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Biomedical subjects

L H Schultz

Publications and source records attributed to L H Schultz.

At least 37 records · Page 2Linked to original sources
PubMed1976

Calcium redistribution into subcutaneous fat at parturition in the dairy cow.

Blood samples and subcutaneous fat biopsies from six dairy cows at -14, 0, 14, and 28 days of parturition were analyzed to test the hypothesis that movement of calcium into subcutaneous fat contributes to hypocalcemia of parturient paresis. Plasma and subcutaneous fat calcium decreased while plasma free fatty acids increased at parturition. No trends were significant in magnesium or total lipid content of subcutaneous fat. Calcium content of subcutaneous fat was related positively to calcium concentration in plasma.

Adipose Tissue↗
PubMed1976

Relationship of insulin concentration to blood metabolites in the dairy cow.

Jugular blood samples were taken at regular intervals from 31 ketosis-prone cows from 2 wk prepartum to 7 wk postpartum. Eleven cows exhibited elevated blood ketones and depressed blood glucose indicative of subclinical ketosis. There were no significant differences between means of normal and subclinically-ketotic cows in serum insulin or blood metabolites prior to calving. However, in early lactation, those cows which developed ketosis showed depressed serum insulin, blood glucose, and plasma triglycerides with elevated ketones, acetate in blood, and free fatty acids and cholesterol in plasma. Milk production was also lower in ketotic cows. Correlations within cow between serum insulin and glucose, total ketones, acetate of blood and free fatty acids, triglycerides, and cholesterol in plasma were .014,--.307, .080,--.421, .413, and -.002 for normal cows and .348, -.425, -.324, -.317, .298, and -.131 for subclinically-ketotic cows. It is suggested that low insulin during ketosis is a reflection of depressed blood glucose and, consequently, adipose lipolysis and hepatic ketogenesis are accentuated while acetate utilization and hepatic triglyceride release are depressed.

Acidosis↗
PubMed1976

Blood and liver metabolites in fed and fasted diabetic goats.

Two trials were to study alloxan diabetes in goats. The data were grouped: 1) normal fed goats (10); 2) 48-h fasted goats (5); 3) fed goats sampled 96 h after alloxan treatment (5); and 4) goats treated with alloxan following a 48-h fast and sampled 96 h after alloxan treatment with continued fasting (3). Groups 1 and 4 exhibited the following means: serum insulin 43.9, 16.4, 9.4, and 6.7 muU/ml; blood glucose 55.0, 47.3, 219.6, and 485.6 mg/100 ml; blood ketones 4.3, 2.6, 36.6, and 28.6 mg/100 ml; blood acetate 4.7, 4.0, 42.7, and 4.9 mg/100 ml; plasma-free fatty acids 1.8, 10.0, 14.4, and 40.5 mg/100 ml; and plasma triglyceride 13.3, 7.0, 47.6, and 12.2 mg/100 ml. Liver samples from five fed goats before and 12 days after alloxan treatment exhibited the following means: phospholipid 27.5 and 26.1 mg/g; triglyceride 21.2 and 98.9 mg/g; and percent lipid 7.2 and 14.4. The diabetes was accompanied by fatty liver development and probably reduction in utilization of acetate and triglyceride in the fed animals.

Animals↗