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James Mah

Publications and source records attributed to James Mah.

4 recordsLinked to original sources
PubMed2003

Three-dimensional relationship between the critical contact angle and the torque angle.

The purpose of this study was to analyze the relationship between the critical contact angle and the torque angle in an orthodontic bracket and archwire assembly in 3 dimensions. Three-dimensional mathematical models were created with geometric bracket-archwire parameters that included 2 slot sizes, 3 bracket widths, and 3 to 4 wire sizes. From this, 3-dimensional mathematical equations (3DMEs) for the critical contact angle and the maximum torque that result in critical contact angles of 0 were derived and calculated. To evaluate the effects of archwire-bracket parameters on critical contact angles, analysis of variance (ANOVA) was performed at the significance level of P < or = .05. For all bracket-archwire combinations, the critical contact angle decreased as bracket width, torque angle, and wire size increased. Therefore, all bracket-archwire parameters except slot height had an effect on the critical contact angle. Results of the critical contact angle produced from our 3DMEs were the same as those produced by 3D computer-aided design (SolidWorks Corp, Concord, Mass), thus confirming the validity of our derived equations. In addition, the effect of a beveled edge was investigated in some archwires. Furthermore, torsional play angles were calculated and found to be similar to those in previous reports. The results of this study provide theoretic and experimental bases for clinical orthodontic practice and indicate that torque angles should be included in the evaluation of the critical contact angle.

Analysis of Variance↗
PubMed2002

Natural variation in the extent of phosphorylation of bone phosphoproteins as a function of in vivo new bone formation induced by demineralized bone matrix in soft tissue and bony environments.

Implants of allogenic demineralized bone matrix were placed in distinct in vivo environments, i.e. calvarial (bony) and subcutaneous (soft tissue) sites. Detailed analyses of the biochemical components were performed. Quantitative levels of osteopontin (OPN), bone sialoprotein (BSP) and calcium phosphate (Ca-P) deposition within each implant environment varied as a function of new bone formation, and were substantially different in samples from calvarial and subcutaneous sites. Quantification of the extent of phosphorylation of affinity-purified OPN and BSP from such implants indicated that: (i) the number of mols of phosphoserine (P-Ser)/mol of affinity-purified OPN or BSP varied as a function of implant time and bone formation within both implant sites, and (ii) the 'effective P-Ser concentration' provided by the total OPN and BSP within each implant site varied and increased as a function of time, being approx. 5-fold higher for BSP in calvarial compared with subcutaneous implants. Peak levels of mols of P-Ser/mol of BSP coincided with maximum rates of Ca-P deposition in calvarial implants. Levels of OPN phosphorylation from both calvarial and subcutaneous implants also indicated fluctuations as a function of bone formation. Hence the present study, for the first time, provides direct evidence of natural variation in the extent of phosphorylation of both OPN and BSP as a function of time of mineralized tissue formation. Further evaluation of the data provides the first evidence of a direct and linear relationship between the rate of Ca-P deposition and the ratio of P-Ser-BSP/P-Ser-OPN for calvarial implants. Data for subcutaneous implants failed to provide such correlation. Overall, the present work demonstrates that the natural biological progression of the process of biomineralization follows strict criteria consistent with the anatomical location. Biomineralization fails to proceed in the same way in a soft tissue environment.

Animals↗