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Biomedical subjects

L Calandre

Publications and source records attributed to L Calandre.

At least 37 records · Page 2Linked to original sources
PubMed1978

[Treatment of lennox syndrome with medium chain triglycerides (author's transl)].

Twelve cases of Lennox syndrome were treated with a ketogenic diet with medium chain triglycerides during an average period of five months. All the cases were resistant to drug treatment. In five cases seizures disappeared, in five the frequency decreased and two remained without changes. The alertness improved in seven cases. Electroencephalographic records did not show any important improvement. The diet was generally well tolerated and it only had to be discontinued in one case.

Child, Preschool↗
PubMedDate not supplied

Risk factors for spontaneous cerebral hematomas. Case-control study.

In a case-control study of 73 cases of cerebral hematoma diagnosed by CT scan, significant risk factors were history of hypertension, chronic alcoholism, evidence of hepatic disease, EKG abnormalities and high hematocrit values. Initial blood pressure was significantly higher in cases, but blood pressure on the third day after admission was not different from controls. Hypertension and alcoholism did not show a clear correlation, but data from other studies explain the role of alcoholism in vascular disease through a relation with high blood pressure. Risk factors were similar in lobar and basal ganglia hematomas.

Adult↗
PubMedDate not supplied

Hematocrit and clinical outcome in acute cerebral infarction.

Hematocrits of 131 cerebral infarction cases were correlated with the outcome at 2 weeks. Bivariate analysis showed that cases with more intense admission deficit had lower admission hematocrit and that cases with poorer outcome had lower Day 2 and 4 hematocrits. However, multivariate analysis of several prognostic factors (including hematocrit, glycemia, blood pressure, age, and sex) showed that hematocrit was not independently related to outcome. Higher hematocrit thus is not indicative of less favorable short-term outcome.

Cerebral Infarction↗
PubMedDate not supplied

[Acute confusional syndrome due to bilateral occlusion of the anterior cerebral artery].

We report a case of acute confusional state (ACS) resulted from bilateral occlusion of anterior cerebral artery. This case is outstanding because development of ACS was caused by a focal lesion, without the presentation of focal neurologic signs. Then, cerebrovascular disease must be included in differential diagnosis of ACS, despite the lack of clinically detectable deficit.

Acute Disease↗
PubMedDate not supplied

[Acute confusional syndrome of unknown cause. Prospective study in the emergency room].

In a prospective study conducted in the Emergency Area of a general hospital during a period of 6 months, the Neurology Service was consulted in 45 cases of acute confusional syndrome. Twenty nine of these patients had no immediate apparent etiology. Among the studied patients 5 had a subarachnoid hemorrhage, 4 suffered a central nervous system infection, 3 had a cerebral infarction, 3 presented a metabolic encephalopathy, and the remaining patients had variate etiologies. Clinical signs and symptoms were insufficient to establish the diagnosis and in several cases complementary tests were required (cerebral tomography and cerebrospinal liquid examination). Even in these cases the diagnosis could not be obtained in some patients. The clinical implications of the present findings were discussed.

Acute Disease↗
PubMedDate not supplied

[Prognosis factors in conservatively treated patients with non-traumatic supratentorial intracerebral hematoma].

We have carried out a retrospective study of 90 patients who were diagnosed of non traumatic supratentorial intracerebral hematoma by computerized tomography and received conservative treatment. Clinical features and laboratory and radiographic findings during acute stage as well as the course of the patients during hospital stay (mean, 34 days) are analyzed. The overall mortality rate was 21%. The factors having a significant influence on prognosis were: degree of neurologic impairment at admission, degree of motor impairment at admission, progression of neurologic impairment after the admission, level of glycemia at admission, extent of the hematoma, and presence of mass effect in CT. We conclude that the extent of the hematoma and the level of glycemia at admission are the primary prognostic factors without being related to each other. The "critical" values were 40 cc for the extent of the hematoma and 130 mg/dl for glycemia at admission. Patients with higher values had a poorer prognosis with a worse clinical condition at discharge.

Adult↗