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Biomedical subjects

L A Barness

Publications and source records attributed to L A Barness.

At least 73 records · Page 4Linked to original sources
PubMed1982

The effect of variations in dietary fatty acids on the fatty acid composition of erythrocyte phosphatidylcholine and phosphatidylethanolamine in human infants.

Human milk, or one of two formulas that derive their fat from vegetable oil, was fed to infants from birth until 4.5 to 6 months of age. Infants fed human mild received 2% of total fatty acids as 20 to 22 carbon polyunsaturated fatty acids. These fatty acids which are not found in vegetable oils, are synthesized by animals from the essential vegetable-derived fatty acids, linoleic and alpha-linolenic acids. Enfamil (Mead Johnson, Evansville, IN) contained three times as much linoleic acid as human milk or SMA (Wyeth Laboratories, Philadelphia, PA); however, the ratios of linoleic/alpha-linolenic acid were 9.0, 18.8, and 11.7 for Enfamil, human milk, and SMA, respectively. Erythrocyte phosphatidylethanolamine and phosphatidylcholine in infants fed human milk had significantly more 20 to 22 carbon polyunsaturated fatty acids than did those infants consuming only vegetable fat. Concentrations of 20 to 22 carbon polyunsaturated fatty acids in the erythrocyte membrane phosphatidylethanolamine and phosphatidylcholine of SMA and Enfamil-fed infants were similar despite very significant differences in the amount of dietary 18 carbon precursor. The degree of unsaturation of both erythrocyte phosphatidylethanolamine and phosphatidylcholine was highest with the feeding of human milk compared to the formulas, but the relative concentration of the four major erythrocyte phospholipids, and the ratio of membrane phosphorus/cholesterol were not affected by these diets.

Breast Feeding↗
PubMed1982

Results of feeding a special formula to very low birth weight infants.

Fifty-eight premature infants weighing less than 1,600 g at birth were fed a special formula. The formula contained nutrients in amounts recommended by the Committee ono Nutrition of the American Academy of Pediatrics for very low birth weight (VLBW) infants. The feeding studies were carried out at newborn nurseries in Tampa, Florida (study A, n = 25), Pittsburgh, Pennsylvania (study B, n = 20), and Oaklawn, Illinois (study C, n = 13). Study subjects were comparable in birth weight, gestational age, and in the duration of follow-up in the nurseries. All study subjects grew at rates of weight acquisition equivalent to the comparative fetal counterpart. Routine anthropometric measurements were similar to those of fetal development curves. Mean protein intake ranged from 2.3 to 3.7 g/kg/day and mean caloric intake from 105 to 150 kcal/kg/day. Late metabolic acidosis in association with prematurity was absent in all subjects studied as demonstrated by normal pH values, bicarbonate, and partial pressure of carbon dioxide. Serum sodium and serum chloride levels were normal. Serum calcium ranged from 8.3 to 10.1 mg/dl and serum phosphorus from 6.0 to 7.5 mg/dl. Total serum protein levels ranged from 4.5 to 5.1 g/dl. Blood urea nitrogen diminished progressively from 5.1 to 2 mg/dl in the course of the study. Serum glucose levels in samples taken prior to and 2 h after feeding did not demonstrate any evidence of reactive hypoglycemia.

Blood Glucose↗
PubMed1980

Failure to thrive.

Failure to thrive is one of the most common diagnostic problems in pediatrics. The term is applied to describe a still poorly defined entity. We will define failure to thrive, review the frequency of various etiologies, and present a logical approach to this problem.

Adult↗
PubMed1979

Chronic diarrhea in children.

Among the many causes of chronic diarrhea in children are food, the most common; inflammation; enzyme deficiencies; metabolic, anatomic, and physiologic abnormalities; and endocrine disorders. The first step in management is to estimate fluid losses and treat dehydration greater than 5%. Workup should include a careful diet history, physical examination, examination of stools and urine, and usually a sweat test. Treatment should be directed to the underlying disease.

Antidiarrheals↗
PubMed1977

Neonatal nutrition.

Doctors should redouble their efforts to encourage mothers to breast feed. This is advantageous both in developed and in developing countries, for in developed countries, morbidity is lowered, while in developing countries, morbidity and mortality are drastically lowered. If the advantages of breast feeding cannot be impressed on the mother, then those formulas which most nearly simulate breast milk should be used. This is economically feasible in any society because of the lowering of morbidity due to conditions such as intestinal disorders, anaemia, eczema and, perhaps, poor brain development; the cost of these diseases should be included in the calculations for formula feedings. Other countries have already made the error and have later improved on feeding cow's milk formulas. Developing countries can avoid this error.

Breast Feeding↗
PubMed1977

Organic acidosis in infants.

Any child or adult who has persistent acidosis after apparently adequate treatment should be suspected of having an organic aciduria, as should any person with a large anion gap and acidosis. The possibility that some can presently be treated, makes early diagnosis urgent. Acidosis occurs in hypoperfused states, and treatment of the hypoperfusion with half- to two-thirds-strength of physiological saline with 40 mEq/1 of potassium may correct the acidosis without there being any need to use bicarbonate.

Acidosis↗