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Biomedical subjects

K U Hamann

Publications and source records attributed to K U Hamann.

14 recordsLinked to original sources
PubMed1980

[Convergence nystagmus: an oculographic analysis].

Convergence nystagmus and nystagmus retractorius represent important ocular motor disorders in cases of periaqueductal involvement and serve as valuable topographical clue in the Sylvian aqueduct syndrome. The oculographic tracings of a 34-year-old male suffering from a pinealoma are presented. It is outlined, under which circumstances convergence nystagmus is elicited, and the typical features of this disconjunctive nystagmus are discussed.

Adult↗
PubMed1980

[Rebound nystagmus (author's transl)].

Rebound nystagmus represents a disorder of ocular motility, encountered in patients with chronic cerebellar lesions. The case history of a 34-year-old patient suffering from cerebellar degeneration is introduced briefly, and the oculographic features of rebound nystagmus are discussed with special remarks on the problems, tracing this kind of ocular motor disturbance.

Adult↗
PubMed1979

Videopupillographic and VER investigations in patients with congenital and acquired lesions of the optic radiation.

In 9 patients with congenital and acquired lesions of the optic radiation, videopupillographic investigations have been employed to determine the pupillographic threshold in the homonymous field defect. The study includes 3 patients suffering from congenital porencephalic cysts. The pupillary response was graded as akinetic, hypokinetic or eukinetic. These classifications yield some diagnostic clues as to the nature of the lesion. An attempt was made using the VER response in these patients to correlate the findings.

Evoked Potentials↗
PubMed1979

[Slowed saccades in various neurological disorders].

4 patients suffering from different neurological disorders are presented, exhibiting a striking slowing down of all saccades. According to their clinical features the problem of slow saccades of supranuclear origin is discussed, to elucidate the question whether these eye movements are saccadic at all. Electro-oculographic traces served to distinguish these particular eye movements, and a computer calculated duration, velocity and acceleration of these saccades, to identify their saccadic nature. 1 patient with Wernicke syndrome showed a remarkable improvement of saccadic velocity following thiamine administration. The findings are reconciled with known saccadic behavior, and a model of the pathophysiological mechanism is cast. The adaptability of the system nearly back to normal in the case of Wernicke syndrome is stressed.

Aged↗
PubMed1979

[Dissociated ophthalmoplegia of abduction: so-called posterior internuclear ophthalmoplegia in Fisher's syndrome].

The anterior internuclear ophthalmoplegia, a dissociated ophthalmoplegia of adduction secondary to an ipsilateral supranuclear paresis of the internal rectus, represents a well-know clinical picture. The dissociated ophthalmoplegia of abduction, however, sometimes referred to as posterior internuclear ophthalmoplegia, is debated. Such an oculomotor disturbance emerging from an ophthalmoplegic polyneuropathy (Fisher syndrome) is presented and illustrated by oculographic tracings. A secondary finding was a limited range of accommodation. Oculographically, the dissociated ophthalmoplegia of abduction reflects a true mirror image of the anterior internuclear ophthalmoplegia. Because of anatomical considerations the dissociated ophthalmoplegia of abduction cannot be regarded as internuclear ophthalmoplegia, and the terminology of posterior internuclear ophthalmoplegia is not tenable. Discussing the pathophysiology of the Fisher Syndrome, it is reflected upon an infranuclear lesion causing an ocular motility disorder featuring supranuclear aspects, and avoiding limitations of peripheral nerve damage.

Accommodation, Ocular↗
PubMed1979

[Systematic deviations of saccadic eye movements in Wallenberg syndrome].

A patient suffering from lateral medullary infarction (Wallenberg's syndrome) is presented, exhibiting a striking bias of all saccadic eye movements toward the side of the lesion. Oculographic tracings demonstrate this oculomotor disorder. Other disturbances of ocular motility which resemble this one superficially are discussed. Interruption of fixation leads to a gliding movement veeringly to the side of the infarction. Since all saccades generated under different circumstances are affected, it is contemplated where the pathological signal is intruded into the prenuclear level, to adulterate all saccades causing lateropulsion of saccadic eye movements.

Adult↗
PubMed1979

[Pseudo-internuclear ophthalmoplegia in a case of myeloma of clivus (author's transl)].

A 72-year-old female suffering from a histologically proven myeloma of the clivus exhibited an eye movement disorder closely resembling a bilateral internuclear ophthalmoplegia. The pertinent clinical findings suggested a peripheral lesions involving both oculomotor nerves. It is discussed under which circumstances an infranuclear lesion can simulate a supranuclear disorder of eye motility.

Aged↗
PubMed1978

[Periodic alternating nystagmus].

In this report 2 cases of periodic alternating nystagmus are presented. The most important parameters of this oculomotor disturbance are shown by oculographic tracings. Discussing the literature it is attempted to cast a model of the pathophysiology of PAN.

Adult↗
PubMed1978

[Impaired visual perception in progressive multifocal leukoencephalopathy; a clinical diagnosis based on sequential computerized axial tomography (author's transl)].

Progressive multifocal leukoencephalopathy (PML) is a viral illness affecting principally cerebral white matter of patients, who have diminished immunologic resistance. A patient is presented, whose initial complaint was blurred vision. Relentless progression over a period of seven months to left hemianopsia, prosopagnosia, alexia with agraphia and eventually cortical blindness was observed. Accompanying the visual impairment was a steady deterioration of mental function. Sequential computerized axial tomography defined low density lesions in the occipital white matter with sparing of the cortical ribbon. As hemianopsia progressed to cortical blindness, the scans confirmed the evolution of bilaterial occipital lesions. The diagnosis was made clinically and established on histological and cultural grounds. Computerized tomography seems to be valuable in diagnosis and monitoring progress of PML.

Aged↗
PubMed1977

[Exogenous keratitis due to chromomycosis (author's transl)].

The author presents a case of exogenous, retrocorneal, limbal infiltration due to chromomycosis, following cataractextraction. Since the etiology of this keratitis was not recognized at the beginning, steroids were applied causing an exacerbation of the keratitis with hypopyoniritis. A perforating, excentric keratoplasty was performed, yielding material for the histological diagnosis, and for the identification of the responsible agent. The cultural and morphological properties were typical of those of a dematiaceous fungus, a chromomycosis. In vitro studies demonstrated this fungus to be less sensitive to 5-fluorcytosin and clotrimazole, and to be less resistant to natamycin, mystatin and amphotericin-B, paving the road for an effective conservative therapy following surgery.

Amphotericin B↗