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J Tollqvist

Publications and source records attributed to J Tollqvist.

4 recordsLinked to original sources

Does long-term concrete work cause silicosis?

The machining of cast concrete often produces heavy exposure to respirable silica dust. Work with concrete in general results in considerably less exposure, however, and only a few cases of silicosis due to work with concrete have been reported. Telephone interviews with detailed questions on occupational history were made with 271 construction workers specializing in concrete work for more than 20 years, and exposure data were evaluated for different work tasks. The mean cumulative exposure of subjects to silica was estimated to be 2.4 mg.years.m-3. Chest radiographs were classified according to the International Labour Office classification. There was no suspicion of silicosis for any of the workers. The risk of contracting silicosis from work with concrete in the Swedish construction industry in recent decades is considered to be slight and does not justify general, periodic chest radiographic screening.

Construction Materials↗

Pleural plaques and lung function in construction workers exposed to asbestos.

Respiratory symptoms and pulmonary function have been evaluated in construction workers exposed to asbestos and in control subjects. Group I displayed pleural plaques but not lung tissue involvement on X-ray, group II had the same history of asbestos exposure as the previous group but had no pleural plaques, nor any lung tissue involvement, and group III constituted non-exposed control subjects. Chronic bronchitis and productive cough were 4-5 times more frequent in group I compared to group III, while non-productive cough was rare. Conventional spirometry gave no significant differences between exposed and non-exposed subjects, while expiratory flow rates during the latter half of the expiration (MEF50, MEF25) were reduced in group I. Closing volume (CV) was markedly increased in group I and the transfer factor of the lung for CO was slightly reduced. The static transpulmonary pressure - lung volume curve was much the same for all three groups. The difference in CV, MEF50, and MEF25 was greater between exposed and non-exposed non-smokers than between exposed and non-exposed smokers. No significant differences were noticed between those in group II and the control group III. The findings indicate that asbestos exposure whcih elicits pleural plaques, may cause pulmonary dysfunction, representing a disease of the small airways.

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