PubMed Health⌕ Search

Biomedical subjects

K Debek

Publications and source records attributed to K Debek.

8 recordsLinked to original sources
PubMed1998

[Amniotic fluid disease: a case report].

A rare case of amniotic disease with extensive malformations of fetus was described. A data obtained from literature was done with special consideration of etiopathology.

Adult↗
PubMed1995

Activation of the peritoneal mast cells and eosinophils in untreated hemorrhagic shock in rats.

The mast cells and eosinophils washed out from rat peritoneal cavity after 75 minutes of untreated hemorrhagic shock were studied in light and electron microscope. A significantly increased total peritoneal cell (PC) number, an increased mast cell (MC) number, decreased eosinophil number, and features of activation of both MC and E were found: heterogeneity and combining of granules, evacuation of their content, build up and enlargement of endoplasmic reticulum, development of microtubular cytoplasmic system, multiplication of cell membrane microvilli, and contacts between MC and E. Ultrastructural examination revealed vesicle formation in Golgie apparatus. These vesicles created characteristic blebs on the cell surface and evacuated their contents outside. Morphological findings suggest that peritoneal mast cells and eosinophils are involved in mechanism of the intestinal injury in shock.

Animals↗
PubMed1992

[TAR syndrome (congenital thrombocytopenia and aplasia of the radial bones) in a 5-months-old boy].

A boy aged 5 months had the TAR syndrome (thrombocytopenia and aplasia of radial bones) of autosomal recessive inheritance. Bilateral reduction of the length of the upper extremities included both forearm bones and humerus, so that the shoulder girdle was connected directly to the hand, with all fingers and thumb normally shaped. These skeletal anomalies were associated with thrombocytopenia. Various other abnormalities were present, with the most controversial anomaly being bilateral cleft of the lip and secondary palate. It may be an accidental coexistence of abnormalities or may suggest a common mechanism of the development of the TAR syndrome, Roberts syndrome and SC-phocomelia belonging to hereditary reduction of the extremities with identical mode of inheritance.

Abnormalities, Multiple↗