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Biomedical subjects

J W Garvey

Publications and source records attributed to J W Garvey.

12 recordsLinked to original sources
PubMed1978

The surgical management of patients with carcinoma of the midthoracic esophagus.

Clinical and pathological findings in 15 patients with carcinoma of the midthoracic esophagus were analyzed. The author concluded that preoperative radiotherapy reduces tumor size and improves resectability. Longer follow-up is required to determine whether or not preoperative radiotherapy improves survival rates in these patients.

Adult↗
PubMed1977

Postoperative nutritional support using needle catheter feeding jejunostomy.

Needle catheter jejunostomy was used as an adjunctive surgical procedure in 110 patients. In 19 patients (or 17%) the jejunostomy was of value for the administration of post-operative nutritional support using an elemental diet and it may serve as an alternative route for the administration of supplementing fluids and electrolytes if intestinal function is intact. The clinical experience with the catheter jejunostomy establishes it as a satisfactory technique for postoperative nutritional support in patients requiring esophageal and proximal gastric resection and repair, and gastric surgery in the elderly and debilitated. It is also useful in patients undergoing complicated biliary, pancreatic, and duodenal surgery in whom anastomotic difficulties are anticipated.

Adult↗
PubMed1976

Haemorrhagic pulmonary oedema: post-pulmonary embolectomy.

A case of haemorrhagic pulmonary oedema after successful pulmonary embolectomy is presented. The relevant literature is reviewed. Thirteen cases are analysed as well as the four survivors. The aetiology appears to be ischaemic damage of the capillary bed. This had previously been called incomplete infarction by Castleman. The incidence is low after acute pulmonary embolectomies but appears to be much higher after chronic endarterectomies, especially with severe pulmonary hypertension. Therapy is outlined.

Adult↗
PubMed1975

Pathologic pulmonary changes in hemorrhagic shock.

Fifty-seven dogs were subjected to hemorrhagic hypotension by a variety of protocols. Histologic pulmonary changes were studied using the light microscope. Of these 57 dogs, 21 had no demonstrable lesions, 8 had minimal changes, and 28 had moderate or severe lesions, all of a focal nature. No correlation was found between the presence of lesions and mean systemic arterial pressure during shock, the udration of the hemorrhagic period, the fate of the animal, preoperative hematocrits and blood volumes, mean postreinfusion arterial pressure, whether the animals were mongrels or purebred beagles, whether they were awake or sedated, whether they breathed spontaneously or were artifically ventilated, whether they had undergone previous splenectomy or not, whether hilar stripping was performed or not, and finally, whether blood was reinfused after hemorrhage or not. Thus we conclude that multiple factors may exert a harmful effect on the lung in hemorrhagic shock, and that shock probably makes the lungs more vulnerable to other injurious agents rather than there being one single pathogenetic mechanism for the pulmonary damage. The term "adult respiratory distress syndrome" rather than "shock lung" is best used for the human clinical entity since it implies a complex ettiology rather than a discrete pulmonary lesion produced by a single pathogenetic mechanism.

Animals↗