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Biomedical subjects

L Ortega Medina

Publications and source records attributed to L Ortega Medina.

At least 19 recordsLinked to original sources
PubMed1991

Juxtaglomerular cell tumor of the kidney. Morphological, immunohistochemical and ultrastructural studies of a new case.

The authors describe a Juxtaglomerular Cell Tumor (JGCT) in a hypertensive young man whose symptomatology disappeared when the JGCT was removed. The clinical diagnosis was made before surgery by selective determination of plasma renin activity level in both renal veins and in the draining vein of the tumor. This neoplasm is benign and usually occurs in young patients. The morphological, immunocytochemical and ultrastructural features are described. Other renin-secreting tumors and the theories about histogenesis of JGCT are discussed.

Adult↗
PubMed1991

[Somatostatin in the treatment of established and severe acute pancreatitis in the rat].

The effects of somatostatin (SS) on the treatment of acute pancreatitis were studied in rats. Acute pancreatitis was established by injecting 5% sodium taurocholate in the biliopancreatic duct. Previously, pancreatic necrosis was determined in this experimental model at several intervals without treatment. Treatment was started according different groups: at 12, 16 and 20 hours after induction of acute pancreatitis (IV bolus of 4 ug/kg body weight followed by a 24h continuous infusion of 4 ug/kg body wt/hour). When somatostatin was initiated at 12 or 16h a decrease in serum amylase and lactodehydrogenase was observed, as well as in pancreatic necrosis resulting in 0% mortality after 24h of treatment. When somatostatin was started at 20h there was no changes in the lethal outcome of the disease.

Acute Disease↗
PubMed1991

[Experimental acute pancreatitis in the rat. The quantification of pancreatic necrosis after the retrograde ductal injection of sodium taurocholate].

Acute haemorrhagic pancreatitis was induced in rats by injecting 5% sodium taurocholate into the common biliopancreatic duct. The condition was associated with an increase in the serum amylase levels as well as progressive pancreatic necrosis resulting in 100% mortality before 36 hours. This experimental model was documented by quantifying nine different parameters of pancreatic necrosis and giving more information about the induced lesion. The extent of pancreatic necrosis was evaluated at different intervals, 5.77% at 12 hours, 14.9% at 24 hours, and the rats died before 36 hours of pancreatitis induction with an average percentual necrosis of 29.9%. This model seems suitable for more pathogenic as well as therapeutic studies on acute pancreatitis in the rat.

Acute Disease↗
PubMed1989

[Ductal adenocarcinoma of the head of the pancreas: associated pathology of body and tail].

Histologic study of the body and tail of the pancreas in a patient in whom total duodenopancreatectomy was carried out for ductal adenocarcinoma of the head (ADC) demonstrates the existence of dysplasia of the pancreatic duct in its entire length associated with intente parenchymal atrophy. It is considered that cephalic duodenopancreatectomy in ductal adenocarcinoma of the head of the pancreas should be based on an adequate anatomic, histologic and functional evaluation of the residual pancreas.

Adenocarcinoma↗
PubMed1989

[Segmental portal hypertension caused by aneurysm of the distal splenic artery].

A case is reported of segmental portal hypertension secondary to compression of the splenic vein by an aneurysm of the distal splenic artery. Splenectomy was used to treat the aneurysm and segmental portal hypertension, as well as the hypersplenism existent. Collateral circulation through the short gastric veins originated grade V gastroesophageal varices, which makes this mechanism of production equivalent to that described in cases of thrombosis of the splenic vein.

Aged↗