The meat was declared unfit for human consumption in every case.

Acidosis↗
PubMed1981

[An uncommon cause of colic (author's transl)].

A rare case of colic in an eleven-year-old New Forrest mare is reported. As treatment was ineffective, the animal had to be slaughtered. Post mortem examination showed that the colic was due to bilateral haemorrhagic corpus luteum, from which blood (appr. 20 liter) entered the abdominal cavity.

Animals↗
PubMed1979

[Congenital tricuspid atresia in a premature foal (author's transl)].

A case of tricuspid atresia in a premature foal associated with large atrial septal and ventricular septal defects is reported. This anomaly is compared with other cases in horses as reported in the literature. As tricuspid atresia in horses is usually not diagnosed so that relatively few cases have been reported, a classification similar to that of human anomalies is not possible. It is suggested that the present malformation is due to abnormal development of the atrioventricular canal. The factors involved in the pathogenesis of this abnormal development are obscure.

Animals↗