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L H Kortbeek

Publications and source records attributed to L H Kortbeek.

11 recordsLinked to original sources

[Traumatic carotid artery stenosis with cerebral infarct].

After an accident causing a wound beside the larynx a 17-year-old male was diagnosed as having a cerebral haemorrhagic contusion. He was treated conservatively, when six weeks later uncertainty about the diagnosis prompted angiography of the common carotid arteries. The diagnosis had to be revised: a traumatic occlusion of the carotid artery. The patient's favourable recovery was credited to development of a good collateral circulation to which his relative youth contributed. Traumatic occlusion of the carotid artery should be considered when neurological disorders are apparent but the patient is conscious, especially when there is a symptom-free interval.

Adolescent↗

Polyneuropathy due to polyangiitis overlap syndrome.

A patient presented with a mild polyneuropathy, developed a large intra-abdominal haemorrhage and died of cardiac arrest within a few days. Microscopic examination revealed a polyangiitis overlap syndrome (Fauci) with lesions in many organs, including the peripheral nerves and the heart. A myocarditis was considered to be the cause of death.

Humans↗

Bilirubin excess, erythrophages and siderophages in differentiation of blood in cerebrospinal fluid.

Non-artificial blood admixture in cerebrospinal fluid (CSF) can be proven by demonstrating siderophages and 'bilirubin excess'. Bilirubin excess exists when the bilirubin concentration measured in the CSF exceeds the expected CSF bilirubin concentration. The expected CSF bilirubin concentration is calculated by multiplying the serum bilirubin concentration by the albumin quotient. Given a certain method of determination, a difference between the above-mentioned concentrations of less than 0.15 mumol/l must not be regarded as excess. Bilirubin excess was exclusively found in CSF with non-artificial blood admixture. Cytological and spectrophotometric studies are sufficient in screening for non-artificial blood admixture. When there are indications of an increased bilirubin concentration, calculations should be made in order to establish whether bilirubin excess exists. Lumbar puncture within 48 hours of the suspected haemorrhage should be avoided if possible.

Bilirubin↗

Siderophages in differentiation of blood in cerebrospinal fluid.

This paper evaluates demonstration of siderophages as a method of differentiating blood in CSF. Of 1147 CSF samples, 125 were selected which had been obtained by lumbar puncture from patients with proven intracranial haemorrhage (spontaneous or traumatic) after an exactly known interval since the onset of clinical phenomena (haemorrhage or accident). No siderophages were found within 24 hours; in a few cases they were found on the second or the third day, and after that time they were found in the majority of cases. This is a pathognomonic finding. Collection of CSF in successive portions, qualification of the tap by the clinician as 'traumatic' or 'atraumatic', and demonstration of crenated red cells, were found to be unreliable methods. A modified apparatus for sedimentation is described.

Cerebral Hemorrhage↗

[Facial neuralgia].

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Facial Neuralgia↗