[Vascularization of the crus in defect substitution by lengthening of fractured fragment using Ilizarov's method (an experimental study)].
Crus vascularization in case of substitution of diaphysial defect of tibia by means of proximal fractured fragment lengthening have been studied by anatomo-functional methods in the experiment with 16 dogs. The results of the experiment demonstrated unequal increase of crus blood-filling with localization of the maximum vascularization zone in the projection of a distraction regenerate, displaced fragment and adjoining bone ends. Increase of the operated segment vascularization was caused by dilatation of circulatory channel and formation of new vessels in tissues, subjected to distraction. Tissue hypervascularization in case of distraction of the osseous fragments in defect is a natural reaction, attending prolonged osteogenesis and is preserved till organ-like restructuring of newly formed bone.