Gigantic left atrial thrombus.
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Atrial Fibrillation↗
Biomedical subjects
Jadwiga Nessler
Publications and source records attributed to Jadwiga Nessler.
[Cardiac tamponade as the first symptom of oesophageal carcinoma -- a case report].
We present a case of a 56-year-old male who was admitted to our hospital due to cardiac tamponade. A 600 ml of purulent fluid was evacuated from the pericardium. The patient received antibiotics, however, due to recurrent pericardial effusion a pericardial drainage was required. Esophagoscopy and computerised tomography revealed oesophageal carcinoma with oesophago-pericardial fistula.
Antineoplastic Combined Chemotherapy Protocols↗
[Cardiac arrest in a 17-year-old boy--a case report].
Cardiac arrest (CA) refers to abrupt cessation of cardiac pump function. Most sudden deaths in young people are of cardiac origin, at the same time most patients have unrecognised prior heart disease. We report a case of a 17-year-old boy with cardiac arrest induced by ventricular fibrillation. Sinus rhythm was restored after one hour's resuscitation. Based upon elevated necrotic markers, ECG changes and echocardiographic examinations the diagnosis of anterolateral myocardial infarction was established. Coronary angiography revealed only a small myocardial bridge. The patient in a poor general and unconscious state was transferred to the Coronary Care Unit of the Department of Coronary Disease. The patient improved after treatment and without neurological deficit or circulatory insufficiency continued cardiac rehabilitation in a spa hospital. The paper reviews differential diagnosis of cardiac arrest, treatment modalities and describes the course of hospitalisation.
Adolescent↗
[Variant angina pectoris associated with Morgagni-Adams-Stokes syndrome--case report].
We report a case of Prinzmetal angina initially manifested with short losses of consciousness in a 55-year-old man hospitalized in the Department of Coronary Artery Disease, Institute of Cardiology, Jagiellonian University Medical College in Cracow. Clinical symptomatology of the presented case, causes and mechanism of loss of consciousness in variant angina as well as treatment methods are discussed.
Adams-Stokes Syndrome↗
Is magnesium deficit in lymphocytes a part of the mitral valve prolapse syndrome?
Magnesium deficiency has been proposed to be related to the mitral valve prolapse syndrome. However determinations of magnesium concentration in blood serum and hemolysates of red blood cells in this disease revealed divergent results. We investigated 49 patients with the mitral valve prolapse syndrome and 30 healthy individuals, matched by age and gender. The concentration of magnesium was measured in blood plasma and lysates of lymphocytes isolated from venous blood. The magnesium concentration in plasma was similar in both the patients and the healthy controls. By contrast, the magnesium concentration in lysates of lymphocytes was significantly lower (p < 0.03) in the patients with mitral valve prolapse syndrome than in the controls (range: 1.94-10.6 mmol/g of protein in Lymphocyte lysate; median 5.98 and 6.7-9.61 mmol/g of protein; median 7.22). The results obtained suggest that magnesium deficiency may be part of the mechanisms of the mitral valve prolapse syndrome.
Adolescent↗
C-reactive protein as a marker of heart failure.
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Adult↗
[Role of physical training in cardiac rehabilitation in patients with congestive heart failure].
Patients with stable heart failure show decreased exercise tolerance and consequently quality of life. The reduced physical capacity in these patients is a result of decreased strength of skeletal muscles and even their atrophy. The physical activity is limited by metabolic disorders and histological changes in myocytes. Dysfunction of respiratory muscles plays a role in the development of exertional dyspnea. Patients with stable heart failure are recommended to perform low or moderate physical training, which improves exercise tolerance. Intensive physical training in patients with reduced left ventricular ejection fraction improves maximal oxygen consumption through increased left ventricular stroke and increased arteriovenous difference. Recent studies indicate that regular physical training has a positive physiological effect on exercise tolerance, and decreases dyspnea and fatigue, in many patients with heart failure.
Dyspnea↗
[Diastolic dysfunction as a risk factor in patients with heart failure].
In the recent decade diastolic dysfunction has been extensively studied. It has been found that the clinical syndrome of heart failure may result from two entirely different disorders--systolic and diastolic impairment or both. Available evidence indicates that the left ventricular filling pattern is altered with progressing diastolic dysfunction. Left ventricular diastolic dysfunction is a sensitive marker of myocardial damage and it can be used to monitor a spectrum of changes ranging from impaired relaxation through pseudo-normalization to restrictive filling pattern. The restrictive filling pattern, irrespective of its etiology, has a negative effect on prognosis, especially when it coincides with reduced left ventricular ejection fraction. Patients with systolic heart failure and restrictive filling pattern show a lower exercise tolerance than their counterparts with non-restrictive filling. This indicates a significant role of diastolic dysfunction in the development of clinical symptoms in patients with left ventricular systolic impairment. Progress in the understanding of diastolic dysfunction may allow for better evaluation of prognosis and therapeutic efficacy and for monitoring of patients with systolic dysfunction.
Diastole↗
[Acute coronary syndrome or perimyocarditis?--case report].
Acute coronary syndrome or perimyocarditis? A case report. A case of a 25-year-old male with a severe chest pain associated with ST segment changes, elevated serum C-reactive protein, myocardial necrosis markers and normal epicardial coronary arteries is presented. The patient recently recovered from upper respiratory infection. Differential diagnosis between acute coronary syndrome and perimyocarditis is discussed.
Adult↗
[Multiple thrombo-embolic episodes in a patient with alcoholic cardiomyopathy - a case report].
A case of 42-year-old male with alcoholic dilated cardiomyopathy is presented. The patient was admitted to the hospital due to new onset heart failure and neurological symptoms due to systemic emboli. Transesophageal echocardiography revealed the presence of left atrial appendage thrombi which were the most probable cause of thrombo-embolic complications.
Adrenergic beta-Antagonists↗
[The modern view on the role of inflammatory factors in chronic heart failure and possibilities of pharmacological modification].
Chronic heart failure (CHF) is the state of reciprocal influences of neurohormonal factors and inflammatory markers. Many years of research and observations proved a particular meaning of cytokines and acute phase proteins in the pathophysiology of CHF. These markers are responsible for the repairing processes. There is a correlation between the activity of inflammatory factors, their plasma concentrations and clinical state of the patient. Basing on the cytokine's concentrations, the level of C-Reactive Protein (CRP) in the patient's blood and their changes in time we can predict the progression of the disease. Known medical references emphasise the fact that the pharmacological modification of inflammatory markers (interleukins, TNF-alfa, CRP) can have significant clinical consequences and in some cases can even cause regression of pathological symptoms. CRP plays an important role in the development and progression CHF and can be the clinical predictor not only in the cases of coronary disease but also can be useful in the group of patients with CHF. The introduction of drugs influencing CRP, interleukins and TNF-alfa levels to CHF therapy can be an effective solution called 'a golden therapy' improving clinical state and quality of patient's life.
Adrenergic beta-Antagonists↗