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K Kamiyama

Publications and source records attributed to K Kamiyama.

At least 91 records · Page 5Linked to original sources
PubMed1983

[Subdural hematoma due to metastatic dural carcinomatosis associated with DIC--a case report].

This 74-year-old female suddenly complained of severe headache, nausea, vomiting and dizziness on June 19, 1981. She was brought to nearby hospital. During the following six days, the state of consciousness gradually worsened and left-sided hemiparesis and convulsion attack arose and she was admitted to our clinic on June 25, 1981. Cerebral angiograms revealed an aneurysm of the right middle cerebral artery. Diagnosis of subarachnoid hemorrhage due to the rupture of an aneurysm was tentatively made and conservative therapy was done. On the second hospital day, she had nasal bleeding and began to excrete tar-like stool. Laboratory examination revealed thrombocytopenia, increase of FDP and prolongation of prothrombin time. Her liver and renal functions gradually worsened after this episode. On the 13th hospital day, she expired. General autopsy showed wide spread adenocarcinoma with metastases to the lung, lymph nodes and bones. Examination of the head revealed an unruptured aneurysm and bilateral diffuse subdural clotted hemorrhage. The dura was tightly adherent to the skull and partially thickened. No abnormal findings were found in the brain. On microscopical examination of the dura, there were fresh hemorrhage and many of the innumerable dilated small vessels contained tumor in the inner dural layer. Even by extensive examination, the origin of the malignancy could not be identified. We concluded that the initial symptoms just like of subarachnoid hemorrhage were due to the dural metastasis and subdural hematoma. Sixteen cases of subdural hematoma secondary to metastatic neoplasm were reported previously. We made some discussion about the pathogenesis and symptomatology of this type of subdural hematoma.

Adenocarcinoma↗
PubMed1983

[Simplified microdetermination of cerebral phospholipase A1, A2 and lysophopholipase].

The purpose of our study was to examine the ischemia induced enzymatic changes of decaylation-reacylation cycle of membrane phospholipids in dog brain. In this study, we developed new modified method for assay of phospholipase A1, A2 and lysophospholipase which is simpler and needs only a smaller amount of materials. For the first report, we introduced this new method and demonstrated some properties of phospholipase A1, A2 and lysophospholipase in dog brain. Crude enzyme solution for assays of phospholipase A1, A2 and lysophospholipase was gained from extraction of frozen brain with aceton, butanol and saline. The level of phosphorus in the enzyme extract was determined and only those extracts which had a level of phosphorus within a certain range were used. The substrates for assays were L-alpha-[beta-palmitoyl-1-14C] phosphatidylcholine, dipalmitoyl for phospholipase A1 and A2 and L-lysophosphatidylcholine-1-[1-14C] palmitoyl for lysophospolipase respectively. Each radioactive substrates was diluted with cold carrier lipid to give the proper specific activity. Reaction system including substrate, buffer [pH 7.0] and enzyme extract was incubated for 10 hours at 38 degrees C. But for the assay of phospholipase A1 and A2, enzyme solution was pre-incubated at 70 degrees C for 5 minutes. In our new method, reaction mixture was directly separated by TLC without extracting lipids. Enzyme activities were calculated from radio thin-layer chromatograms. Furthermore, we made a comparison between our method and the former one. The value of each enzyme activity was slightly higher in our method than in the former one. However, it was revealed that the results were reproducible in both methods.

Animals↗
PubMed1983

[Copper needle insertion for the treatment of spontaneous carotid cavernous fistula].

Electrothrombosis by copper needle insertion was applied to three cases of spontaneous carotid cavernous fistula. These cases had been observed conservatively for about six months or more, but their symptoms did not improve. On angiogram, the feeding arteries were branches of both the ipsilateral internal and the external carotid artery. Frontotemporal craniotomy was performed and copper needles were inserted transdurally into the cavernous sinus. The closure of fistula was verified by intra-operative angiography in all cases and their symptoms improved after operation. However, postoperatively one case had transient third nerve palsy. Another case had Gerstmann syndrome due to narrowing of the internal carotid artery by one needle and the preoperative six nerve palsy became worse. In one case, the procedure could be uneventfully performed. Based on our experiences, several problems of this technique were discussed in this paper.

