HYPERBARIC OXYGENATION. FACTORS INFLUENCING CLINICAL OXYGEN TOXICITY.
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Adrenal Glands↗
Biomedical subjects
J W BEAN
Publications and source records attributed to J W BEAN.
Tris buffer, CO2, and sympatho-adrenal system in reactions to O2 at high pressure.
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Adrenal Glands↗
Cerebral O2 in exposures to O2 at atmospheric and higher pressure, and influence of CO2.
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Brain↗
Brain blood flow, chlorpromazine (thorazine) and its protective action against the toxicity of O2 at high pressure.
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Brain↗
Compensable occupational diseases under the North Carolina Workmen's Compensation Act.
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Humans↗
Influence of rhythmic and tonic contraction of intestinal muscle on blood flow and blood reservoir capacity in dog intestine.
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Animals↗
Intestinal blood flow as influenced by vascular and motor reactions to acetylcholine and carbon dioxide.
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Acetylcholine↗
Effect of sympathetic blocking agents on the toxic action of O2 at high pressure.
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Humans↗
Effects of low O2 on intestinal blood flow, tonus and motility.
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Hemodynamics↗
Reserpine, chlorpromazine and the hypothalamus in reactions to oxygen at high pressure.
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Chlorpromazine↗
Somatic reflexes and blood oxygenation under positive intrapulmonic pressure.
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Cell Respiration↗
Epinephrine and neurogenic factors in the pulmonary edema and CNS reactions induced by O2 at high pressure.
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Adrenal Glands↗
Adrenocortical response to single and repeated exposure to oxygen at high pressure.
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Adrenal Cortex↗
Intestinal blood flow in curarization.
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Cardiovascular System↗
Cardiac changes induced by O2, at high pressure, CO2 and low O2, as manifest by the electrocardiogram.
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Carbon Dioxide↗
The hypophysis as a determinant in the reaction of the mammal to oxygen at high pressure.
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Animals↗
Influence of hypophysis on pulmonary injury induced by exposure to oxygen at high pressure and by Pneumococcus.
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Lung↗
Intestine in reflex chemical control of breathing.
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Intestines↗