Arteriovenous Fistula↗
PubMed1982

[Orbital neurilemmoma originated from the supraorbital nerve: case report].

The patient, 41 years old, female, was admitted to our hospital with the chief complaint of the progressively increasing painless swelling of the right eye. On examination, the right eye slightly protruded compared with the left and upward movement of the right eye was limited. Skull x-ray film (posteroanterior) showed a dome-shaped expansion in the roof of the right orbit. Orbital venogram showed inferior and medial displacement of the P1 and P2 portions and the second segment of the superior ophthalmic vein. CT scan in coronal view showed a tumor in superior par of the right orbit. Operation was performed by subfrontal approach. It was noticed that the supraorbital nerve entered the tumor. The mass, 5 g in weight, was successfully removed. Pathologically, the diagnosis of neurilemmoma was made. Postoperatively, the proptosis diminished and the ocular movements became normal in all directions. Neurilemmoma of the orbit is an uncommon tumor, accounting for only 1.7-5.6% of all orbital tumors. Identifying the original nerve of the tumor has been even rarer. Including this case, there have been only 8 individually reported descriptions. In three cases the tumor originated from the supraorbital nerve, in three cases from the ciliary nerve, in two cases from the infraorbital nerve.

Adult↗
PubMed1981

[A case report: Osler-Weber-Rendu disease with cerebral aneurysm, cerebral arteriovenous malformation and pulmonary arteriovenous fistula (author's transl)].

Osler-Weber-Rendu disease (hereditary hemorrhagic telangiectagia) is a rare vascular disorder which is characterized clinically by inherited telangiectasia of skin, mucous membrane and recurrent hemorrhage. However, it is not rare that various organs, for instance, lung, liver, intestine are inflicted by vascular disorder in this disease, and central nervous system is not an exception. A 60-year old man was examined for repeated epistaxis and an episode of subarachnoid hemorrhage. In this case, left middle cerebral artery aneurysm, arteriovenous malformation fed by left posterior cerebral artery and pulmonary arteriovenous fistula were revealed by angiography and computerized tomography. It is interesting that various vascular disorders were found in one case.

Arteriovenous Malformations↗
PubMed1978

[Intraventricular extravasation of contrast media through ruptured intracranial aneurysm--report of two cases (author's transl)].

Extravasation of contrast media through ruptured intracranial aneurysm has been reported as an unusual case. Among the reported cases, a ventricular opacification is very rare, and in such cases, the prognosis is worst. In this paper, two cases with an aneurysm bleeding into the ventricular system were reported. Case 1. A 37-year-old woman was admitted because of severe headache and disturbance of consciousness. On admission, she was deeply comatose with decerebrate rigidity. Carotid angiogram taken 2 hours and 30 minutes after the attack revealed an aneurysm of the anterior communicating artery and an extravasation of contrast media into the lateral ventricle. Although a continuous ventricular drainage was instituted immediately, the patient died after five days. Case 2. A 33-year-old man suddenly developed severe headache with subsequent loss of consciousness. Two hours after the onset, the patient was admitted in comatose state. Angiography taken 3 hours after the attack demonstrated an aneurysm of the anterior communicating artery. The posterior horns of the lateral ventricles were opacificated. The patient died after 24 hours. In case 1, it was interesting that a remarkable stenosis of the cisternal portion of the internal carotid artery was demonstrated at the 2nd carotid angiography (lateral view), but the third carotid angiography taken after the continuous ventricular drainage showed a complete recovery from the stenosis. This stenosis was supposed to be caused by a compression of the internal carotid artery against the interclinoid ligamentum. Moreover, a rapid enlargement of the aneurysm was noticed by repeated angiography.

Adult↗